升高

The changes of renin,aldosterone and blood preesure were observed before and after test.

结果 随着肾功能的恶化，高肾素型高血压的比例(5 6 %)逐步下降，低肾素型的比例明显升高(P 0 5 )。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，"PMN-EC"相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Cells were cultured for 6 days after the intervention, cell content of alkaline phosphatase decreased significantly over the blank control group, each group of Chinese Medicine groups compared with the control group increased in varying degrees, and with the increased dose alkaline phosphatase content moving in upward trend.

各组细胞干预6天后，对照组细胞内的碱性磷酸酶含量较空白组明显下降，而中药各组较对照组均有不同程度升高，且随着药量的加大碱性磷酸酶含量呈上升趋势。

Rapid alkalinization factors were polypeptide signals in the plant, belonging to a large gene family. The activity of RALF could induce extra-cellular pH increasing rapidly. Many studies on RALFs were in Solanum, including the structure and maturation, the gene expression, receptors and functions and so on.

快速碱化因子类基因属于植物多肽类信号分子，是一类大的家族基因，它的活动引起胞外pH值快速升高，有关RALF类基因的研究在几种茄科植物中开展的较多，包括RALF蛋白信号分子的结构及加工、基因表达、受体研究以及功能等方面。

It was observed that the Pt 4f binding energies in the same-sized Pt nanoclusters modified with alkylamine, alkylthiol, PVP, PVA or PPh〓 increase by 0.5-0.8eV relative to those of bulk Pt.

观察到具有相同金属尺寸的Pt金属内核而外层配体分别为长链胺、长链硫醇、PVP、PVA或PPh〓的纳米簇的Pt 4f电子结合能相对于块体Pt样品升高了0.5-0.8eV。

During the reaction of alkylation, general contained 0.2%~2.0% butadiene will produce viscous heavy composite materials, which will raise final boiling point of alkylate and decrease octane, what is more, it will react with acid to produce corresponding ester, which will increase the consumption of acid, Therefore, we must remove butadiene by selective hydrogenation, at the same time, it will convert some of 1- butane to 2- butane by isomerization and raise octane of alkylate.

一般含有0.2%~2.0%的丁二烯，其在烷基化反应中，可生成一种分子量很高的粘稠重质迭合物，使烷基化油的干点升高，辛烷值降低，并且它还和酸反应生成相应的酯而加大酸耗量。因此必须经过选择性加氢除掉丁二烯，同时，将部分1-丁烯异构化为2-丁烯，以提高烷基化油的辛烷值。

ALL-PRO OFFROAD's new coil-overs are adjustable from 0-3.5 of lift and exclusively feature Walker-Evans shocks, new upper shock mounts, and 650-lb coils.

ALL-PRO OFFROAD的新的减震延长套装是从0-3.5可调升高，包括WALKER-EVANS的减震器，新的减震上部安装座，和650磅的弹簧。

After being given therapy integrated Chinese and Western medicines,allopathy and nutritional treatment,all circadian parameters of the case came back in gear and discharged at last.

MODS的发生机制非常复杂，在严重感染、创伤、缺氧等打击下，失控的过度炎症反应激活多细胞因子和炎症介质的释放，使MODS发生率升高。

objective to raise the level of regarding congnition,prevention,diagnosis and therapy on multiple organ dysfunction syndrome.methods report the process and correlative therapy on one case who occured with mods involving 6 organs after hemorrhagic shock lead to heartbeat and respiration stopping suddenly and resuscitated resulting in left nephrostome titanium nip falled off accidentally from left kidney lymphatic fastening.at the same time,the author reviewed correlative literature and introduce the investigative progress about mods.results one case who happened with mods following hemorrhagic shock,heartbeat and respiration stopped suddenly was diagnozed and resuscitaed.6 organ dysfuncted including brain,heart,lung,liver,kidney and the digestive system.after being given therapy integrated chinese and western medicines,allopathy and nutritional treatment,all circadian parameters of the case came back in gear and discharged at last.conclusion the mechanism about mods is very complicated.because of serious infection,hurt and oxygen-lack,excessive inflammatery reaction activates multifarious cell factors and inflammatery mediums,which improve the happen-rate of mods.wiping off pathogeny,providing life-sustain treatment and recognising sirs and mods in time and adopting corresponding therapy,such as treatment integrated chinese and western medicines,can improve the hit-rate of cure on mods.

作者：王静恩，蔡金芳，王志华，谢晓洪多器官功能障碍综合征；中西医结合；治疗；休克，出血性目的提高对多器官功能障碍综合征（multiple organ dysfunction syndrome，mods）的认识、预防、诊断、治疗水平。方法报告成功抢救1例经腹腔镜左肾淋巴管结扎手术后左肾静脉钛夹意外脱落失血性休克，导致心跳呼吸骤停复苏后再手术并发mods累及6个脏器功能衰竭过程及有关治疗，并复习相关文献，介绍目前关于mods的研究进展。结果确诊1例因失血性休克心跳呼吸骤停复苏后并发mods，累及脑、心、肺、肝、肾、血液、胃肠等6个脏器。经积极采取中西医结合对症、支持救治，各生理指标全部恢复正常，康复出院。结论 mods的发生机制非常复杂，在严重感染、创伤、缺氧等打击下，失控的过度炎症反应激活多种细胞因子和炎症介质的释放，使mods发生率升高。尽早去除病因，尽早给予各种生命支持治疗，尽早对可能发生全身炎症反应综合征、mods识别并给予干预治疗，包括中西医结合治疗能改善mods的救治成功率。

The oyster extract has a significantly protective effect on increasing of blood glucose in model mice induced by alloxan.

牡蛎提取物对四氧嘧啶所致小鼠血糖升高有显著的保护作用。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。