去甲肾上腺素

The concrete usage of noradrenalin: mix 4mg noradrenalin or 8 mg noradrenaline bitartrate (The potency of 2mg noradrenaline bitartrate equals to that of 1mg noradrenalin) with 250ml equilibrium liquid with or without salt. They will form 16ug/ml noradrenalin solution or 32ug/ml noradrenaline bitartrate solution.

去甲肾上腺素的具体用法：将去甲肾上腺素4mg或重酒石酸去甲肾上腺素8mg(2mg重酒石酸去甲肾上腺素效价与1mg去甲肾上腺素相同)加入250ml含盐或不含盐的平衡液中，产生16ug/mL去甲肾上腺素液或32ug/mL重酒石酸去甲肾上腺素液。

RESULTS: Basilar artery, thoracic aorta and ventricular papillary muscle Resting tension of basilar artery was increased by DL but decreased by niof basilar artery and thoracic aorta induced by noradrenaline bitartrate and KCl can be relaxed by DL in a dose dependant manner, and the half-effective concentration was (6.69±3.12)×10-4,(3.41±1.52)×10-3mmol/L for basilar artery, and (1.49±0.59)×10-3,(2.91±0.99)×10-3 mmol/L for thoracic aorta, displaying stronger inhibition on the constriction of basilar artery induced by noradrenaline bitartrate than on the contraction induced by KCl.

重酒石酸去甲肾上腺素、KCl所致基底动脉及胸主动脉收缩，二氢石蒜碱使之呈剂量依赖性松弛，其半数有效浓度值：对基底动脉分别为(6.69±3.12)×10-，(3.41±1.52)×10-3 mmol/L；对胸主动脉分别为(1.49±0.59)×10-3，(2.91±0.99)×10-3 mmol/L。拮抗重酒石酸去甲肾上腺素所致基底动脉收缩作用明显强于KCl。尼莫地平对KCl所致收缩的抑制显著强于对重酒石酸去甲肾上腺素。

Consider to discover, OSAHS patient appears because of relapsing in nightly Morpheus disease of blood of low oxygen, tall carbonic acid, consensual as disorder as parasympathetic nerve function, already made coronary heart disease (the cardiovascular disease such as CHD) sparks importantly element, can cause all sorts of rhythm of the heart wrong, aggravating cardiac muscle is short of blood, produce sudden death of heart source sex even. 1 O SAH S and hypertensive disease consider to discover, OSAHS patient's nightly small arousal can bring about sympathetic stimulant increase, adrenalin can nerve medium if adrenalin, go adrenalin secretes armour grow in quantity, heighten of material of phenolic amine of catechu of nightly make water.

探究发现，OSAHS患者夜间睡眠中因反复出现低氧、高碳酸血症、交感和副交感神经功能紊乱，已成为冠心病等心血管疾病重要的触发因素，可诱发各种心律失常、加重心肌缺血，甚至发生心源性猝死。1 O SAH S和高血压病探究发现，OSAHS患者夜间的微觉醒可导致交感神经兴奋性增强，肾上腺素能神经介质如肾上腺素、去甲肾上腺素分泌增多，夜间尿儿茶酚胺类物质增高。

The hearts were extracted from rats and set up as acute hibernation models.The spontaneous and stimulationevoked releases of myocardial noradrenalin during acute hibernation were determined by using a highperformance liquid chromatography method,the effect of desipramine and tyramine on release of myocardial noradrenalin was also evaluated.

用高效液相色谱法测定急性心肌冬眠时心脏去甲肾上腺素的自发性和电场刺激引起的释放，并评价去甲丙咪嗪和酪胺对去甲肾上腺素释放的影响及酪胺对心率的影响。

After treatment with the above hormones, the rats were established anesthesia with urethan (1g/kg, i. p.); polyethylene catheters were inserted into the external jugular vein and the common carotid artery for drug administration, and then blood pressure was recorded. The arterial catheter was connected to a pressure transducer, and the blood pressure was displayed on the channel of BL-310 Experimental System of Biological Function through IBM computer. We assessed the changes of resting arterial blood pressure, the pressor responses of angiotensin Ⅱ(Ang Ⅱ, 0.01mg/kg), noradrenaline (NA, 0.01mg/kg) and adrenaline (Adr, 0.016mg/kg). The depressor responses of Ach (0.006mg/kg) and carotid sinus baroreflex were also discussed after treatment with the hormones in OVX rats.

