促进

The Chinese compound prescription can improve the bone formation and prevent bone loss via inhibiting bone absorption and improving ossify,bone mineral deposition and mineralization as well as increasing BMD,which leads to prevention and treatment of bone loss.

中药复方能明显促进骨形成作用，有效抑制骨吸收作用，减弱溶骨过程从而促进骨矿盐的沉积和矿化，增强BMD而达到防治骨丢失的作用；有效的防护和治疗失重性骨丢失采取联合措施很有必要。

Osteitis fibrosa is a manifestation of sencondary hyperparathyroidism.

动物实验已经证实，PTH对骨代谢具有双相作用，即间歇给药促进成骨，而大剂量持续给药则促进骨吸收。

Conditions of the seepage and stress is a dynamic process, and also shows its interaction and interdependence. Owing to the closure of the fracture, the hydraulic gradient and the permeability pressure of the fracture turn large, and also promote the expansion of water cracks, and fissures connectivity increasing. The increase of the crack opening width will result in the increase of the infiltration capacity and seepage flow, and the reduction of seepage pressure. Because of the dynamic dependence of the fracture aperture, pressure and seepage, under certain conditions, changes in fracture aperture can lead to the formation of local water-passage, and the local emission of high pressure water, and thus promotes the formation of the sudden outgush disasters.

结果表明：洞室开挖完成后，在围岩渗流与应力耦合作用下，围岩中裂隙隙宽、裂隙中水压及其渗透流量的变化是一个动态过程，且相互作用与相互依赖；裂隙的闭合使得结构面水力梯度变大，作用在裂缝上的渗透压力增大，促进导水裂缝扩展，裂隙连通性增加；裂缝张开度增大，渗透能力增强，渗流量增大，其渗流压力相应降低；由于围岩中裂隙隙宽、压力和渗流量的动态依赖性，在一定条件下，裂隙隙宽的改变可导致局部水力通道的形成，高压水头从局部涌出，从而促进突水灾害的形成。

Ransforming growth factor beta TGF-β is a potent growth suppressor of normal epithelial cells. However, many types of invasive cancer cells (e.g. breast cancer frequently overproduce TGF-β to enhance their growth and metastasis. Quite frequently, these cells do not possess a functional type II TGF-β receptor, indicating an aberrant TGF-β signaling is responsible for promoting cell growth.

Transforming growth factor beta 对於正常的上皮细胞是一个很强的生长抑制因子，然而，许多种具有蔓延侵犯性的癌细胞常常是表现大量的 TGF-β来促进细胞生长与转移，这些癌细胞通常不表现具有功能性的第二型 TGF-β receptor，显示有另类的 TGF-β讯息传递可以促进细胞生长。

Alu dna was appeared on low level cam and no alu was found in control group.conclusion the results suggested that ph|20 may increase the palingenesis of blood vessel and stimulate human breast cancer cell growth and metastasis.

目前关于这方面的研究不多，本课题组曾将人ph|20 cdna转染到人乳腺癌细胞株mda231中，初步发现了ph|20可能通过肿瘤细胞释放fgf和促进血管生长来促进mda231增殖[5]。

The results showed that exogenous substances could promote the upper leaf area increase, and accommodated the thickness of palisade tissues, spongy tissues and the cell density of palisade tissues dynamic changes, and decreased leaf thickness, loosen the tissue, and increased the potassium content, while reduced nicotine content.

结果表明，外源物质可促进上部叶面积增大，调节叶片栅栏组织、海绵组织厚度和栅栏组织细胞密度的动态变化，促使叶片厚度降低，组织变得较为疏松，促进钾素含量提高和烟碱含量降低。

The chest and breast massage will promote blood circulation, improve metabolism, so sympathetic and parasympathetic nervous system active, to promote breast development.

按摩会促进胸部和乳房的血液循环，提高代谢能力，使交感神经和副交感神经系统活跃起来，促进乳腺发育。

The results showed that 2,4 D had promotive effects on the growth of pavlova viridis, especially in the early stage of culture, and the optimum mass concentration was 0.1 mg/L. 6 BA had lethal effect on pavlova viridis, and the lowest lethal mass concentration was 1.0 mg/ L.

结果表明，在试验浓度范围内，2 ，4— D对绿色巴夫藻生长的影响在培养早期比后期明显，最适浓度为 0 。1 m g/L ；6-BA质量浓度≥ 1 mg/L时对绿色巴夫藻有致死作用，质量浓度为 0 。1～ 0 。8mg/L时，对绿色巴夫藻的生长有促进作用，促进生长的最适质量浓度为 0 。5mg/L。

The effect of the accelerators in phosphating solution on phosphatization was investigated. The reaction mechanism was also studied.

为此研制了一种含硝酸肌和氯酸钠的复合促进剂，考察了复合促进剂所含物质对磷化液磷化效果的影响，对其作用机理进行了讨论。

PIAS proteins play roles mainly through two mechanisms: acting as SUMO E3 ligases to promote modifications of some transcriptional factors and cofactors, especially sumoylation, and to regulate their transcriptional activation; acting as structural proteins to offer platforms for protein-protein interactions and to promote the abstraction and recruitment of other regulators in the cellular signal pathway complexes or gene transcriptional complexes, companied by the subnuclear location of target proteins.

PIAS蛋白的调控机制主要有两种：一种是通过其自身所具有的SUMO(small ubiquitin-related modifiers)E3连接酶活性，促进对一些转录因子、转录辅因子的化学修饰，尤其是SUMO化修饰，从而调控它们的转录活性；另一种是作为构架蛋白，为蛋白质之间的相互作用提供平台，促进细胞信号通路复合物或基因转录复合物中其它调节蛋白的去除和募集，并涉及到靶蛋白的亚核定位。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。