作用于

It is also suggested that ACh plays an important role in the maintenance of excitability of cortical neurons.

这一作用可能是通过作用于M受体，影响突触前递质释放来实现的。

Conventional tennis wants you to shuffle all the time and then take but one step, which is arrhythmic, causes you to lose your balance, promotes an open stance, and sends you and your momentum off to the side instead of into the ball.

传统的网球指望你总是侧滑步，然后上一步，这样没有节奏，会使你失去平衡，促使你用开放式站姿，使你和你的冲量侧身作用而不是正向作用于球。

The Michaelis-Menten constant of xylanases in the enzymatic hydrolysis of birchwood xylan is 5.37g/L, and the maximal reaction velocity is 0.94μmol/min.

该木聚糖酶作用于Birchwood木聚糖的米氏常数为5.37g/L，最大反应速率为0.94μmol/min。

In the presence of cellobiose, CDH can oxidize PSC to produce water soluble substances. The substanes formed could not be oxidized by CDH. But they had the ability to reduced K3Fe6 reagent.

在纤维二糖的存在下，CDH可以作用于PSC生成水溶性的物质，此物质不被CDH氧化，但具有还原力，可以还原铁氧化钾。

It is postulated that IP induces the release of endogenous protective molecules that interact with cognate receptors to activate PLC through mediation of the chincough virus sensitive proteins. PLC hydrolyzes membrane phospholipid and produces diacylglyccerol,that,by working together with calcium hydronium,translocates PKC from cytoplasm to cell membrane and activates PKC.

推测缺血预处理细胞保护作用机制是：缺血预处理引起内源性保护物质释放，作用于细胞膜上的相应受体，通过百日咳病毒敏感性G蛋白的介导而激活磷脂酶，磷脂酶水解膜磷脂，产生二酰基甘油(diacylglyccerol，DAG)，DAG和Ca2+协同作用于PKC，使其从胞浆转位到细胞膜并且被活化，PKC在膜上使底物蛋白磷酸化，引发一系列相关信号转导通路的激活，调控保护性基因的表达，从而提高细胞对缺血再灌注损伤的耐受性。

Objective\ This study was to investigate the effect of crocidolite fibe rs on IL-8 protein and mRNA levels in A549 cells cultured supernatant and the r o le of tyrosine kinase inhibitor PD98059 on IL-8 expression.

目的 研究青石棉纤维作用于A5 4 9细胞后IL 8蛋白和基因表达水平及酪氨酸激酶抑制剂PD980 5 9对IL 8表达的影响。

That has nothing to do with the bottle and nothing to do with the goose either. You have to wake up. It has something to do with YOU.

它并不作用于瓶子，也不作用于鹅，你必须苏醒——这就是你所要做的，它作用于你。

As the DDP group A549DDP cells were treated with IC10 of cisplatin for 48 hours. As the DDP＋Rh2 group A549DDP cells were treated with IC10 of cisplatin and IC5 of gensenoside Rh2 for 48 hours. As the controlling group A549DDP cells were not treated with any kinds of drugs. Changes in cellular morphology were observed by fluorescence microscopy. Peak value apoptosis of A549DDP cells were inspected by flow cytometry. State of PTP was evaluated by ultraviolate spectrofluorometer. Changs of calcium in cells and membrane potential of mitochondrion were determined by flow cytometry. Release of cyt-c from mitochondrion with westton blot Radiative activity of〓Tc-MIBI ingested by cells was measured withγcounter. Results: IC50 of cisplatine to A549 cells was 24uM.

以无毒浓度的Rh2单独作用于A549DDP细胞作为Rh2组，以低效浓度的顺铂单独作用于A549DDP细胞作为DDP组，以无毒浓度的Rh2和低效浓度的顺铂联合作用于A549DDP细胞作为Rh2+DDP组，作用时间为48小时，以不加药物干预作为对照组，Hoechst33258染色荧光显微镜下观察细胞形态的变化，流式细胞仪检测细胞凋亡峰；紫外光分光光度计检测线粒体PTP开放情况，流式细胞仪检测细胞内钙离子浓度及线粒体跨膜电位的变化，Westton blot检测线粒体内细胞色素C释放情况及Caspase-3的活性，γ计数仪检测细胞摄取〓Tc-MIBI放射性活性。

749Mg/ml and 0.778u/ml, respectively. 5. Study on the viscosity changes of reaction solution and analysis of enzymatic hydrolysis products by TLC, HPLC showed that ChiB was an endo-type cleavage enzyme. It decreased viscosity of reaction solution, hydrolyzed β-(1, 4) glucosidic bond of chitosan, acted on GlcN-GlcN, GlcN-GlcNAc and/or GlcNAc-GlcN glucosidic bond. It did not act on GlcNAc-GlcNAc glucosidic bond. Hydrolysis products of chitosan were composed of chitotriose, chitotetraose and chitopentose.

通过测定酶解液体系粘度和对酶解产物的TLC、HPLC分析，在国内首次深入地研究并报道了内切、外切壳聚糖酶的酶解作用模式，结果表明：酶ChiB是一种内切酶，以内切方式水解壳聚糖的β-（1，4）糖苷键，即作用于GlcN-GlcN、GlcN-GlcNAc和／或GlcNAc-GlcN糖苷键，不能作用于GlcNAc-GlcNAc糖苷键；酶解终产物以壳三糖、壳四糖、壳五糖为主。

The survival rates of CEM cells cultured with cis-diamminedichicloroplatinum added 24 h later were higher than that cultured with hTERT ASODN and DDP added 24 h later. The survival rates of CEM cells cultured with DDP were similar with that cultured with hTERT SOND and DDP. In morphological observation of apoptotic cells using Giemsa staining, cells treated with DDP or DDP combined with hTERT ASODN ro SODN at 48 h, displayed classic apoptotic changes. Apoptosis rates of CEM cells treated with DDP for 48 h after 24 h of exposure to ASODN significantly increased. There was significant difference in the percentage of apoptotic cells of CEM cells between hTERT ASODN plus DDP and SODN plus DDP or DDP alone, respectively.

结果： hTERT ASODN作用于CEM细胞24 h再加入柔红霉素、长春新碱、足叶乙甙，对细胞生长的抑制分别与单用柔红霉素、长春新碱、足叶乙甙及hTERT正义寡核苷酸联合柔红霉素、长春新碱、足叶乙甙组相比，统计学上无显著差异(P＞0.05)。hTERT反义核酸作用于CEM细胞24 h加入顺铂，再共同作用48 h，CEM活细胞均数为2.318×108 cells/L，与单用顺铂组(3.250×108 cells/L)及hTERT正义核酸联用顺铂组(3.175×108 cells/L)相比，对细胞抑制明显增强(P＜0.05)。hTERT ASODN作用于CEM细胞24 h再加入顺铂作用48 h，细胞出现典型的凋亡形态学改变。hTERT ASODN与2.5 μmol/L顺铂联合作用于CEM细胞48 h的凋亡细胞百分率(19.47%)分别同SODN与顺铂联合作用组(6.97%)、单用顺铂作用组(6.02%)进行比较有显著差异(P＜0.01)。

As she looked at Warrington's manly face, and dark, melancholy eyes, she had settled in her mind that he must have been the victim of an unhappy attachment.

每逢看到沃林顿那刚毅的脸，那乌黑、忧郁的眼睛，她便会相信，他一定作过不幸的爱情的受害者。

Maybe they'll disappear into a pothole.

也许他们将在壶穴里消失