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原纤维

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Methods The disorder of myofibrils,swelling of mitochondria with broken cristae and decrease of cytochrome C oxidase activity caused by ADR were prevented by the administration of teapol.

结果 阿霉素所造成的大鼠心肌细胞肌原纤维排列紊乱、线粒体肿胀,嵴断裂及细胞色素C氧化酶活性降低,可明显地被抗氧化剂天力体保所防止。

Each system is formed by a series of rings called lamellae, which are deposits of mineral salts and collagen fibers.

每个系统是由一系列叫做片晶的圈构成的,而这些片晶又是矿物盐类以及胶原纤维的沉积物。

Results: The surface of discs showed dim, shallowing wave-like structure in short indentation time, while in long indentation time we found gel losing, collagen bundles disclosing and cave-like damage.

结果:短时压缩使关节盘表面波纹状结构变浅、模糊;过长时间压缩可使其凝胶层脱落,胶原纤维束暴露或洞样缺损,尤其在关节盘中带。

The injured gill cells had a different ultrastructure, with endoplasmic reticulum dilated and vesiculated, mitochondrial cristaes partially disintegrated, gill fibre cuticles denatured and dropped. The ultrastructural damages to hepatopancreatic cells were characterized by the decrease of microvilli, swelling and breaking up of mitochondria, expansion of endoplasmic reticulum, disruption and vacuolization of nucleus, tumefaction of nuclear membrane and increase of lipid droplets. As for the toxicological changes of myocardial cells, the mitochondria were swelling, having a lot of vacuolus and disintegrating, while the myofibrils were irregular with endoplasmic reticulum dissolved.

鳃细胞的损害表现为鳃丝水肿,细胞器溶解,角质层变薄,线粒体、内质网的肿胀、解体;肝胰脏细胞的主要损害特征为肝管微绒毛减少,线粒体水肿解体,内质网扩张,细胞核空泡化,核膜水肿和脂肪滴增加;心脏细胞的毒理变化为线粒体内嵴肿胀、瓦解,肌原纤维不规则,内质网溶解。

Results The physically and chemically treated SIS was pure with more hollows on the surface and undamaged collagen fibers. The cartilage cells could grow, adhere to and differentiate on the layer of SIS and enter into the hollows, secrete a great deal of extracellular matrices.

结果 经物理和化学处理的SIS纯度高、孔隙多,胶原纤维未受损;软骨细胞在SIS材料上生长、黏附、增殖,并能长入材料的孔隙内,分泌大量的细胞外基质成分。

To master fluid mosaic model of cell membrane;peripheral proteins and integral proteins;the type of extracellular matrix;Collagen;glycosaminoglycan and proteoglycan;elastin; Occliding junctions;anchoring junctions;desmosome hemidesmosome、the structure and function of adhesion belt and focal adhesion;cell coat;cadherins;selectin;Ig-superfamily; integrins

掌握生物膜的流动镶嵌模型;膜周边蛋白和膜内在蛋白;细胞外基质的类型;胶原纤维;糖胺聚糖和蛋白聚糖;弹性蛋白;封闭连接;锚定连接;桥粒与半桥粒、粘着带与粘着斑的结构与功能;细胞外被;钙粘素;选择素;免疫球蛋白超家族;整联蛋白

In fact, their incubation in a free cell system gives a stable dimerized form of alpha-synuclein that has been described as the critical rate-limiting step for its abnormal fibrillation.

事实上,他们在一个游离细胞系统培育产生一个稳定的Α-synuclein二聚物形式,已经被描述作为异常原纤维形成的关键限速步骤。

The lung was fixed in 10% formalin solution and embedded in paraffin for Haematoxyffin-eosin staining to observed the form of embolisms, Masson staining to assess collagen and fibrin/fibrinogen distribution and Toluidine blue staining was to observe mast cell distribution.

以10%甲醛固定肺组织,制成组织切片,行苏木精-伊红染色观察血栓的形态,Masson染色观察栓塞血管的胶原纤维分布,甲苯胺蓝染色观察血管栓塞处肥大细胞分布。

With the same treatment on SHA-collagen, the digestion extents are 52%, 43% and 42%.

相对地、三种不同SHA含量的胶原纤维降解百分率为52%、43%、42%。

Act or process of forming fibrils .

形成原纤维的动作或过程。

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