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一氧化氮

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Objective To explore the effects of nitric oxide and lipid peroxidation in the cases of corneal alkali burn.

目的:探讨一氧化氮与脂质过氧化反应在角膜碱烧伤中的作用。

Nitrogen monoxide ; sulfure dioxide ; baryta/γ-alundum ; waste gas treatment

一氧化氮;二氧化硫;氧化钡/γ-氧化铝;废气处理

If the NOS activity is changed in CAM; if it has the relation with CAM amyotrophy; If it is the important component element of the CAM pathogenesis that NOS induces the oxidatant-antioxidant status imbalance in the skeletal muscle, which all hasn't been reported domestically and abroad.

比较分析慢性酒精中毒性肌病中的氧化-抗氧化状态和一氧化氮合酶的改变特征,进一步地探讨该病发病机制,为CAM的防治提供理论依据。

In order to understand the mechanism of anti-inflammatory effect of PA and carvacrol, the superoxide dismutase, glutahoine reductase, glutahoine peroxidase activities in liver and malondialdehyde , cyclooxygenase 2 (COX-2), tumor necrosis factor-α levels were eveluated. Also observed inducible nitric oxide synthase and cyclooxygenase-2 (COX-2) expression and the pathological histology in λ-carrageenan induced paw edema in mice.

本研究首先以醋酸扭体试验评估左手香水抽提物及其活性成分香芹酚是否具有镇痛效果,以福马林舔足试验辨别其镇痛作用是在周边或是中枢,再以λ-角叉菜胶诱导小鼠足跖肿胀试验探讨其抗发炎作用,并分析其肝脏组织中的超氧歧化酶、麸胱甘肽还原酶、麸胱甘肽过氧化酶活性、发炎足跖组织中的脂质过氧化指标产物丙二醛、肿瘤坏死因子-α及环氧化酶-2之含量变化,观察诱导型一氧化氮合成酶及环氧化酶-2之蛋白质表现量,并将肿胀足跖做病理切片观察之,以探讨左手香水抽提物及香芹酚之抗发炎机转。

Subject: The purpose of this.thesis is to explore the therapeutic mechanism of Radix Astragli injection from the point of view of modern medicine through studying its clinical therapeutic effect on patients with chronic glomerulonephritis characteristic of "qi" deficiency in lung - kindney , and its effect on urine protein, plasma albumin, plasma cholesterol, plasma creatinine, urea, hemodynamies urine2-microgiobulin , T lymphocyte , plasma endothelin , nitric oxide .

目的:本文通过研究黄芪注射液对肺肾气瘀证慢性肾小球肾炎患者的临床治疗效果以及对蛋白尿、血浆白蛋白、血浆胆固醇、血肌酐、尿素氮、尿β_2-微球蛋白(β_2-MG)、血流变学、T细胞亚群、血浆内皮素、一氧化氮的影响,从现代医学的角度探讨其治疗作用机理。

Chinensis and Kuruma prawn M. japonicus increased at 12 and 48 hours post-inoculum, but decreased sharply after 36 and 72 hours post-inoculum respectively. LPS inducible NOS activity also decreased significantly with the time elapsed in WSSV infected shrimps. Results of WSSV detection with the Dig-DNA Probe dot blot hybridization test showed that WSSV of the two penaeid species couldn't be detected until iNOS started decreasing.

在此基础上,我们建立了对虾血细胞中诱导型一氧化氮合成酶的鉴定方法—硝基四氮唑蓝还原法和血细胞形态法以及酶活力的测定方法—L-瓜氨酸分析法和亚硝酸盐分析法并优化了鉴定和测量条件,结果显示:对虾血细胞在4℃下与L-精氨酸和LPS孵育8h以及当L-精氨酸浓度为2.5mmol l〓LPS浓度为100μg ml〓,Ca〓浓度为2.5mmol l〓时,iNOS活力的测定结果最佳。

In the cardiovascular system, the endogenous NO, CO, and H2S form three specific and relevant pathways with their corresponding enzymes including nitric oxide syntheses, the home oxygenize and cystathionine-r-lyric enzyme, whose affect can relieve the atherosclerosis.

血管系统中内源性气体信号分子NO、CO、H2S作为一种独特的血管活性物质,分别与其相应的合成酶一氧化氮合酶、血红素加氧酶和胱硫醚-r-裂解酶形成独立而又相互关联的体内重要抗氧化系统,可以减少动脉粥样硬化的严重度。

The rate of enteric transit was measured, and the histological changes in nitriergic nerves in intestinal myenteric plexus of scalded rat were observed as well. The densities of neurons and ganglions with positive nitric oxide synthetase as well as ICC in duodenal myenteric plexus were assessed for both groups.

用酶组织化学方法观察回肠肌间神经丛内氮能神经的组织学改变,并对两组大鼠回肠肌间神经丛内一氧化氮合酶阳性神经元、神经节和Cajal间质细胞的密度进行测定。

Chemical antagonism between superoxide anions generated by NADPH oxidases may be the molecular mechanism responsible for the inactivation of endogenous NO following treatment with high-dose morphine.

由NADPH 氧化脢产生的过氧化离子之拮抗作用可能用高剂量吗啡后内源性一氧化氮去活化的分子机转。

Methods the models of epilepsy were made by intraperitoneal injection of pentylenetetrazol and the contents of no,mda and sod of all the epileptic hippocampus were assayed and dealt with all data by statistical analysis.

健康sd大鼠38只,分5组,采用戊四氮腹腔注射制作癫痫模型,测定各组大鼠海马一氧化氮、丙二醛的含量和超氧化物岐化酶的活性。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。