关节炎

The osteoarthritis,also called recession arthritis,osteoarthropathy,are the synovial membrane joint are accompanied by around the joint the ossein proliferation to lose the result disease for the characteristic cartilage,assumes the chronicity, carries on the change.

骨关节炎，亦称退行性关节炎、骨关节病，是滑膜关节伴有关节周围骨质增生为特点的软骨丧失所致疾病，呈慢性、进行性改变。

CD40/CD40L interactions were also shown to play a significant role in the maintenance of autoimmunity,High-level expression of CD40L and CD40have been detected on monocytes/macrophages from patients with active sgstemic lupus erythematosus,rheumatoid arthritis and other autoimmune diseases,indicating that the expression of CD40/CD40L on monocytes/macrophages could account for the formation of synovitis and osteoclasia.

CD40/CD40L是免疫细胞间的重要信号转导途径，单核巨噬细胞表达CD40与CD40L，分泌许多促炎性因子，参与类风湿关节炎滑膜炎形成、骨和软骨破坏。在系统性红斑狼疮、类风湿关节炎和其他自身免疫病中已检测出高水平表达的CD40与CD40L。

ABSTRACT Osteoarthritis is a common degenerative arthritis disease with articular cartilage damage, and the main pathologic character is degeneration of cartilage, osteophyma production, cellular proliferation, synovitis and narrow joint space.

骨关节炎(Osteoarthritis，OA)又称退行性关节炎，是以关节软骨损坏为特征的一种慢性进行性关节疾患，其主要病理特征是关节软骨的变性破坏和丢失，。。。

Hyperuricemia and acute gouty arthritis model was made with Oxonic Acid potassium and Sodium Urate injected into abdominal cavity and right ankle joint cavity respectively.

目的研究痛风速效灵对急性痛风性关节炎模型大鼠不同时相步态和关节组织形态学的影响，探讨其治疗急性痛风性关节炎的作用机制。

Objective To explore the effect of total glucosides of paeonia on the serum levels of NO and IL-1β in patients with knee osteoarthritis.

骨性关节炎(osteoarthritis，OA)是一种多发生在老年人群的骨关节疾病，其中以膝关节的骨性关节炎发生率最高。

In histopathologic examine, pannus was infiltrating the articular cartilage during 10 to 31 days after injection and monocuclear leukocyte infiltration was found.

实验性化脓性关节炎早期关节软骨的MRI改变是单纯的信号改变，化脓性关节炎后期软骨形态开始发生变化。

The human cartilages are composed of chondrocyte and extracellular matrix,the form of chondrocytes are hypertrophy and the quantity are less;the ECM of cartilage are compised of type II collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury,the ommilayer injury ofcartilage can be recovered by marrow,but because of without stimulation mechanism,the new tissues are merely fibrocartilages,they can not be coincide with hyaline cartilage in menchanics;the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation,thus,the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.Ever since,people tried their best to find a way to reconstruct articular cartilage.

造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte.

造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

The human cartilages are composed of chondrocyte and extracellular matrix , the form of chondrocytes are hypertrophy and the quantity are less; the ECM of cartilage are compised of type Ⅱ collagen and proteoglycan. Articular cartilages are all hyaline with little fibers. Trauma and arthritis are the main cause of cartilage injury, the ommilayer injury ofcartilage can be recovered by marrow, but because of without stimulation mechanism, the new tissues are merely fibrocartilages, they can not be coincide with hyaline cartilage in menchanics; the purely damage of articular cartilage can not stimulate chondrocyte to regenerate because of without blood circulation, thus, the plerosis of articular catilage can not depend on the proliferation of local chondrocyte. Ever since, people tried their best to find a way to reconstruct articular cartilage.

中文题名人骨髓基质干细胞成软骨诱导及多孔复合材料作为细胞载体的体外实验研究副题名外文题名 Cartilage induction of human mesenchymal stem cells and experiment on compound porous materials as cells' scaffold in vitro 论文作者刘晓岚导师周江南学科专业外科学研究领域\研究方向学位级别博士学位授予单位中南大学学位授予日期2003 论文页码总数68页关键词骨组织工程软骨细胞骨髓基质干细胞壳聚糖高分子外消旋聚乳酸馆藏号BSLW /2003 /R68 /10 造成人体关节软骨损伤的原因主要为创伤和关节炎，关节软骨全层损伤可由于骨髓中间充质干细胞的高速增殖修复，但这种修复由于缺乏相应的刺激机制，只能形成纤维软骨，而不能形成符合关节生理、力学要求的透明软骨；单纯软骨部分损伤软骨组织内无血管，软骨细胞迁移迟缓，无法使损伤区域软骨细胞再生，因此，关节炎及关节创伤后的软骨修复不能依赖于软骨细胞的增殖和迁移。

Objective: To analyze the clinical characteristics of diagnosis and therapy of pyogenic sacroiliitis. Methods: 4 cases were analyzed retrospectively and reported; and related literatures were reviewed.

目的：回顾分析4例误诊为强直性脊柱炎的化脓性骶髂关节炎的病例，探讨化脓性骶髂关节炎的临床表现、诊断、治疗方法。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。