内皮

Inhibition of early steps of platelet adhesion to the ischemic endothelium and the subendothelial matrix may offer a novel and safe treatment strategy in acute stroke.

抑制缺血性内皮组织和内皮下基质的血小板粘附的早期步骤，是一种治疗急性卒中的新颖且安全的治疗策略。

By investigating the regeneration feasibility of mesenchymal stem cells,which is derived from endothelial and subendothelial cells of human umbilical vein,to neuron-like cells in vitro,we want to provide new evidence for the transplantation of hUV-derived MSCs to nervous tissue.

探讨来源于人脐静脉内皮及内皮下分离的间充质干细胞向神经元样细胞分化的可能性，以期为脐静脉MSCs的神经移植提供理论依据。

In rat,no matter the young or old,there is only endotheliumin the tunica intima,there are no subendothelial layer and internal elastic membranes.

而在大鼠冠脉不论幼年或老年，其内膜仅由内皮构成，无内弹性膜和内皮下层，因而从幼年至老年始终没有内膜增厚和管腔变窄现象。

Results After 2 or 6 hours of LPS intravenous administration, severe lung injury and positive signals of NT occurred with the latter located in the pulmonary macrophages, endothelia, subendothelial and muscular layers of the pulmonary artery.

结果 LPS静脉注射2 h和6 h 后，肺脏有明显损伤及硝基酪氨酸产生的阳性信号，该信号主要分布于肺泡壁巨噬细胞、肺小血管和细小支气管周围的巨噬细胞以及部分肺动脉的内皮细胞、内皮下层及肌层。

There were significant differences between all the experimental groups and the control group (F=193.25, P＜0.01). Conclusions IL-1β and TNF-α induce a strong expression of MCP-1 mRNA and protein in HUVECs. Both cytokines may be involved in the atherogenesis by inducing the liberation of MCP-1 by endothelial cells and increasing the recruitment of monocytes into the subendothelial space.

IL-1β和TNFα均能显著增强内皮细胞MCP-1 mRNA和蛋白的表达，提示它们通过诱导内皮细胞产生MCP-1，吸引单核细胞迁入动脉内膜而参与动脉粥样硬化的发生和发展。

In aged,the internal and external elastic membranes both disappeared,the subendothelial layer is evidently thickened,in which there are some smallblood vessels and local calcification can be seen occasionally.

而在大鼠冠脉不论幼年或老年，其内膜仅由内皮构成，无内弹性膜和内皮下层，因而从幼年至老年始终没有内膜增厚和管腔变窄现象。

VEGF can encourage vascular endothelial cell to divise、propagate、migrate, and add vasopermeability, facilitate producing vascellum sustentaculum, to profit blood supply in ischemic region.

血管内皮生长因子能促进微血管内皮细胞分裂、增殖、迁移，增加血管通透性，促进血管支持物的生成，有利于缺血区血液的供应。

The authors hypothesize that, in endothelial cells, decreased expression of CADM1 interacts with protein C deficiency to impair the activated protein-mediated endothelial barrier protection in response to thrombogenic stimuli like thrombin generation, thereby increasing the risk of venous thromboembolism.

我们推测，由于血液的蛋白C缺陷与内皮细胞的细胞粘附分子1缺陷的相互影响，从而损坏了内皮的屏障功能，最后增加了静脉血栓形成的风险，这样可以解释以往很少被人理解的魏克氏三特征的第三个因素：血管壁的受损。

To provide the relevant experiment data to the current understanding of basic mechanisms that explain the signal transudation pathway occurring inside the endothelial cells under the effect of SS, we use flowing chamber method to exert SS on Rat Brain Microvascular Endothelial Cells.

为探索剪切力对内皮细胞作用的信号传导途径，以鼠脑微血管内皮细胞为研究对象，运用荧光染色、免疫组化染色等定性定量方法辅以计算机图象处理，对微血管骨架结构和黏附分子ICAM-1在剪切力作用下的变化规律进行了研究。

At 4 wk, the cells in the liver bud migrated into the domain of transversum mesenchyme, where endothelial cells began to form vascular structures, and branched to form a number of hepatic cords.

结果：3wk时靠近肝芽的间充质中的血管内皮细胞多，而肝芽内未见血管内皮细胞及造血细胞：4wk时，肝索形成处可见较多正在形成的新生血管增多，肝索间开始出现少量的造血细胞：5wk时肝索间出现原始的肝血窦结构，血窦内造血细胞的数量较4wk明显增多。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新