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中轴骨骼

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The sacrum is the most frequent site of occurrence in the axial skeleton.

骶骨是中轴骨骼上最常见的发病部位。

FOXC2 protein and mRNA were extensively expressed in pregnant 40 day embryonic somite and neural tube mesoderm which would form cardiovascular and axial skeleton primordia, especially strong expressed in the cells of mesenchyme surrounding notochord and dorsal aorta approaching the head.

这个基因广泛表达在孕40天胚胎体节和神经管周围未来分化为心血管和中轴骨骼始基的中胚层,靠近头部围绕脊索和背主动脉的间充质细胞核中表达强烈。

In this investigation fragments of Acropora pulchra were used in a semiprotected nursery in southern Taiwan between 1996 and 1998 to test, in situ, the possible effects of different factors on the generation of new branches and the initial skeletal extension rates of transplants.

在调查研究的美丽轴孔珊瑚是以半保护的养殖牧场在1996年到1998年於南台湾做测试,在那里,有可能会影响移植片段中的新分叉生长和骨骼生长速率之不同因子;这些变因在此研究中有片段的来源及长度、它们新的定向、存在的组织伤害和片段的位置。

Primitive, solid, skeletal structure derived from specialized mesodermal cells.

脊索,一种动物体在未形成脊柱以前的细胞组织,状如绳索,为胚胎时期的原始中轴骨骼

Pregnant 50 day onward, FOXC2 mainly expressed in dorsal aorta, arch arteries, archae-endocardium and cells of vertebrae rudiment. The expression of FOXC2 could be seen in cordis great vessels, endocardium, chondrocyte and periost of axial skeleton during pregnant 70 days, and only in primary ossification center of axial skeleton and coronary artery during pregnant 80 day.

孕50天后表达于背主动脉、弓形动脉和原始心脏内膜、锥体雏形细胞;孕70天时,在心脏大血管、心内膜和中轴骨骼的软骨细胞和骨膜细胞表达;孕80天时仅表达于中轴骨骼的骨化中心和冠状动脉。

Osteoid osteomas usually occur in the axial skeleton in bone cortex of young males in the second decade of life.

骨样骨瘤常见于十几岁男性中轴骨骼的骨皮质。

Congenital cardiovascular anomalies and axial skeleton defects are one of the principal diseases menacing human health.

先天性心血管和中轴骨骼发育缺陷是威胁人类健康的主要先天性疾病之一。

Utilizing human embryos first time, this research systematically studied the effect profile of FOXC2 on cardiovascular and axial skeleton morphogenesis.

本项目首次以人胚胎为研究对象,系统研究了FOXC2基因在人胚期心血管和中轴骨骼发生、发育过程中的作用模式。

The results of FOXC2 expression profile in human embryos suggest that mutations in FOXC2 gene could lead to cardiovascular and axial skeleton development defects in human, and there are novel polymorphisms of FOXC2 gene in Han people.

根据FOXC2在人胚的表达模式,提示该基因变异可能导致人心血管和中轴骨骼发育缺陷;在汉族人群可能存在新的FOXC2基因单核苷酸多态性。

Homazygous Foxc2 deficient mouse exhibits cardiovascular anomalies and axial skeleton defects.

FOXC2基因纯合子缺损鼠可引起心血管和中轴骨骼发育异常。

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The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

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