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After over expression of EL, the bionomics of endothelial cells is changed, and these changes may participate the pathophysiology of atherosclerosis.

内皮细胞高表达EL后对内皮细胞自身的生物学活性有影响,这可能与动脉粥样硬化的病理生理过程有关。

After over expression of EL, the bionomics of endothelial ce1ls is changed, and these changes may participate the pathophysiology of atherosclerosis.

内皮细胞高表达EL后对内皮细胞自身的生物学活性有影响,这可能与动脉粥样硬化的病理生理过程有关。

LPS-induced ICAM- 1 expression of endothelial cells possessed time- dependent and dose-dependent characteristics, and tolerated to LPS chronical stimulation.

LPS刺激和内皮细胞表面ICAM-1的表达具有一定的时间和剂量依赖性,内皮细胞ICAM-1的表达对LPS长期刺激可产生耐受性。

The rate of c-kit+ cells in the heart increased gradually from E10 with the highest level of 12.6±3.2% at E11, which was nearly similar to the fetal liver, except for E12 when the c-kit+ cells was 3.4±1.2% in the heart compared to the fetal liver with 11.6±4.1% c-kit+ cells. The rate of c-kit cells in the suspension cells from the heart at E11 maintained stable after 24h culture based in the stromal cells of the heart and AGM region with the same conceptus age, but the rate of c-kit cells obviously decreased in the condition of the stromal cells from the fetal liver. The c-kit cells of the three conditions were all at low level at 48h without significant difference.

通过流式检测c-kit+细胞我们发现,胚胎期心脏和胎肝c-kit+细胞比率几乎一致,E11时c-kit+细胞明显增多,分别高达(12.6±3.2)%和(9.6±2.8)%,但E12时心脏中c-kit+细胞明显减少,仅为(3.4±1.2)%,此时胎肝中c-kit+细胞为(11.6±4.1)%。E11的悬浮细胞与心脏内皮细胞和AGM区基质共培养24h时,c-kit+细胞数量保持稳定,但在胎肝基质细胞中c-kit+细胞数量明显减少,在共培养48h,三组中的c-kit+细胞均呈较低水平,组间没有显著性差异。

In this study, we investigated the migration of Daudi cells across confluent monolayers of the human vascular endothelium cells.

为此,我们观察了CD40分子激发对Daudi细胞与血管内皮细胞的相互作用及其穿越血管内皮单细胞层的影响。

Results: radiation-induced depolymerization of F-actin microfilament stress fibres was dependent on the radiation dose.

电离辐射对血管内皮细胞骨架蛋白F-actin的破坏可能是血管内皮细胞辐射损伤机体机理之一。

While the'endothelial cells were cultured in the media containing CGRP, the expression of ICAM-1 significantly diminished and the expression of NOS and TIMP-2 raised, especially in ECs induced by diamide.

当CGRP作用于正常内皮细胞和联胺诱导启的内皮细胞,对ICAM-1的表达有明显的下调作用,对NOS和T1MP-2的表达则有明显的上调作用。

This is a normal signal from normal native endothelial cells. This is the signal from the regenerated endothelium.

这是从正常的原有的内皮细胞得到的信号产物,这是是从再生内皮细胞上得到的信号产物。

Here in the presence of the endothelium, protection here, no protection there.

在有内皮细胞存在的部位,原生内皮细胞对血管有保护作用,而再生的则没有。

AIM: To study the effects of probucol on injured endothelial cells and regenerated endothelium.

目的:研究普罗布考对受损内皮细胞增殖和再生内皮细胞功能修复的影响。

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然而,正如其名字所指出的那样,CD盘不能写,也不能用任何方式改变其内容。

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