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To investigate the effects of arsenious acid on expression of HIF-1α in human hepatocellular carcinoma HepG2 cells.

目的 探讨亚砷酸对人肝癌细胞HepG2中HIF-1α表达的影响。

Objective:To evaluate the safety and efficacy of combining radiofrequency ablation with arsenious acid locally to treat liver VX2 tumor in rabbits,and to explore its mechanism.

目的:探讨射频消融联合亚砷酸局部治疗对兔肝VX2肿瘤的有效性和安全性,并探讨其机制。

Inhibitory effect of arsenious acid on expression of HIF-1α. in human hepatocellular carcinoma HepG2 cells were through post-transcriptional mechanisms.

亚砷酸抑制人肝癌细胞HepG2中HIF-1α蛋白表达是通过转录后机制。

Objective To explore the mechanism of sodium arsenite inducing apoptosis of NB4 cells.

目的 探讨亚砷酸钠诱导NB4细胞凋亡的作用机制。

Objective To explore the effect of sodium arsenite(NaAsO2) on antioxidative ability in human keratinocytes.

目的 研究亚砷酸钠(NaAsO2)对人皮肤角质形成细胞抗氧化能力的影响。

Objective To investigate the effect of sodium arsenite combined with cigarette smoke solution on expression of Bcl-2 and Bax in rat lymphocytes and study their interaction.

目的 研究亚砷酸钠和香烟烟气溶液联合作用对凋亡相关基因Bcl-2和Bax表达的影响,并探讨它们对这2种基因表达的影响是否存在交互作用。

Sodium arsenite and cigarette smoke solution have interactions on protein expression of Bcl2 and Bax.

亚砷酸钠和香烟烟气溶液对大鼠淋巴细胞的Bcl2蛋白表达、Bax蛋白表达的影响存在交互作用,交互作用方式为协同作用。

Key words: occupational and environmental health; sodium arsenite; liver and brain; oxidation/antioxidation

关键 词:劳动卫生与环境卫生学;亚砷酸钠;肝脑;氧化/抗氧化

Sodium arsenite could obviously inhibit the proliferation of BGC823 cells:induce cell cycle arrest and apoptosis and necrosis of the cells.

亚砷酸钠对BGC823细胞的生长有明显的抑制作用,并可诱导细胞周期阻滞及细胞凋亡和坏死,其机制可能与其抑制ROS的清除,上调Caspase3蛋白的表达有关。

The electrolyte was purified by copper arsenite to effectively remove Sb and Bi impurities in the copper electrolyte .

为有效脱除铜电解液中的Sb、Bi等杂质,采用亚砷酸铜净化电解液。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。