分泌作用的

These results suggest that the activate spleen-energy method can achieve appetitive purpose by not only affecting the gastrointestinal tract function in periphery, but also regulating the process of ingestion in brain. The basic research about ingestion indicate: one hand, after food enters the gastro-intestinal tract, the endocrine cell of the intestinal tract is activated and secrete many kinds of brains intestines peptide, the level of the brain intestines peptide in blood as the periphery signal spreads to the central nervous system regions that control digest and feeding behavior. On the other hand, it has been shown that dorsal parabranchial neurons, containing CCK-8S, extend fibers to the VMH and are involved in the inhibition of feeding .

关于摄食控制的基础研究表明，一方面食物进入胃肠道后，激活肠道的内分泌细胞，分泌多种脑肠肽如CCK-8S，这些脑肠肽在血中的水平作为外周信号可通过"肠—脑轴"传入中枢神经系统介导消化和摄食行为的部位，影响摄食中枢神经元的活动调节动物的摄食行为；另一方面中枢神经元可合成释放内源性的神经肽直接作用于摄食中枢神经元调节摄食，如中枢鳃旁体神经元的神经纤维就可延伸到达VMH，并且合成释放内源性的CCK-8S。

The effects of FPL andits three ingredients on growth of ovary,on micro-and ultra-structure andAlkaline phosphatase activity of endometrium,on structure andkeratinization rate of exfoliated cells in vaginal epithelium in mice,onmicrocirculation in rat and on uterine kinesis in rabbit were respectivelyobserved by the techniques of light and electron microscopes,histochemistry,microcirculation and organ cinetography.The results have confirmed themechanism of FPL for the sterility caused by ovary standstill and persistentcorpus luteum to be as follows:(1) a weak estrogen-like role which canprompt development of ovary,proliferation and secretion of endometrium andvaginal epithelium and cornification of exfoliated cells in vaginal epithelium;(2)enhancing AKP's activity of endometrium;(3)improving microcirculation;(4) exciting uterus.

分别采用光学显微镜和电子显微镜技术、组织化学技术、微循环技术和器官运动描记等现代医学实验技术，观察了促孕液对小白鼠的卵巢发育、子宫内膜的显微和超微结构和碱性磷酸酶活性、阴道上皮结构和脱落细胞的角化率、对大白鼠的微循环和对家兔的子宫运动的影响，结果证实了促孕液治疗卵巢静止、持久黄体的机理：(1)微弱的雌激素样作用，能刺激卵泡发育，促进子宫内膜和阴道上皮增生、分泌和阴道上皮脱落细胞角化；(2)提高子宫内膜碱性磷酸酶的活性；(3)改善微循环；(4)促进子宫运动。

Itamin D from the skin and diet is metabolized in the lier to 25-hydroxyitamin D (Figure 1), which is used to determine a patient's itamin D status1,2,3,4; 25-hydroxyitamin D is metabolized in the kidneys by the enzyme 25-hydroxyitamin D-1-hydroxylase (CYP27B1) to its actie form, 1,25-dihydroxyitamin D.1,2,3,4 The renal production of 1,25-dihydroxyitamin D is tightly regulated by plasma parathyroid hormone leels and serum calcium and phosphorus leels.1,2,3,4 Fibroblast growth factor 23, secreted from the bone, causes the sodium–phosphate cotransporter to be internalized by the cells of the kidney and small intestine and also suppresses 1,25-dihydroxyitamin D synthesis.5 The efficiency of the absorption of renal calcium and of intestinal calcium and phosphorus is increased in the presence of 1,25-dihydroxyitamin D (Figure 1).2,3,6 It also induces the expression of the enzyme 25-hydroxyitamin D-24-hydroxylase (CYP24), which catabolizes both 25-hydroxyitamin D and 1,25-dihydroxyitamin D into biologically inactie, water-soluble calcitroic acid.2,3,4

从皮肤和食物来的维生素D在肝中代谢为25-羟基维生素D（图1），被用来决定病人体内维生素D情况的1，2，3，4；25-羟基维生素D在肾中被25-羟基维生素D1羟化酶（CYP27B1）转变为有活性的1，25-二羟基维生素D 。1，2，3，4由肾产生1，25-二羟基维生素D是被血浆甲状旁腺激素和血清钙，磷水平紧密调节。1，2，3，4由骨分泌的成纤维细胞生长因子23使钠磷协同转运蛋白被肾和小肠细胞内化及抑制1，25-二羟维生素D合成。5 在1，25-二羟基维生素D作用下肾和小肠吸收钙及磷的效率增高(图1)。2，3，6 它也包括25-羟四- 24 -羟化酶的表达(CYP24)，且将1，25二羟基维生素D和25羟基维生素D异化成无生物活性，水溶性的维生素D3-23羧酸。2，3，4

Soya Bean Polypeptide that relies on rapid activities of H+ ion exchange in the gastro intestine dispossesses the intestinal passage, shortening fat's duration of stay in the intestine, particularly blocking the major absorption of cholesterol before speedily excreted. Meanwhile, part of theSoya Polypeptide stimulates squeezing pressure on the bile to boost secretion that breaks down the fat in diminishing the formation of triglyceride in the liver, bringing along the decreased appetite.

