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ABSTRACT The present study was performed to explore the effect of endogenous nitric oxide on synaptic transmission between neurons located in Forel′s field H and vertical oculomotoneurons innervating the inferior oblique muscle in cats.

本研究探讨内源性一氧化氮对猫中脑。间脑结合部Forel′s field H区神经元与下斜肌(inferior oblique, IO)眼球运动神经元(oculomotoneurons, OMNs)之间兴奋性突触传递的影响。

The mechanism of JPJDHZF may be increasing the limen of pain of bowel, eliminating hypersusceptibility of bowel, and decreasing excitability of nerve cells by decreasing the contents of 5-HT and SP in IBS model rats.

健脾解毒化浊方的作用机制可能是通过降低模型大鼠血清5-HT、血浆SP含量,减弱背角神经元兴奋性,提高内脏痛阈,消除肠道过敏,从而达到治疗目的。

These conditions are distinguished by the presence of clinical and electrical myotonia and a delay in muscle relaxation after contraction because of prolonged excitation of the muscle membrane.

肌强直综合征可以通过以下表现加以鉴别:临床检查及电生理检测军发现肌强直、因肌膜兴奋性延长所致的肌肉收缩后松弛延时缓慢。

High intensity, long duration and repeated high frequency electric stimulation will degrade the excitability of nerve fiber.

高强度、长时间、快速反复的电刺激将使神经纤维兴奋性降低。

The results showed that GABA was the excitatory transmitter of presynaptic inhibition.

以上结果表明:GABA是突触前抑制环路中,最后一级突触的兴奋性递质。

According to the conduction velocity of afferent fibers and stimulus threshold, evoked EPSCs that are mediated by A-like or C-like fibers were distinguished.

以传入纤维的传导速度与刺激阈值为指标,可以区分A 样纤维与C 样纤维兴奋性突触后电流。

This study demonstrates that the degrade excitability of dynamoneure may be internal mechanism of electric acupuncture cure hemiparalysis of spasmodic limbs after cerebral infarction.

降低脑梗死后脊髓α运动神经元兴奋性可能是电针治疗脑卒中后偏瘫肢体痉挛的内在机制之一。

Little is known of excitatory effects taking place in the central nervous system during this procedure.

很少有人知道发生在中枢神经系统中的兴奋性效应。

BACKGROUND: Excitatory amino acids play an important role in morphine tolerance.

背景:兴奋性氨基酸在吗啡耐受产生过程中起着重要的作用。

Glutamate is the major fast excitatory transmitter in the central nervous system.

谷氨酸是中枢神经系统主要的快速兴奋性递质。

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