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Objective To study the resistant development of Rattus norvegicus to anticoagulant rodenticide after its application in Qichun county.

目的 探讨蕲春县应用抗凝血灭鼠剂以来,褐家鼠对抗凝血灭鼠剂的抗药性变化。

To adopt system menstruum deal with fermented products of mycelium from Tremella aurantialba, then mycelium from tremella aurantialba was studied with the clotting time of the thrombogen, the whole blood clotting time in mice and thrombosis experiment in mice.

采用系统溶剂法处理金耳菌丝发酵物,得乙酸乙酯部位、正丁醇部位及水溶部位,通过凝血酶原实验、小鼠凝血实验及小鼠体内血栓形成实验等来筛选具有抗凝血作用的有效部位。

Methods The changes of prothrombin time activated partial thromboplastin time, thrombin time, and fibrinogen content were determined by automated coagulation analyzer in 65 patients with liver cancer before treatment and in the 1, 3, 5, 15 and 30 days after intervention treatment.

采用全自动血凝分析仪检测65例肝癌患者介入治疗前后第1、3、5、15、30天的凝血酶原时间、活化部分凝血活酶时间、凝血酶时间、纤维蛋白原含量变化。

Tea polysaccharide can prolong activated partial thromboplastin time of human plasma significantly,but has no effects on prothrombin time and thrombin time.

茶叶多糖能明显延长人体血浆APTT值,而对PT、TT值的影响不显著,表明茶叶多糖主要通过影响内源性凝血系统而发挥抗凝血作用。

Tissue factor is an initiator of extrinsic route of blood coagulation,and causes thrombosis through blood coagulative activation.

组织因子是外源性凝血途径的启动因子,通过介导凝血激活促进血栓形成。

MOF was sequentially found in the digestivetract,central nervous system,kidneys,coagulatory andfibrolytic system,heart,endocrine and metabolism,andlungs;Mortality was sequentially due to the failure of thelungs...

各器官衰竭的发生率序次是消化道、中枢神经系统、肾、凝血和纤溶系统、心、内分泌及代谢、肺;病死率序次是肺、肾、中枢神经系统、凝血和纤溶系统、内分泌及代谢、心脏和消化道(P<0.05)。

Random selected 60 patients ,then they were admitted for surgery treatment of Selective Decongestive Devascularization Shunt of Gastrosplenic Region and Splenectomy and Periesophagogastric Devascularization . Base on the difference of coagulation function, they were divided into two groups: Group A: The anticoagulated group ,29 patients , were administered Low Molecular Weight Heparin and Salvia Miltiorrhiza injection, Group B: The contral group, 31patients, were administered for the conventional treatment.

随机选取对肝硬化门静脉高压症实施选择性脾胃区减断分流术和脾切除加贲门周围血管离断术的60例病人,将其依据凝血功能分为A组:抗凝组29例(应用低分子右旋糖苷联合复方丹参注射液等进行抗凝治疗),B组:对照组31例,未行抗凝治疗。

Multiple factors participated in the process of secondary brain edema and nerve injuries,such as the hydrostatic pressure during formation of the hematoma and clot retraction, activation of blood coagulation cascade, generation of thrombin,erythrocytolysis and the toxicity of hemoglobin, the activation of the complement system, space-occupying effects, rupture of the blood-brain barrier,secondary ischemia around the hematoma, inflammatory reaction of the neurons around hematoma and cell apoptosis.

脑出血后继发水肿及神经损害有多种因素参与,血肿形成过程中的流体静力压和血凝块的回缩、凝血瀑布的激活、凝血酶的产生、红细胞溶解和血红蛋白的毒性、补体的激活、占位效应、血脑屏障的破坏、血肿周围继发缺血、血肿周围组织神经细胞炎症反应及细胞凋亡等。

Results:The etiologic mechanism of steroid-induced necrosis of femoral head is:Disorder of lipometabolism;increasing of intraosseous pressure;Osteoporosis and poisonous action;vasculitis,coagulation before steroid administration and intrava...

结果 :激素性股骨头缺血坏死存在如下发病机理:脂肪代谢紊乱;骨内压增加;骨质疏松及激素的毒性作用;血管炎、前凝血状况及血管内凝血。

Results:The etiologic mechanism of steroid-induced necrosis of femoral head is:Disorder of lipometabolism;increasing of intraosseous pressure;Osteoporosis and poisonous action;vasculitis,coagulation before steroid administration and intravascular coagulation.

结果:激素性股骨头缺血坏死存在如下发病机理:脂肪代谢紊乱;骨内压增加;骨质疏松及激素的毒性作用;血管炎、前凝血状况及血管内凝血。

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