主动脉内膜炎

Results The body weight, visceral fat and body weight ration, as well as the levels of blood glucose, insulin, triglyceride, total cholesterol, LDL, and total FFA, were significantly lower in high fat-fed group than in control group (P.01). The aortic tunica intima was intact, the number of foam cells was decreased, and inflammatory cells infiltrated into the med-intima smooth muscle of the n-3 PUFA group. However, with no such changes were observed in control and n-3 PUFAs groups.

结果 n-3脂肪酸组大鼠体重第1~11周均明显降低，空腹血糖、血清胰岛素、TG、TC、LDL-C、总FFA和内脏脂肪占体重百分比均明显低于HF组，HDL-C则明显高于HF组，与对照组无明显差别；n-3脂肪酸组大鼠主动脉主动脉内膜完整无内皮细胞脱落现象，内膜下泡沫细胞及炎细胞浸润减少，内膜的NF-κB呈弱阳性表达，IκBα呈阳性表达，ICAM-1和MCP-1均呈阴性或弱阳性表达。

The aortic tunica intima was intact, the number of foam cells was decreased, and inflammatory cells infiltrated into the med-intima smooth muscle of the n-3 PUFA group. However, with no such changes were observed in control and n-3 PUFAs groups. Little or no expression of NF-κB, ICAM-1 and MCP-1, and a high expression level of IκBα were detected in aortic tunica intima of the n-3 PUFA and control groups, while high expression levels of NF-κB, ICAM-1 and MCP-1, and little expression of IκBα were detected in aortic tunica intima of the high fat-fed group.

结果 n-3脂肪酸组大鼠体重第1~11周均明显降低，空腹血糖、血清胰岛素、TG、TC、LDL-C、总FFA和内脏脂肪占体重百分比均明显低于HF组，HDL-C则明显高于HF组，与对照组无明显差别；n-3脂肪酸组大鼠主动脉主动脉内膜完整无内皮细胞脱落现象，内膜下泡沫细胞及炎细胞浸润减少，内膜的NF-κB呈弱阳性表达，IκBα呈阳性表达，ICAM-1和MCP-1均呈阴性或弱阳性表达。

And rheumatic valves also accounted for 70 cases (88.5%) of aortic stenosis plus insufficiency. The rheumatic valves was decreased dramatically as the cause of the aortic valve disease, aortic root dilation and degenerative valves increased significantly, while others had no significant changes.

随着社会进步和人口老龄化，风湿性病因所占比例大幅下降，主动脉根部扩张导致的主动脉关闭不全有较大幅度的上升，退行性主动脉瓣有所上升，二叶式主动脉瓣、感染性心内膜炎所致瓣膜损害变化不明显。

The characteristics and changes of etiology were analyzed. All cases with aortic diseases were divided into single aortic stenosis, single aortic insufficiency, and combined aortic stenosis plus insufficiency group.

分别以主动脉瓣狭窄、主动脉瓣关闭不全、主动脉瓣狭窄合并关闭不全3组分析，具体病因包括风湿性、二叶式主动脉、退行性主动脉瓣、主动脉根部扩张、感染性心内膜炎、少见及未确定等6类病因。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。