不改变的

This change of state is a physical change and not a chemical one.

这个状态的改变是物理改变，不是化学改变。

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我想改变世界，但我无能为力，于是我改变自己，并且保证自己不再被改变/个性/自由/民主/好奇/电影/书籍/运动/AV/希望有一天推特中文圈推的全不是政治

We may not change past , neither could do nothing about making any changes of given charactoristics , as well as inevitableness .

我们不能改变过去，不能改变别人特定的行为方式，不能改变必然发生的事情。

"If we didn't want to change that, why change the lineup?"

"如果我们不想改变去年的一切，那为什么还要改变座次呢，我们不会改变我们的打法的"。

It you don't loke something ,change it ,It you can't change it ,change your attitude ,Don't complain.

如果不喜欢什么，那就去改变它。如果你无力改变它，那就改变你的态度。不要报怨。

Also be aware that changing the orientation or size of a parent part changes all of it's "children" as well. If you shrink the WingRin for instance, the whole wing, plus engines and landing gear will shrink with it. Changing the CF_D0 will usually affect the whole plane. Speaking of the CF_D0, this is the most important part of the plane. All the other parts are connected to it, and reorienting it can sometimes cause the plane to fly incorrectly. Not all plane parts are oriented properly in the first place, so simply replacing the CF_D0 in the folder might lead to a fuselage 90 degrees off angle, etc, but changing the orientation sometimes changes the flight profile, etc.

同样要注意你改变"父模型"的方向或尺寸的话依附它的"子模型"也会跟着改变，如果你缩小机翼，那么机翼上的发动机和起落架会跟着缩小，改变CF_D0通常会影响整个飞机，说到CF_D0，它通常是整个飞机最重要的部分，其他的部分都依附于它，改变它有时会使飞机飞行不正常，通常第一次导入不是说有的部分都能准确定位，所以简单的替换CF_D0可能会导致机身偏转90度，而有时后改变方位会影响飞行轨迹，这是很让人头疼的。

RESULTS: The mild traction group showed angulus anterior individual neuron slightly engorged, neuron and nerve fiber morphous was normal essentially; individual substantia alba demyelinates in fragmentis; medullary sheath and auxiliary fibers of nerve root were hydropsia slightly without conspicuous myelinolysis. The moderate traction group demonstrated myelinolysis change in the spinal cord, and neurofibras lined up chaos, auxiliary fibers disaggregated, neuron dropsy, tigroid body disappeared with karyopycnosis and anachromasis in neurons; nerve root showed myelinolysis. The severe traction group displayed a great demyelination region; anterior motor neurons with karyopycnosis and anachromasis; myelinated nerve fiber of nerve root shrinked, decreased in number, with severe demyelination changes.

结果：轻度牵拉组牵拉侧前角个别神经元稍肿胀，神经元及神经纤维形态基本上正常，个别白质小片状脱髓鞘样改变，神经根髓鞘和轴索轻度水肿，脱髓鞘不明显；中度牵拉组白质有脱髓鞘改变，神经纤维排列紊乱，轴索崩解、断裂；灰质神经元水肿明显加重，前角运动神经原细胞尼氏体消失，核固缩、深染，神经根髓鞘和轴索水肿，局部轻度脱髓鞘改变；重度牵拉组牵拉侧白质为不规则的大片脱髓鞘区，前角运动神经元胞体固缩、变形、核深染，神经根有髓神经纤维萎缩、数目减少，重度脱髓鞘改变。

The femoral circumflex arteriography of the normal leg was done in 8 patients, and three of them were simultaneously done in a postion of internal rotating of straight leg. Results Comparison with the arteriographic findings in absence of traction, all retinacular arteries but inferior arteries were depicted few branches and less femoral head perfusion in all 9 patients, especially in that of medial femoral circumflex arteriography, as hip joint was tractating in neutral position with the same speed and volume of injecting contrast medium and, the veins were demonstrated poorly or delayed.

结果同一血管部位，同等对比剂注射流率、总量造影，与中立位无牵引状态下造影所见比较，9例患髋中立位牵引时，除外下支持带动脉，所有未损伤的支持带动脉均有不同程度闭塞，相应股骨头颈部灌注不佳或几乎无灌注，静脉显影延迟或显示不清，4例牵引力由3kg增至5kg时，上述改变更为明显；5例患髋维持伸直内旋位时，所有支持带动脉尤其细小分支未见明确显示，股骨头灌注差或未见明确灌注，改变甚于牵引时；2例正常髋内旋位时造影改变类似。

NodD binds to and bends target promoters through anchoring two tandem and individual specific DNA sites. NodD functions as a tetramer, which has a V-shaped main body. Tetrameric NodD is to change its own conformation rather than its oligomeric forms in response to small signal molecules. The specific interaction between each NodD DNA-binding domain and each specific DNA site does not alter itself in spite of naringenin induction, and the induced conformational change is transferred from protein to DNA. Only the DNA conformation incited by induced NodD is competent for RNA polymerase to form the transcriptional open complex. It cannot be excluded that NodD may have protein-to-protein contacts with RNA polymerase, and that the NodD conformational change may also directly contribute to the transcriptional open complex formation. However, the NodD conformational change itself cannot serve as the determinant of the transcriptional molecular switch.

通过研究，我们提出了初步的NodD操纵子激活模型：第一，四聚体是NodD蛋白的功能单位，它通过铆钉两个串联的相对独立的DNA靶位点结合被诱导的启动子；第二，小分子配基的结合是改变NodD四聚体的构象而不是引发不同形式的寡聚体，在我们的模型中，NodD四聚体缩小其V形主体的弯折角，进而缩短其DNA结合功能域的间距；第三，小分子信号的诱导并没有改变NodD的DNA结合域和其DNA靶位点的相互作用，NodD的构象改变由蛋白质经其双铆钉位点传递给DNA；第四，只有诱导状态的NodD激发的DNA构象才能有效地使RNA聚合酶形成转录开放复合物；第五，不排除NodD与RNA聚合酶可能有直接的相互接触位点，不排除NodD构象的改变可能直接有利于RNA聚合酶形成转录开放复合物，但是我们认为NodD构象改变本身不是充当转录激活开关的决定因素。

The only thing we can do is alerting our attitude.

我们不能改变过去，不能改变别人特定的行为方式，不能改变必然发生的事情。

In the United States, chronic alcoholism and hepatitis C are the most common ones.

在美国，慢性酒精中毒，肝炎是最常见的。

If you have any questions, you can contact me anytime.

如果有任何问题，你可以随时联系我。