刺激血管的

Many study indicated that VEGF is the most important and direct factor to stimulate vessel growth, and play an important role in neovascularization.

许多研究认为，血管内皮生长因子是目前发现的最重要、最直接的刺激血管生长因子，在新生血管的形成中起重要的作用。

Here, we have developed vascular endothelial growth factor high-expressing, transiently modified stem cells for the purposes of promoting angiogenesis.

一个关键的问题在于经过改造的细胞不能产生足够数量的刺激血管生长的关键蛋白——诸如血管内皮生长因子。

They stimulate endothelial cell proliferation and the growth of new blood ve els by releasing endothelial mitoge and chemotactic factors, such as bFGF and vascular endothelial growth factor .

它们能刺激内皮细胞的增殖和血管的生长，通过释放内皮有丝分裂和化学趋化因子，例如bFGF和血管内皮生长因子。

Many factors, such as vascular endothelia growth factor and fibroblast growth factor, can stimulate the formation of new vessels.

血管内皮生长因子、成纤维细胞生长因子等许多因素均可刺激新生血管的形成。

Recombinant hANG had no effect on the expression of VEGF and bFGF in both SMCC-7221 cells and HUVEC cells, suggested that hANG exerted its antiangiogenic role not through the inhibition of the autosecretion and parasecretion of angiogenic stimulatory factors VEGF and bFGF.

重组人血管抑素对SMMC-7721肝癌细胞及HUVEC细胞中两种血管生长刺激因子VEGF和bFGF的表达没有影响，提示血管抑素对肿瘤血管生长的抑制作用不是通过抑制血管生长刺激因子的自分泌和旁分泌来实现的。

The results showed that though testosterone or 17-β-estradiol did not affect the expression of these AM in unstimulated HUVEC, both of them transiently increased the expression of E-selectin and VCAM-1 in TNF-α-stimulated HUVEC.

近几年研究发现，粥样硬化与炎症及免疫反应有关，TNF-α等细胞因子可以通过刺激血管内皮细胞粘附分子的表达及血管平滑肌细胞的增殖等而在粥样硬化的形成中起重要作用。

Though the primary stimulator of 1H is not clear, it is related to physical, cell and body fluid IH is a chronic process involving multiple faction injury and blood flowing abnormal are revamped as the main reason, the proliferation level depend on the length and width of the injure of vessel large amount research illustrate injury cause many growth factor infolded in IH, for example: template derived growth factor, fibroblast growth factor, transmission growth factor , endothelin (ET-1) et al, some early responsive gene such as c-fos、c-jun c-myc also take part in SMC proliferation after vessel injury.

虽然IH的确切起始刺激因子并未阐明，但已知与物理、细胞和体液因子有关。IH是一个多因素参与的慢性过程，损伤和血液动力学异常一真被认为是IH的主要因素。内膜增殖的程度有赖于血管损伤的长度和深度。近年来大量的实验研究结果表明，损伤等刺激引起的众多的生长因子样物质在IH中起关键作用。如血小板源性生长因子、碱性纤维母细胞生长因子、转化生长因子β、内皮素-1（ET-1）等诸如C-fos、C-jun、C-myc等早期应答基因也参与了血管损伤后SMC的增殖。

Our experiment showed that addition of benzydamine induced vasorelaxation in the denuded aorta precontracted with KCl in the normal krebs.

实验结果亦显示，benzydamine能抑制高钾所引起的收缩作用，并且藉由在无钙之Krebs溶液中，经benzydamine前处理的血管给予高钾刺激之后，再给予钙离子刺激血管收缩，发现其能抑制外钙内流所造成的血管收缩作用。

To further characterize these activities, we developed the culture of bovine aortic endothelial cell and investigated the mitogenic activities of baFGF on these cells.

除了刺激3T3细胞的DNA合成外，FGF另外一些显著的生物学功能包括：刺激血管内皮细胞生长和促进神经元的存活及突起生长。

One medicine in development is a DNA therapy that stimulates the growth of blood vessels and is intended to reduce the need for amputations.

一种正在研发的药物是DNA疗法，它能刺激血管的增长，减少截肢的可能性。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。