原细胞

RESULTSdiabetic rats showed typical symptoms involving elevated serum glucose and weight loss (2)The rats in DM+VaD group reacted slower and made more error numbers in Y-maze than the rats in VaD group did, with a prolonged total reacting time in a whole day.(3)More neurons with serious damages, such as edema, ischemia or pyknosis could be found in DM+VaD group than in VaD group. More disarrangement of cone neurons, obvious glia proliferation, and more neuron apoptosis were found in CA area of hippocampus. CONCLUSIONS (1)2-VO in STZ-induced diabetic rats succeeded in establishing VaD animal models.(2) The cognitive dysfunction induced by VaD rats had been deteriorated by diabetes mellitus.(3)Morphology evidences proved that diabetes mellitus aggravated brain damages induced by VaD.PART II Effect of diabetes mellitus on the cholinergic nervous systemin CA1 area of hippocampus of VaDOBJECTIVE To estimate the role of cholinergic nervous system in hippocampus during the process of cognitive dysfunction aggravated by diabetes mellitus in DM+VaD group. METHODS Observe the changes of ChAT protein by immunohistochemistry stain. Chose three rats randomly from each group at each pointat two week, four week, and eight week, then sepa

经腹腔注射链脲佐菌素诱导慢性实验性糖尿病，1周后永久性结扎双侧颈总动脉(2-VO)制作VaD模型记录术后2周、4周和8周各组大鼠的体重及血糖；应用Y-迷宫检测空间定向学第二军医大学博士论文中…丈摘…要1ia；一色恤~﹁染澎糕行HE染色观察组织病理学孪叱；胶质细胞原纤维酸性蛋白protellZ，Cf冰尸标记观察星形胶质细舱的激活情况、橄声软公点娜育票票众糯黑橄居端粗默孺篇筑价井袱第二部分糖尿病对血管性痴呆大鼠海马c心区胆碱能神经系统的影响探讨海马CAI区胆碱能神经系统在糖尿病加重确D一大鼠认知障碍中所实验动物及分组同第一部分，应用免疫组织化学染色方法现察海马CAI{介狱牛第晕军l袭拔常博粗{论义

Efficacy: it can effectively discompose melanin, inhibit tyrosinase activity, cut off root of melanin generation, deep whiten and nourish skin, remove sallow and dull skin, present whitening and crystalline skin, resist oxidation and remove free radical, promote substrate cells'regeneration and splitting, promote elastic fiber and collagen fiber growth, and resume skin elasticity and luster.

功效：有效分解黑色素，抑制酪氨酸酶的活性，切断黑色素生成的根源，深层美白营养肌肤，去除黄气暗哑，绽放肌肤净白莹采，抗氧化清除自由基，刺激基底层细胞再生分裂，促进弹力纤维和胶原纤维生长，重现肌肤弹性和光泽，玫瑰精油源源不断供给肌肤充足的水分和营养，深度改善细纹和干燥现象，改善肤色，给肌肤前所未有的白皙亮泽。

Cortical neurons had well-preserved morphological state and normal electrophysiological properties after dura mater was treated by the enzyme.

从细胞内记录和胞内染色的结果可以看出，用胶原酶软化硬脑膜对皮层神经元的功能和形态均无影响。

Dystrophic epidermolysis bullosa is a group of genetic disease caused by loss-of-function mutations in the type Ⅶ collagen gene. The clinical phenotype is characterized by the blistering and scaring of the skin from trauma and there is increased complication of squamous cell carcinoma, which is the major cause of death in patients with DEB.

营养不良性大疱性表皮松解症是一种由于人体Ⅶ型胶原基因突变或缺失所引起的遗传性疾病，临床特征为创伤诱发的水疱和瘢痕，并发症以恶性鳞状细胞癌多见，是导致患者死亡的主要因素，目前仍无有效的治疗方法。

All 7 cases were characterized by deposition of dystrophic or psammatous calcifications accompanied with chronic inflammatory cells infiltration in the stroma. In some cases, there was formation of germinal centers.

特徵性形态学表现为在胶原化的纤维组织间可见散在的钙化灶或砂砾小体，间质内伴有多少不等的淋巴细胞和浆细胞浸润灶，部分病例中可见生发中心形成。

Here, we report on the first systematic long-term study of fibroblast therapy in a mouse model for recessive dystrophic epidermolysis bullosa, a severe skin-blistering disorder caused by loss-of-function of collagen VII.

我们报道了第一例长期性系统性的关于成纤维细胞治疗隐性营养不良性大疱性表皮松解症小鼠模型的研究。隐性营养不良性大疱性表皮松解症是一种严重的由于VII-胶原蛋白功能丧失引起的出疱性皮肤病。

Objective To explore the apoptosis induced by hydrogen peroxide( H2O2) in protoscolex of Echinococcus granulosus.

摘要目的探讨过氧化氢（H2O2）体外诱导细粒棘球蚴原头节细胞凋亡。

All of the FGFs family are the mitogen,affecting the ectoderm,entoderm and the mesenchymal cells.

全部的FGFs都是广谱的有丝分裂原，作用于外胚层、内胚层和间充质来源的细胞。

The other cells of the tetrad undergo degeneration. The mature embryo sac consists of an egg apparatus, a central cell, and 3 antipodal cells. The embryo sac is a monosporic Polygonum type. The zygote develops into a mature embryo going through proembryo, globular, heart, and torpedo stages.

胚囊发育类型为单孢子蓼型胚囊； 2胚的发育经过原胚、球形胚、心形胚和鱼雷胚等阶段发育为成熟胚；胚柄在球形胚时期最发达，但胚柄短小，只由3个扁平细胞组成，之后逐渐退化。

But at this stage, skin cells begin to flatten, and less collagen and elastin means more sag.

但是，在这个阶段，皮肤细胞开始拉平，减少胶原蛋白和弹性蛋白意味着更多的凹陷。

Lugalbanda was a god and shepherd king of Uruk where he was worshipped for over a thousand years.

Lugalbanda 是神和被崇拜了一千年多 Uruk古埃及喜克索王朝国王。

I am coming just now,' and went on perfuming himself with Hunut, then he came and sat.

我来只是现在，'歼灭战perfuming自己与胡努特，那麼，他来到和SAT 。