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Rats in model control group were treated with distilled water while those in silibinin group were treated with 100 mg/kg silibinin. Meanwhile, 100 mL/L ethanol and hyperalimentation feed were given for 4 weeks. After animals were killed, TG, SOD, GSH and MDA levels were measured with liver suspension.

同时继续给予100 mL/L乙醇和高营养饲料,4周后在麻醉状态下处死大鼠,取大鼠肝块进行病理观察,测定三酰甘油、超氧化物歧化酶、还原性谷胱甘肽和丙二醛水平。

Results showed that L-THP possessed negative inotropic and negative chronotropic action, lowed left ventricular pressure and it's infinitesimal calculus, decreased heart output in per min., shorten 〓 and 〓 of action potential of ventricular papillary muscle of guinea pig and even the plate-phase of action potential disappeared in larger dose group , but prolonged 〓. we studied transmembrane transportation of 〓 after administering L-THP , L-THP not only depressed uptake and increased release of Ca 2+ in normal myocardium, but also it showed stronger inhibition in cell injured by ISO, and fully eliminated the effect of ISO increasing uptake and decreasing release of 〓. By detecting the 〓 content of ventricular single myocyte using fluorescent probe Fluo-3/AM, results correspond to that done above, L-THP decreased 〓 content inside of cell significantly. Using whole-cell mode, we observed the effect of L-THP on Ica in ventricular single cell, it was found that L-THP decreased Ica, the effect increased with increasing it's dose, and had voltage, frequency-dependency. Dose highly corelated to effect, corelative constant is 0.87. When washing cell with normal tyre's solution, Ica could recover, which proved the blocked action of L-THP on calcium channel once again. Meanwhile, L-THP reduced production of MDA in heart injured by ISO in dose-dependent manner, improved content of GSH-Px, in case tissue or cell free from free radical damage.

结果显示,该药物具有负性肌力和负性频率作用,降低左室压及其微分,终使每分心输出量减少;缩短豚鼠心室乳头肌动作电位时程的〓和APD〓,大剂量使动作电位平台消失,〓延长;同位素示踪法研究证实L-THP抑制〓摄取,促进〓释放,降低细胞内的〓含量;单通道水平的研究证实,L-THP减少豚鼠心室肌单细胞内向钙电流,且具有剂量,电压,频率依赖性,其作用易于洗脱,药物剂量与Ica也具有高度相关性(相关系数r为-0.87);L-THP还可抑制ISO损伤心肌组织所致的丙二醛含量增加,增加谷胱甘肽过氧化物酶的量,使组织细胞免受损伤。L-THP阻断L-型〓通道,抑制〓的摄取,促进〓的释放,降低细胞内〓含量L-THP作用的离子基础。

The activities of glutathione peroxidase, superoxide dismutase, catalase, the content of malonaldehyde in serum and lens were detected. At the same time, the activities of Na+-K+-ATPase, the content of macromolecular weight protein and infusibility protein in lens were detected too.

比色法测定各组血清及晶体中超氧化物歧化酶、谷胱甘肽过氧化物酶、过氧化氢酶活力和丙二醛含量以及晶体中Na+-K+-ATP酶活力、高分子质量蛋白和不溶性蛋白含量。

According to the results described above, we try to synthesize the long peptide chain of Lys fragment to prove these results and also to get enough sample to do X-ray and 2D NMR analysis.

根据上述结果我们试图对长链26肽进行化学合成,一方面从合成角度进一步证实以前的结果;同时为了得到足够的样品进行晶体结构或二维核磁共振的研究。

estradiol obviously accelerate ANP synthesis of myocardial cells in ovariotomy rats.

雌二醇可明显促进卵巢切除大鼠心房肌细胞心房肽的合成。

The effects of the food additive butylated hydroxyanisole and the antibiotic Norfloxacin on swordtail fish were measured by considering a number of parameters, including glutathione S\|transferase, catalase, 7-ethoxyresorufin-o-dealkylases and malondialdehyde contents.

研究了叔丁基对羟基茴香醚(butylated hydroxyanisole, BHA)和诺氟沙星(norfloxacin, NFLX)对剑尾鱼的急性毒性及其肝脏谷胱甘肽硫转移酶、丙二醛、过氧化氢酶及7-乙氧基异吩恶唑酮-脱乙基酶活性的影响。

The expression ephthase de ep autous he orge eis telos,"the wrath hath come upon them unto the end"(ii, 16), naturally refers to the destruction of Jerusalem (AD 70) as an accomplished punishment of the Jews for killing the Lord Jesus.

表达ephthase日内啡肽autous他奥尔顺telos ,"上帝的愤怒后,他们来到你们的结束"(二16 ),自然是指破坏耶路撒冷(公元70 ),作为一个多才多艺的惩罚杀害犹太人的上帝耶稣。

Superior mesenteric artery was clipped for 60 min to cause ischimia, and then unclipped to make reperfusion for 2 hours after reperfusion, the liver tissues were gathered and homogenized respectively, and the contents of SOD, GSHPX, MDA and ATPase,and morphological changes of hepatocytes were assayed.

夹闭大鼠肠系膜上动脉60 min造成缺血,再灌注2 h后取出肝组织制成匀浆,测定超氧化物歧化酶、谷胱甘肽过氧化酶、丙二醛、Ca2+Mg2+ATP酶的含量及肝形态细胞学变化。

Superior mesenteric artery was clipped for 60 min to cause ischimia,and then unclipped to make reperfusion.2 hours after reperfusion,the renal tissues were gathered and homogenized,and the contents of SOD,GSHPX,MDA and Ca2+Mg2+ATPase,and morphological changes of renal were assayed.

夹闭大鼠肠系膜上动脉60min造成缺血,再灌注2h后取出肾组织制成匀浆,测定超氧化物歧化酶、谷胱甘肽过氧化酶、Ca2+Mg2+ATPase的活性及丙二醛含量,观察肾组织形态细胞学变化。

These revealed that high dose copper can destroy function of erythrocytic membrane and cause oxidative stress, which may be one of the mechanisms of goat erythrocytic haematolysis induced by high dose copper. 3 High copper caused the increase of osmotic fragility of erythrocyte and AGLT50 in acor glycerine solution.

高铜导致红细胞膜超氧化物歧化酶和谷胱甘肽过氧化物酶活性降低,氧化产物丙二醛含量增多,引起红细胞抗氧化功能降低,诱发氧化胁迫,使得红细胞处于氧化应激状态,这可能是高铜致羊红细胞溶破机制之一。

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Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气,收缩臀部肌肉;拱起身体,尽量抬起头来,右腿伸直朝向天花板(膝微屈,以避免肌肉紧张)。

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然而,要让一个真正的引用,你需要提供详细的个人和财务信息。