卵巢

Methods］ A total RNA from 10 cases withovarian papillary serous cystadenocarcinoma and normal ovarian tissues were extracted by a single step method of gene chip. The cDNA retro?鄄transcribed from equal quantity mRNA derived from 10 cases with ovarian papillary serous cystadenocarcinoma and normal ovarian tissues. The cDNA strand was labeled with fluorescent Cy5 and Cy3(normal ovarian epithelial cells) as probes.

方法］ 采用基因芯片技术，按一步法分别抽提10例卵巢浆液性乳头状囊腺癌患者的癌组织和正常卵巢组织的总RNA并纯化mRNA；逆转录合成以Cy5荧光标记卵巢癌细胞和Cy3荧光标记正常卵巢上皮细胞的cDNA作为探针，在含有8 464点人类体细胞基因模板的表达谱cDNA芯片上进行杂交。

The results showed that LRK could significantly inhibit the mammary hyperplasia caused by estrin, reduce the content of serum 〓, raise P level, promote the production of DA in hypothalamus and mammary tissue, decrease the release of 5-HT, inhibit the secretion of serum PRL, reduce the sensitivity of mammary tissue to estrin and progestogen, prevent the expression of mammary tissue on PCNA, the effect was equal to triphenylamine oxide, obviously superior to the control group of Ru Zeng Ning.

通过实验研究，建立了具有肾虚肝郁证侯群的乳腺增生病理模型，利用免疫组化技术、图像分析系统、荧光分光光度法、放射免疫法等现代医学研究方法，从病理形态学、医用体视学、神经内分泌学、阴道细胞学等方面，观察了利乳康口服液对模型动物整体状态、乳腺组织病理形态、血清激素、神经递质以及卵巢功能的影响，结果表明利乳康口服液能明显抑制雌激素引起的乳腺组织增生，降低血清E〓含量，升高P水平，促进下丘脑及乳腺组织中DA的产生，减少5-HT释放，抑制血清PRL分泌，降低乳腺组织对雌、孕激素的敏感性，阻止乳腺组织PCNA的表达，其效应与三苯氧胺相当，明显优于乳增宁对照组；另外，利乳康口服液还能缓解模型动物肾虚肝郁证侯群，增加其体重，调节其整体状况，改善模型动物不规则动情周期，减轻其子宫及卵巢的病变，抑制子宫内膜的过度增长，调节卵巢功能，揭示了中药利乳康口服液对神经内分泌和卵巢功能的整体调控作用，以及对本病发生、发展的预防作用

both a high level of testosterone and a more number of follicle can increase the synthesis of estrogen after the ovary excitation test.the sensitivity of foh ovary to the ovary excitation test is higher when the follicle number is more.

雄激素水平和卵泡数量均可影响卵巢兴奋试验后e2的合成，且具有协同作用，卵泡数量较多的患者对卵巢兴奋试验更加敏感。多囊卵巢综合征；功能性卵巢高雄；雌激素

Results 1.VEGF-C expression and MVD in epithelial ovarian carcinoma were higher than those in normal ovarian tissue (P.05),TSP-1 expression were lower than those in normal ovarian tissue (P.05);2.In epithelial ovarian carcinoma tissue,VEGF-C expression and MVD increasing according to progression of the FIGO phase but TSP-1 expression decreasing according to progression of the FIGO phase.

结果 卵巢上皮癌中VEGF-C表达及MVD均明显高于正常卵巢组织(P.05)，TSP-1表达低于正常卵巢组织(P.05)；卵巢上皮癌组织中，VEGF-C表达及MVD随国际妇产科联盟分期的进展而增高(P.05)，TSP-1表达随FIGO分期的进展而降低(P.05)。

We detected the expression of OCY-66, OCY-189 and OCY-142 in 28 ovarian cancers and 30 normal ovarian tissues by RT-PCR, and studied the expression of OCY-142 in these ovarian cancers and normal ovarian tissues by nonradioactive hybridization in situ.

采用RT-PCR和原位杂交的方法研究卵巢癌相关肿瘤抗原新基因在卵巢癌组织和正常卵巢组织中的表达，分析其与卵巢癌临床指标的关系。

The goal of this study was to compare long-term health outcomes and mortality after oophorectomy or ovarian conservation in 29,380 women participants of the Nurses' Health Study who had a hysterectomy for benign disease. Of these, 16,345 (55.6%) had hysterectomy with bilateral oophorectomy, and 13,035 (44.4%) had hysterectomy with ovarian conservation.

