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共济失调

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Detrusor-external sphincter dyssynergia is a common cause of bladder outlet obstruction in SCI patients and associated with many urologic complications.

逼尿肌外括约肌共济失调是脊髓损伤患者常见的膀胱功能障碍,且是造成这些病患后续泌尿系统并发症的主要原因。

The therapy effect,change of liver function,time of excitation and dystaxia disappearance,and time of consciousness awakening were detected.

比较两组的临床疗效和兴奋及共济失调症状消失时间、神志清醒时间以及肝功能损害情况。

Results Of the cases, 23 were cured, 1 spontaneously ceased treatment and was discharged, 1 had dystaxia and 1 tottered (recovered 2 months after follow-up treatment).

结果 23例治愈出院,1例未坚持治疗自动出院,1例遗留共济失调,1例走路不稳(后随诊2个月恢复正常)。

Results Of the cases, 23 were cured, 1 spontaneously ceased treatment and was discharged, 1 had dystaxia and 1 tottered (recovered 2 months after follow-up treatment).

结果 23例治愈出院,1例未坚持治疗自动出院,1例遗留共济失调, 1例走路不稳后随诊2个月恢

NS syndrome:dystaxia, spastic paralysis,deafness and mental disability, but normal stature,normal or milder decreased function of thyroid gland.

神经性综合征:表现为共济失调、痉挛性瘫痪、耳聋和智能低下为特征,但身材正常、甲状腺功能正常或轻度减低。

Objective To construct spinocerebellar ataxia 3 (SCA3) eukarotic expression vector with en hanced green fluorescent protein as report gene and to observe its expression in PC12 cells.

目的 构建以增强型绿色荧光蛋白作为报告基因、带有脊髓小脑性共济失调3型(SCA3)基因的真核细胞表达载体,并转染PC12细胞,观察外源基因的表达情况。

Results The clinical manifestations of TOBS comprised transient unconsciousness, vertigo, dyskinesia of eyeball, abnormality of pupils, dyskinesia of limbs, hemianopsia or cortical blindness, ataxia and memory impairment. MRI showed the local infarction in thalami, cerebellum, midbrain, pons, occipital lobe, temporal inner surface. The features of DSA were occlusion or stenosis in basilar artery and vertebral artery.

结果 TOBS的临床主要表现为一过性意识障碍、眩晕、眼球运动障碍及瞳孔的变化、肢体运动障碍、偏盲或皮质盲、共济失调及记忆功能损害;磁共振显示梗死灶在丘脑、小脑、中脑、脑桥、枕叶、颞叶内侧面;数字减影血管造影显示为基底动脉及椎动脉闭塞或狭窄。

The former group showed severe sensory ataxia and predominantly large myelinated fiber loss in the sural nerve.

前一组表现为严重的感觉性共济失调,主要是大的有髓纤维缺失,特别是在腓肠神经。

Affected individuals may present with sleep disturbances, personality changes, ATAXIA; APHASIA, visual loss, weakness, muscle atrophy, MYOCLONUS, progressive dementia, and death within one year of disease onset.

受感染的人可以有睡眠紊乱,个性改变,共济失调,失语症,视觉丧失,物理,肌肉萎缩,肌阵挛,进行性痴呆等症状,并且会在发病的一年内死亡。

Although most individuals develop spastic paraparesis, mild ataxia, and occasional mild cognitive deficit in their second decade, the course of the disease is relatively stable.

虽然大多数患者下肢轻瘫、轻度共济失调和偶而出现在十几岁时的轻度认知障碍,此病的进程相对稳定。

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