内脏痛

Results showed:(1) The number of ChAc-IR neurons (62. 25±1. 45) in the group of visceral pain increased obviously. The positive neurons (46. 38±1. 27) in the group of electroacupuncture and visceral pain were lower than those of VP (p.05). These indicate that there were changes in cholinergic neurons' activity, increasing of ChAc function and turbulence of intestinal motility in rat's ileum following the acute colonitis.

结果发现：（1）内脏痛组ChAc-IR神经元（62.25±1.45）明显增高，电针+内脏痛组阳性神经元（46.38±1.27）明显低于内脏痛组，二组之间存在显著差异（p.05），提示急性结肠炎可致继发性回肠内在胆碱能神经元活动的改变，ChAc活性增高，肠动力紊乱，而电针可抑制这一改变。

Therefore, one of the important mechanisms suppressing visceral pain by acupuncture is the regulation on ENS functional condition.

因此，针刺抗内脏痛的重要机制之一是调整肠神经系统的功能状态。

The model was established by injection 100μL of 5% formalin into sigmoid colon of rats.

给大鼠乙状结肠注射5%的福尔马林100μL制作急性结肠炎引发内脏痛大鼠模型。

Visceral pain is characterized by spontaneous pain and referred hyperalgesia.

内脏痛是以自发性疼痛和痛觉增敏为特征。

Aim To study the distribution regulation of primary neurons and sympathetic neurons of skin associated with viscera on the theoretical basis of treating visceral pain by intracutaneous- injection drug.

目的在皮内注药治疗遗尿症及内脏痛有效的基础上，研究皮肤与内脏相关的初级神经元和交感神经元分布规律。

The distribution characteristics of primary neurons in skin referral area and gastric mucosa are that spinal ganglions present segmental distribution and superposing with each other,moreover,sympathetic ganglions present diffused distribution and have no segmental distribution,which may be the neural basis of treating visceral pain by intracutaneous- injection drug.

结论胃痛的皮肤牵涉区与胃黏膜初级神经元分布的特点是在脊神经节呈节段性分布且相互重叠，在交感神经节呈弥散性分布，无节段性分布，也相互重叠，这可能是皮内注药治疗内脏痛的神经基础。

The mechanism of JPJDHZF may be increasing the limen of pain of bowel, eliminating hypersusceptibility of bowel, and decreasing excitability of nerve cells by decreasing the contents of 5-HT and SP in IBS model rats.

健脾解毒化浊方的作用机制可能是通过降低模型大鼠血清5-HT、血浆SP含量，减弱背角神经元兴奋性，提高内脏痛阈，消除肠道过敏，从而达到治疗目的。

There are ENS changes in the secondary visceral pain following an acute colonitis and electroacupuncture analgesia can make the changes improved, internal environment stabilized and the turbulence of intestinal motility resumed.

在急性结肠炎继发内脏痛时可出现肠神经系统的改变，电针镇痛使这些变化得到改善以利于内环境的稳定，肠动力紊乱得到纠正。

AIM To study the role of N-methyl-D-aspartic acid NMDA and non-NMDA receptors in processing nociceptive visceral information in the spinal cord.

目的 研究NMDA N-methyl-D-aspartic acid和非NMDA受体在介导脊髓内脏痛传入中的作用。

The expression of GFAP in ILN was significant at 60 mm and it decreased obviously at 120 min after animal model induced. Conclusion Behavior changes in rats are mediated by visceralgia, which induces acute colitis. The change of GFAP in gliocyte, which is closely related with pain, emerged early and remarkably. The change of ILN, which is intensely related with emotion, appears lain.

大鼠痛行为学的变化是由于急性炎症引起的内脏痛而导致，与疼痛变化密切相关的胶质细胞中GFAP在脊髓内反应早且明显，而在与情绪变化密切相关的丘脑内的板内核群中变化相对较晚。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。