这项研究的目的在於比较卵巢切除术或是卵巢保留，对参与护理人员健康研究之29，380位因良性原因切除子宫之女性的影响；在这些病患中，16，345位（55.6%）接受子宫摘除与双侧卵巢切除，而13，035位（44.4%）接受子宫摘除与卵巢保留。

ASCL2 gene escapes genomic imprinting in skeletal muscle,fat, heart,liver,spleen,lung,kidney,stomach,small intestine,uterus,ovary and pituitary.(2) Both of the transcripts of NNAT gene is paternally expressed in skeletal muscle,fat,heart, liver,spleen,lung,kidney,stomach,small intestine,uterus,ovary and pituitary.(3) DIRAS3 gene is paternally expressed in skeletal muscle,fat,heart,liver,spleen,lung, kidney,stomach,small intestine,uterus,ovary and pituitary.(4) DLX5 gene is maternally expressed in skeletal muscle,fat,spleen,lung,stomach,small intestine,but escapes imprinting in heart,liver,kidney,uterus,ovary,testicle and pituitary.

1ASCL2基因在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中均逃脱印记；(2)NNAT基因两个转录本在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中均为母系印记：(3)DIRAS3基因在肌肉、脂肪、心、肝、脾、肺、肾、胃、小肠、子宫、卵巢和垂体中母系印记：(4)DLX5基因在骨骼肌、脂肪、肺、脾、胃和小肠中表达母本来源的等位基因，即表现为父系印记；而在心、肝、肾、子宫、卵巢、睾丸和垂体中表达双亲来源的等位基因，即逃脱印记。3。

Methods Expression of p16 was examined in 30 cases of normal ovarium, 32 benign ovarian tumor, 8 borderline ovarian tumor and 103 ovarian cancer by using ABC immunohistochemistry. The association between p16 expression and clinicopathology was also analyzed.

用免疫组化 ABC法检测30例正常卵巢、32例卵巢良性肿瘤、8例交界性卵巢肿瘤和 10 3例卵巢癌组织中的抑癌基因 p16蛋白表达情况，分析 p16蛋白表达与卵巢癌分级分期、转移预后的关系。

Down-regulated expression of PTEN and up-regulated expression of VEGF〓 were considered as two important events in tumorigenesis of ovarian cancer and could act as molecular markers to indicate the pathobiological behaviors of ovarian cancer. Decreased PTEN expression and increased VEGF〓 expression were closely associated with tumorigenesis and pathobiological behaviors of ovarian endometrioid and serous cancer respectively. Reduced expression of PTEN gene might be involved in carcinogenesis and progression of ovarian cancer by up-regulating the VEGF〓 expression to enhance angiogenesis.

在卵巢癌组织中PTEN基因mRNA表达下调和VEGF〓基因mRNA表达上调是卵巢癌发生的重要分子事件，可作为反映卵巢癌病理生物学行为的指标，且分别与卵巢内膜样癌和浆液性囊腺癌的发生和病理生物学行为关系密切；PTEN基因mRNA表达降低可能通过上调VEGF〓基因mRNA表达促进新生血管形成，进而参与卵巢癌的发生和侵袭。

Methods: Fifty-four female SD rats (8-month-old) were randomly divided into three groups, 18 rats in each group, They included a sham-operated group, an ovariectomized group and a tonifying kidney recipes therapy group, After 3 months, the animal models of osteoporosis were established, pour titanium smooth columnar implants were placed in the proximal metaphyses of left tibiae of the rats.

选用纯种8月龄雌性SD大鼠54只，随机分为3组：假手术组，卵巢切除组，卵巢切除加中药治疗组，每组18只。切除卵巢切除组及中药组大鼠双侧卵巢，假手术组大鼠仅切除双侧卵巢附近的脂肪组织。术后3个月，在三组大鼠胫骨近中干骺端分别植入纯钛光滑柱状种植体。

However, as the name（read－only memory）implies, CD disks cannot be written onorchanged in any way.

然而，正如其名字所指出的那样，CD盘不能写，也不能用任何方式改变其内容。

Galvanizes steel pallet is mainly export which suits standard packing of European Union, the North America. galvanizes steel pallet is suitable to heavy rack. Pallet surface can design plate type, corrugated and the gap form, satisfies the different requirements.

镀锌钢托盘多用于出口，替代木托盘，免薰蒸，符合欧盟、北美各国对出口货物包装材料的法令要求；喷涂钢托盘适用于重载上货架之用，托盘表面根据需要制作成平板状、波纹状及间隔形式，满足不同的使用要求。