乳酸

These enzyme existence is big to the lactic dehydrogenase response disturbance.

这些酶的存在对乳酸脱氢酶反应的干扰较大。

Convenient assay measures activity of lactate dehydrogenase released from damaged cells.

能方便的测定损伤细胞释放的乳酸脱氢酶活性。

For example, lactate dehydrogenase as a tetramer.

例如乳酸脱氢酶为四聚体。

In blood, luteotropin significantly increased at DCAB 400 fed sample, estradiol (E2) and urea decreased at DCAB 400 and 500 fed sample. However, Lactate dehydrogenate, creatine phosphokinase and total protein had a significant increase for DCAB 300 and 400 fed sample.

血液指标中，DCAB 400组提高了促乳素，DCAB 400和500组提高了雌二醇浓度和尿素；DCAB 300和400组乳酸脱氢酶、肌酸磷酸激酶和总蛋白显著提高；处理组钙含量显著高于对照组。

Methods Rabbit pulmonary alveolar macrophages were cultured in vitro. The cell death rate, the malandialdehyde, lactate dehydrogenate and superoxide dismitase contents in the cultured cells were determined at different incubation time and different dose of chrysotile asbestos for the cytotoxicity of the material.

采用体外细胞培养技术，以兔肺泡巨噬细胞死亡率、乳酸脱氢酶、超氧化物歧化酶活力及丙二醛含量变化来表示不同剂量、不同培养时间的纤状蛇纹石石棉对巨噬细胞的毒性。

SMART analysis suggested that the ORF encoding protein contained a Ldh_1_N and a Ldh_1_C domain. Psite analysis showed that there was lactate dehydrogenate active site in the protein, which localized in cytoplasm according to PSORT program analysis.

SMART分析表明ORF编码的蛋白质具有Ldh_1_Ndomatn和Ldh_1_Cdomain，Psite分析表明有L-乳酸脱氢酶活性位点，PSORT程序分析将其定位于细胞质中。

A first demonstrator of the nanopotentiostat for lactate measurement has already been produced

第一个测量乳酸的nanopotentiostat已经产生。

Dialdehyde starch not only improves the mechanism function of PLA,but also has good effect on cells.And it doesn'〖KG-3〗t affect the growth and metabolism of cells.So it is feasible as scaffold of tissue engineering artificial skin.

双醛淀粉作为良好的增柔剂在改善聚乳酸网架机械性能的同时，也使其具有良好的细胞相容性，不影响细胞的生长增殖和代谢，可以进一步用作组织工程皮肤的支架材料。

After developing 48 hours, hepatocytes enters 2.0 mM t-butyl hydroperxide or 2.0 mM diamide to induced cell blebbing. The samples collected at 0, 15, 30, 60 minutes to analyze cell about membrane protein thiol deletion; the leakage of lactate dehydrodenase; the change of glutathione; the lipid peroxidation; the variation of actin and tubulin, and use Confocal fluorescent microscope to observe the change of actin and tubulin in rat hepatocytes.

肝细胞在48小时培养后，分别加入2.0mM t-buty1 hydroperoxide或2.0 mM diamide诱发细胞膜小泡生成，并分别於0、15、30、60分钟取样，分析细胞膜硫醇流失程度；乳酸去氢□渗漏；□胱甘□浓度变化；脂质过氧化；肌动蛋白与管蛋白变化；并利用雷射共轭焦萤光显微镜观察肝细胞中肌动蛋白及管蛋白的变化。

The result indicates the t-BHP or diamide both cause the mass loss about the membrane protein thiol in hepatocytes, the fast oxidization about glutathione in hepatocytes, the leakage of lactate dehydrodenase, but the level of t-BHP is larger than of diamide. In the aspect of lipid peroxidation, only the t-BHP produces apparent change. As to actin and tubulin, they both find fall-off and present time-dependency effects. The result in Confocal fluorescent microscope also show these two cytoskeletal proteins are related to cell blebbing, but they don''t play the same role during the porcess. Tubulin may be more important than actin. If the study predispose dithiothreitol before adding t-BHP or diamide, then the damage by the diamide is repressed, but just partly protection for the damage by t-BHP.

结果显示t-BHP或diamide均可造成细胞膜蛋白硫醇大量流失与胞内GSH快速氧化，亦显著导致乳酸去氢□渗漏，但t-BHP诱发的渗漏程度大於diamide；在脂质过氧化方面，则仅t-BHP处理组产生明显的脂质氧化情形；至於肌动蛋白与管蛋白单体均可见减少且呈现时间依赖效应；在雷射共轭焦萤光显微镜观察中，亦显现这两种架构蛋白与细胞膜起泡有关，但两者在小泡生成的过程中扮演的角色并不完成相同，可能管蛋白较肌动蛋白重要；如果细胞在t-BHP或diamide加入前预先以dithiothreitol处理，则diamide 所造成的伤害显著的受到抑制，但DTT对t-BHP所诱发的伤害则仅有部分保护效果。

But we don't care about Battlegrounds.

但我们并不在乎沙场中的显露。

Ah! don't mention it, the butcher's shop is a horror.

啊！不用提了。提到肉，真是糟透了。