用不同雌激素处理21天后，将动物用25％的氨基甲酸乙酯腹腔麻醉（1g／kg），做颈总动脉插管，通过压力换能器，将血压变化输入计算机，通过BL-310智能型生物信号显示与处理系统的压力测定模块，观察去势鼠及不同雌激素替代治疗大鼠静脉注射血管紧张素Ⅱ（AngⅡ，0.01mg／kg）、去甲肾上腺素（NA，0.01mg／kg）、肾上腺素（Adr，0.016mg／kg）的升压反应以及乙酰胆碱（Ach，0.006mg／kg）的降压反应和压力感受性反射的变化。

ADRENOCHROME / Recreational / 30 Minutes / Adrenochrome is an oxidation product of adrenaline.

肾上腺素/休闲/ 30分钟/肾上腺素是一种氧化产物肾上腺素（ ephinephrine ，去甲肾上腺素）。

Serotonin (5-HT), its precursors tryptophan and 5-hydroxytryptophan (5-HTP), major metabolites 5-hydroxyindoleacetic acid (5-HIAA); and 3-methoxy-4- hydroxyphenylethylene glycol , homovanillic acid , the respective major metabolites of noradrenaline and dopamine were measured by a method of HPLC.

应用高效液相色谱法测定其中5—羟色胺（5-HT）系列代谢物：色氨酸、5-羟色胺酸（5-HTP）、5-HT、5-羟吲哚乙酸（5-HIAA）；多巴胺的主要代谢产物3-甲氧基-4-羟基苯乙酸；去甲肾上腺素的主要代谢产物3-甲氧基-4-羟基苯乙二醇等。

METHODS: The maximum number of platelet binding sites and their affinity for [3H]-rauwolscine, a selectie alpha 2-AR antagonist, were measured in unmedicated and medicated major depressed patients and in normal olunteers.

背景：曾认为抑郁症可以伴随中枢α2肾上腺素受体密度的降低，继发突触前儿茶酚胺神经元负性反馈的损害，导致刺激后去甲肾上腺素输出和释放去抑制功能的减弱。

Results AE from CM had vasodilating effect on the contracti on of mesentery blood capillary in guinea pigs and the contraction of rat thoracic aortic ring induced by l0μmol/L phenylephrine, even under the condition of pre-treating with glibenclamide (1μmol/L) or propranolol (3μmol/L). When the endothelium of the rat thoracic aortic ring was removed, AE from CM had not vasodilating effect hut constrictive effect. AE from CM could also increase the contents of NO, NOS and cNOS in the aortic tissue and had no obvious effect on the NO content in plasma and iNOS in aortic tissue of rats.

结果 桑白皮丙酮提取物有抑制去甲肾上腺素收缩豚鼠肠系膜微血管的作用，抑制苯肾上腺素(10μmol/L)收缩离体大鼠胸主动脉环的作用，预加优降糖或普蔡洛尔(3μmol／L)后仍然有松弛作用；大鼠胸主动脉环去内皮后药物无松弛作用，反而起收缩作用；显著升高血管的NO和NOS、cNOS含量，对大鼠血浆的NO和血管iNOS的含量影响不大。

In the spleen of the adult mouse, GAP-43-like immunoreactive fibers were extensively distributed not only in association with vascular plexuses, but also in the parenchyma of the spleen,.especially in the inner layer of the peripheral arterial Lymph Sheath of the white pulps, where T lymph

在PALS外层、边缘区和红髓，阳性纤维相对较少。6-OHDA化学损毁法证明，脾脏内的GAP-43免疫反应纤维主要是去甲肾上腺素能，也有少数不含去甲肾上腺素的纤维，可能是肽能神经纤维。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。