去神经

The activation of NRM neurons by electroaeupuncture was eliminated after decerebration at superior colliculus level, indicating that this effect of EA depends mainly on the supra-midbrain structure. Furthermore, all treatments including AR isolation, disconnection of #beta#-endorphinergic tract and micrpinjection of naloxone or anti-#beta#-endorphin serum into PAG, would make the effect of EA disappear. Thus, it is the #beta#-endorphinergic pathway between AR and PAG-NRM system that mediates the major excitatory effect of EA on NRM.

检验AR至 PAG-NRM的神经通路在电针效应中的作用：实验显示上丘水平去大脑后，电针兴奋NRM的效应完全消失，表明此效应主要依赖中脑以上结构实现，进一步分别用Halfisz氏刀游离AR、切断AR至PAG的β-End能束、PAG内注射纳洛酮或抗β-End血清后，也使电针效应基本上完全消失，说明电针使NRM神经元兴奋的效应，主要通过AR与PAG-NRM间的β-End能神经通路实现。

The author uses genetic algorithm to optimize the structure of neural network and solves this problem.

而该文提出了用遗传算法去优化神经网络的结构，很好的解决了神经网络的结构问题。

Objective To explore the effect of controlled release of nerve growth factor on peripheral nerve defect repaire by acellular nerve graft.

目的 探讨化学去细胞异体神经复合缓释神经生长因子（nerve growth factor，NGF）修复周围神经缺损的效果。

During the relatively refractory period a neuron may be stimulated to fire, given increased stimulation. Neurons are capable of representing differences in input only through rate of response. Myelinted fibers conduct the wave of depolarization more rapidly than demyelinated fibers, primarily due to saltatory conduction.

在相对反抝期一条神经可能会被刺激过度，特定增加的刺激，神经单位是足以表现在输入的差异性，仅透过反应的比率，有髓的纤维引导去极波比脱髓鞘纤维更加快速，主要是由於跳跃性的传导。

A total of 150 mesenchymal neoplasms of the GI tract, abdominal cavity, and retroperitoneum were retrieved from consult and surgical pathology archives, including 54 GISTs (8 KIT-negative; 13 desmin-positive), 17 GI leiomyosarcomas, 11 GI mural leiomyomas, 13 leiomyomas of the muscularis mucosae, 12 gastric schwannomas, 15 inflammatory myofibroblastic tumors, 9 cases of mesenteric desmoid fibromatosis, 10 dedifferentiated liposarcomas, and 9 malignant peripheral nerve sheath tumors.

我们从会诊病例及活检病例中找出150例间叶性肿瘤（来自胃肠道、腹腔和后腹膜），其中包括54例GISTs（8例KIT阴性、13例desmin阳性）、17例GI道平滑肌肉瘤、11例GI道肌壁间平滑肌瘤、13例GI道粘膜肌层平滑肌瘤、12例胃的神经鞘瘤、15例炎性肌纤维母细胞瘤、9例肠系膜韧带样纤维瘤、10例去分化型脂肪肉瘤和9例恶性外周神经鞘膜瘤。

None of the GISTs, desmoid tumors, inflammatory myofibroblastic tumors, schwannomas, dedifferentiated liposarcomas, or malignant peripheral nerve sheath tumors showed cytoplasmic reactivity for smoothelin.

所有GISTs、韧带样纤维瘤、炎性肌纤维母细胞瘤、神经鞘瘤、去分化脂肪肉瘤和恶性外周神经鞘膜瘤病例的瘤细胞浆均不表达smoothelin。

Extraterritorial neuropathic pain correlates with multisegmental elevation of spinal dynorphin in nerve-injured rats. Pain 2000;86:185-194.[16]Knox RJ, Isaac L. Effects of selective and non-selective kappa-opioid receptor agonists on cutaneous C-fiber-evoked responses of rat dorsal horn neurons.

在慢性炎症及神经损伤或过敏等引起的伤害性感受中，大量的Dyn A生成，通过NMDA 受体直接或间接地产生一些炎性介质或细胞因子，从而加强神经病理性疼痛的形成，其作用机制有待于我们进一步去探讨。

The fMRI studies showed that,(1) when information obtained from self-perspective covered the information obtained from other-perspective, compared to a deductive reasoning task, the mental inference task induced increased neural activity in precuneus, decreased neural activity in ventral MPFC and enhanced functional connectivity between these two brain areas;(2) this neural activity pattern was observed in understanding human's belief, but not in understanding artificial intelligence;(3) when information obtained from other-perspective covered the information obtained from self-perspective, the mental inference task induced additional increased neural activity in dorsal MPFC and the right frontal cortex, as well as enhanced functional connectivity between these two cortical areas in addition.

EEG实验发现，额叶alpha频段神经活动在信念理解任务的早期（400-800 ms）表现为去同步化，顶叶alpha频段神经活动在信念理解任务的较晚时程（600-1000 ms）则表现为同步化。

When the effect ofNTP was examined after depletion of monoamines in the spinalcord by intrathecal neurotoxins, the antiallodynic and antihyperalgesiceffects were still observed after serotonergic denervation,but not after noradrenergic denervation.

鞘内给予神经毒素耗竭脊髓单胺递质后检查 NTP 的作用时，去除5羟色胺能神经支配后 NTP 的抗触觉性痛感和抗痛觉过敏作用仍然存在，但是去除去甲肾上腺素能神经支配后则消失。

Neural stem cells have a strong self-renew mechanism and it can transform after a little break. Neural stem cells have a long term survival, which mean that it has more probability of wrong copy than mature cells. These cells are formed glioma stem cells in the end. The genes who adjust neural stem cells can express in glioma stem cells, which hold out glioma stem cells from neural stem cells. There is another presume that glioma stem cells come from differentiated cells. Through the gene break of these cells, they can obtain characteristics of stem cells, then form glioma stem cells.

神经干细胞具有很强的自我更新机制，获得较少突变即有可能恶性转化，而且干细胞存活时间较长，这意味着干细胞比成熟细胞发生细胞复制的错误几率更大，因外界环境的刺激而发生突变的机会更多，最终形成脑胶质瘤干细胞，同时调节神经干细胞增殖和自我更新的基因在脑胶质瘤的脑胶质瘤干细胞中也表达，这也是支持神经干细胞是脑胶质瘤干细胞来源的；也有推测认为它可能起源于已分化的细胞，由这些细胞突变发生去分化得来，并通过基因突变而获得了干细胞自我更新的特性，从而形成脑胶质瘤干细胞。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。