大豆多肽—依据H＋离子在胃肠的快速运转，抢占脂肪在胃肠的吸收通路，减少脂肪在胃肠的停留，特别是阻止胆固醇在大肠的重吸收，并使之迅速排除体外，大豆多肽的部分片段可以促进胆囊收缩增加胆汁分泌，促进脂肪分解，减少甘油三酯在肝脏的合成，也有降低食欲的作用。

Clinical Study on treatment of prostatic carcinoma high Intensity hocused ultrasound combined with hormone in the;2. To evaluate the roles and mechanism of IGF-2 on fetal growth and development during late gestation, pregnant ewes and rats were employed to investigate relationship between IGF-2 and metabolites, hormones of pregnant animals and their fetuses.

本文以绵羊、大鼠为实验对象，观测围产期妊娠母羊、胎儿和新生儿血浆IGF-2水平的动态变化及其与其它内分泌激素和代谢物水平之间的相关性，并研究了给予外源性IGF-2片段、葡萄糖、胰岛素和皮质醇对血浆IGF-2、胰岛素和代谢物水平影响，综合分析IGF-2在动物胚胎、新生儿生长发育中的作用，着重探讨IGF-2与营养代谢和内分泌之间的关系以及分泌的调节因素。

This year, his group showed that EDR1's role is to weaken a plant alarm signal called salicylic acid, a close chemical relative of aspirin that is produced by cells in diseased parts of the plant and orchestrates the plant's defences against the infection.

今年，他的小组显示，edr1的作用在于削弱一种称为水杨酸的植物预警信号，水杨酸是阿斯匹林的化学近亲，由植物罹病部位的细胞分泌，负责组织植物抵抗感染。

Water can make the redox inside body is able to run, in order to produce the quantity of heat that body place needs, nutriment, it is the haemal circulation with be maintained the temperature, balance that maintains the electrolyte inside body, normal, OK still that water can adjust make digestive gland secretes digestive juice to promote food to digest absorb, the phenomenon that reachs precaution to improve constipation, water still can make your skin water bright water bright, advantage is great.

水可以使体内的氧化还原作用得以运作，以产生身体所需的热量、营养素，水可以调节体温、维持体内电解质的平衡、维持正常的血液循环、还可以使消化腺分泌消化液促进食物消化吸收、及预防改善便秘的现象，水还可以使你的皮肤水亮水亮，好处多多。

One year later, Slit was confirmed as the ligand of Robo and the Slit-Robo signaling is know involved in the repulsive effect of the commission axons crossing the mid-line, and also involved in the repulsive effect of the guidance of neuronal migration and leukocyte migration as well.

而对 Slit 功能展开的研究起于1998年，先是发现其受体 Robo突变型果蝇的中线处交叉神经元的导向发生缺陷。1999年的一系列遗传和生化的研究又发现，分泌性因子 Slit 正是 Robo 的配体， Slit － Robo 信号起到了排斥一些轴突，使之无法穿过中线的作用。

Results: 1 Tritiated thymidine incorporation assay showed that 1μM adenosine plus coformycin slightly inhibited CTLL-2 proliferation, while 5-20μM adenosine elicited marked inhibition on CTLL-2 proliferation responded to IL-2 a dose-dependent fashion. 2 Coformycin alone could also inhibit CTLL-2 proliferation at the concentration of 2.5μM. The underlying reason maybe due to the endogenous adenosine produced by CTLL-2 cells.

结果：11μM腺苷对CTLL-2增殖仅有轻微的抑制作用，而5、10、20μM腺苷的存在可显著抑制CTLL-2的增殖，并且呈剂量相关，P值均小于0.05.2CTLL-2经过2.5μM助间型霉素的单独处理也可引起轻度的增殖受抑现象，分析其可能原因是T细胞自身可以分泌腺苷，所以该抑制现象的出现与内源性腺苷有关。3本实验所用腺苷浓度对CTLL-2的活细胞百分率影响轻微。

In the first cultivation stage,as relatively high concentration of phosphorus,phosphorus from water column was removed to a certain extent by periphyton.The end of the incubation,as low concentration of phosphorus periphyton releases phosphorus to water column instead of removal phosphorus from water,this may closely relate to the alkaline phosphatase secreted by periphyton in water body.

从培养过程中水体磷的浓度变化来说，培养初期，水体中磷的浓度比较高，附着生物对其有一定的去除作用；培养后期，水柱中磷的浓度比较低，附着生物不但不去除水中的磷，而且还向水柱中释放磷，这可能与附着生物向水体分泌碱性磷酸酶密切相关。

Plunder melds and run with this jewel!

掠夺melds和运行与此宝石！

My dream is to be a crazy growing tree and extend at the edge between the city and the forest.

此刻，也许正是在通往天国的路上，我体验着这白色的晕旋。