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We hypothesized that endothelial cells may play an important role in cholesterol efflux from cells by transformating it to oxysterols, which can prevent the lipid aggradation into the tissue under endothelial cells.

如果血管内皮细胞可将过多的胆固醇等脂类物质由细胞内排出,可阻止过多的脂质进一步向内皮下沉积,进而阻止或减缓动脉粥样硬化的发生和发展。

The effect of alizarin and acetylcholine on endothelial cell release of nitric oxide in human umbilical vein was also observed.

通过在培养的人脐静脉内皮细胞中的研究表明,Alz可促使内皮细胞释放一氧化氮,其作用比乙酰胆碱更为显著。

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加;半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程;胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用;胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

One hundred seventy-seven eyes of 161 patients consecutive who had corneal edema resulting from Fuchs endothelial dystrophy, pseudophakic bullous keratopathy, aphakic bullous keratopathy, failed graft or iridocorneal endothelial syndrome.

连续的161位患者的177只眼,都有角膜水肿。病因分别为FUCHS角膜内皮营养不良,人工晶体眼大泡性角膜病变,无晶体眼大泡性角膜病变,移植术失败和虹膜角膜内皮综合征。

Correlation between corneal neo vascularization and the expression of VEGF in cornea and aqueous humor of rabbits

血管内皮祖细胞起源于骨髓,对于缺血组织的血管新生和损伤血管的复内皮化起着很重要的作用。

The disobviousness of cell loss in clinical studies may be due to the protective action of similar factors in aqueous humor.

临床上未能检测到内皮细胞缺失,可能是由于房水中存在保护内皮细胞损伤的因子。

Objective: To study the effect of endothelial lipase on the bionomics of human umbilical vein endothelial cell.

目的:探讨内皮脂肪酶对人脐静脉内皮细胞生物学特性的影响。

After over expression of EL, the bionomics of endothelial cells is changed, and these changes may participate the pathophysiology of atherosclerosis.

内皮细胞高表达EL后对内皮细胞自身的生物学活性有影响,这可能与动脉粥样硬化的病理生理过程有关。

After over expression of EL, the bionomics of endothelial ce1ls is changed, and these changes may participate the pathophysiology of atherosclerosis.

内皮细胞高表达EL后对内皮细胞自身的生物学活性有影响,这可能与动脉粥样硬化的病理生理过程有关。

Corn bract can regulate the level of NO and ET, reduce endothelial apoptosis rate and promote endothelial repair.

玉米苞叶煎剂可调节高脂大鼠血清NO和ET水平,降低内皮凋亡率,促进内皮修复。

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