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半乳糖

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Then to improre the change tendency of Senile rat's bone metabolism.Conclusion Intaking high does of D-galactose for a long time can cause Wistar male rat aging and induce the changes of sclerotin meanwhile.But zeaxanthin can delay the occurrence of the changes.

结论长期大量摄入D-半乳糖导致大鼠衰老的同时可能诱发其发生骨质衰老变化,而玉米黄质可以延缓此类致老性骨质变化的发生。

Calculate the percentage composition of the two forms of D-galactose at equilibrium.

计算达到平衡时这两种形式的D-半乳糖的百分含量。

Galactose-induced expression did not affect the nucleosome positioning in the coding region of NPTII.

半乳糖诱导表达不影响核定位中的编码区NPTII 。

Recent research to α-galactosidase indicates that the enzyme has widely application prospect in fields of feedstuff、 refining sugar、 foodstuff and pharmaceutical industry, et al.

现今对α-半乳糖昔酶的研究表明,该酶在饲料、制糖、食品、医药工业等领域具有广泛的应用前景。

Objective To determine the distribution and influence of lysosomal neuraminidase, protective protein/cathepsin A and β-galactosidase in the inner ear of the mouse, and to observe their auditory alterations in enzyme deficiency.

目的:研究溶酶体神经氨酸酶(neuraminidase, Neul)、保护蛋白/组织蛋白酶A(protective protein/cathepsin A, PPCA)和β-半乳糖苷酶(β-galactosidase,β-gal)在小鼠内耳的分布及相互影响,观察各自酶缺乏时的听功能改变。

Wuhan Technology, the Group identified that the First Hospital of Wuhan City,"purslane water extract of anti-D-galactose-induced brain injury toxic mechanism of" scientific research, research and development for the aging, drug prevention and treatment of Alzheimer's offer A valuable scientific basis.

武汉市科技局专家组鉴定认为,武汉市第一医院的《马齿苋水提液抗D-半乳糖诱导的脑神经毒性损伤作用机制》科研成果,为研发延缓衰老、防治老年痴呆的新药提供了可贵的科学依据。

Study on Gene Cloning, Expression and Expression Profiling in Tissues of Polypeptide: N-acetylgalactosaminyl Transferase 2

多肽:N-乙酰氨基半乳糖转移酶2(pp-GalNAc-T2,E.C.2.4.1.4)的基因克隆、表达及组织表达谱研究

"The oligosaccharides used in the studies were a combination of galactose oligosaccharide and fructose oligosaccharide," said Sinn.

&低聚糖研究报告使用的是一个组合,半乳糖低聚糖和果糖低聚糖,说:&新。

Experimental Center of National Level One, Hubei Traditional Chinese Medica l College, Wuhan 430061)Abstract To investigate the protective effects of Jiaweisinisa n on acute liver injury induced by intraperitoneal injection of D-ga lac-tosamise, serum superoxide dismutase, malondialdehyde , glutathioe contents were measured. The pathological changes of liver wer e observed simultaneously. The results showed that JWSNS could decrease serum MD A content, but increase serum SOD and GSH activities in acute liver injury induc ed by D-GalN. It was also found that JWSNS could alleviate the degeneration and necrosis of the hypatocytes.

为研讨加味四逆散防治肝损伤的作用机理,通过动物试验,先用加味四逆散预防给药,再采用D-氨基半乳糖造成大鼠急性肝损伤模型,然后检测大鼠血清超氧化物岐化酶、丙二醛、还原型谷胱甘肽水平,同步观察该方对急性肝损伤大鼠体内脂质过氧化反应及肝脏病理改变的影响,结果表明该方可明显升高大鼠血清SOD、GSH水平,降低MDA水平,肝脏病理学检查亦表明该方使肝细胞变性坏死程度减轻。

This indicated that γ-crystalline mostly localized in lens nucleus,β-crystalline in cortex.

实验组皮质≤1,核≤3/4,说明障明滴眼液和抗白内障1号具有维持晶体蛋白自然组成的趋势,保持γ晶体蛋白的含量,也就是保持了晶体内的还原状态,从而抑制半乳糖对晶体的氧化损伤,防止白内障的发生发展。

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