与肺有关的

deep vinous thrombosis, operation, fibrinogen, cholesterol, plasminogen, smoking are independent and important risk factors for VTE.

下肢深静脉血栓形成，手术，高纤维蛋白原，胆固醇，纤溶酶原，吸烟是肺栓塞的独立危险因素，肺栓塞发病与多种因素有关。

ConclusionDeep vinous thrombosis, operation, fibrinogen, cholesterol, plas-minogen, and smoking are independent and important risk factors for VTE.

结论下肢深静脉血栓形成、手术、高纤维蛋白原、胆固醇、纤溶酶原、吸烟是肺栓塞的独立危险因素，肺栓塞发病与因素有关。

Compared with the control groups,we found that only fraction Ⅳ decreased the lung coefficient of RDS mice in a dose dependent manner and ameliorate the decrease in blood PaO_2 and the protein exudation of RDS rabbits.Because of the anticomplement activity of fraction Ⅳ, it was suggested that this ...

结果显示：与对照组相比，组份Ⅳ使实验组小鼠的肺系数降低且呈量－效关系；使实验组家兔PaO2下降幅度减少，支气管－肺泡灌洗液蛋白渗出量减少；组份Ⅳ具有抗补体活性的作用，其缓解油酸型RDS的作用可能与此有关。

We observed a significant increase in a genomic signature of phosphatidylinositol 3-kinase (PI3K) pathway activation in the cytologically normal bronchelloal airway of smokers with lung cancer and smokers with dysplastic lesions, suggesting that PI3K is activated in the proximal airway before tumorigenesis.

在此项研究中，Adam Gustafson及其同僚在具备或没有肺癌的吸烟者中以及那些具备较高罹患肺癌危害的人寿检测了在覆衬着主要肺气道的细胞中的属于差别的与癌症有关的通路的基因表达程度。

We found for the first time that the epithelium of bronchus of normal broilersexpressed iNOS mRNA.iNOS activity in the epithelium of bronchus of preascitic andascitic broilers were higher than normal broilers,but there was no iNOS activity inpulmonary vessels of all the three types broilers.The results indicated that the increasedNOS expression in pulmonary vessels of preascitic and ascitic broilers was not iNOS.

首次发现在肉鸡肺动脉高压发展过程中，肺血管NOS的表达是增加的，尤其是正常鸡无NOS表达的肺小血管内皮和平滑肌在腹水鸡却呈高水平表达，且NOS活性增加的肺小血管也同时存在管壁平滑肌层增厚或无肌型血管肌化的结构重建现象，结果提示NOS表达增强可能与肺血管中膜增厚有关。

Results Univariate analysis demonstrated a significant association between early postoperative death and the following variables: weight at the time of surgery,age for cyanosis to be present,SaO2,AO/MPA,McGoon ratio,Nakata index,Qp/Qs,/Qs,multiple aortopulmonary collateral arteries and transannular patching.

结果 单因素分析结果显示与术后早期死亡有关的指标包括：手术时体重、发生青紫年龄、动脉血氧饱和度、升主动脉与肺动脉干直径比AO/MPA 、McGoon比值、Nakata指数、肺体循环血流量之比Qp/Qs 、右向左分流量占体循环血流量比例/Qs 、体肺侧支循环和跨瓣补片。

Lung Ang Ⅱ correlated positively with MIP-2 in BALF in all study rats. These results indicate that local angiotensin system is involved in the pathogenesis of VILI and suggest that blockade of Ang Ⅱ might have potential therapeutic implications in alleviating VILI.

这些结果表示局部血管紧缩素系统与机械通气导致肺伤害的病理生理机转有关，并且建议阻断血管收缩素可能有治疗机械通气导致肺伤害的潜在功效。

NAC also blocked the up-regulation of TGF-β in in vivo model of VILI. Conclusion TGF-β1 and TGF-β2 were up-regulated in VILI. Oxidant injury mediated up-regulation of TGF-β in VILI. NAC, which attenuated oxidant injury and blocked TGF-β up-regulation in VILI, could be a future therapeutic strategy in VILI.

结论TGF-β1和TGF-β2在通气机所致肺损伤中的表达增加并在血清中上调；TGF-β上调的机制与氧自由基损伤有关但与肿瘤坏死因子无关；N-乙酰半胱氨酸可对抗氧自由基损伤，能阻断TGF-β的上调，可能成为呼吸机相关肺损伤的一种新治疗途径。

The ultimate regulation of inflammatory factor was involved, at least in part, in the mechanism of action of Curcumine and TyrphostinA1 effects.

本研究结果表明，姜黄素和TyrphostinA1能抑制油酸诱导的肺损伤，其作用机制可能与调节细胞因子（TNF-α、IL-6和IL-10）的合成与释放有关。

Imaging appearances of AIDS complicated with pulmoanry tuberculosis coexisted with multiple foci of infection and polymorphism that distributed in multiple lobus. Most of AIDS patients were without typical tuberculous proliferative foci and lyphadenectasis, possibly to be associated with decreasing extent of immune function and the progress of disease at various stages in the young patients observed.

AIDS合并肺结核的胸部影像学特征多表现为多性质的病灶共存，多形态、多叶段分布，以两下肺明显的渗出性、多形态病灶；纤维化、钙化、肿块样阴影少见，无典型的结核增殖灶、肺门淋巴结肿、胸水产生与有关文献报道有异，可能与本组患者为年青人及免疫下降程度和病程发展不同阶段等因素有关。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。