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The antibiotic overuse on viral, fungal or other nonbacterial dog disorders is alarming!

抗药性overuse 在病毒,霉菌或其他nonbacterial 狗混乱是惊心的!

Now that nobody gets "nonparalytic polio" anymore since there are vaccines against the 4 paralytic polio viruses (I don't understand the logic either, but the diagnosis has completely disappeared) we all just have mysterious viral illnesses that leaves us with what would be diagnosed as post polio syndrome if we had been diagnosed with polio first but otherwise it's just FMS and CFIDS.

现在,没有人获得&nonparalytic小儿麻痹症&了,因为有4对麻痹性脊髓灰质炎病毒疫苗(我不明白的逻辑下去,但诊断已完全消失),我们都只是具有不可思议的病毒性疾病是留给我们的将是什么脊髓灰质炎后综合征的诊断,如果我们被诊断患有小儿麻痹症的第一步,但否则只是FMS和CFIDS。

It is usually a nonprogressive disease and can be caused by viral infections, drugs, vitamin A deficiency, or genetics.

该疾病通常是非渐进性的,可由病毒感染、药物、维生素A缺乏或遗传因素引起。

It is usually a nonprogressive disease and can be caused by viral infections, drugs, vitamin A deficiency, or genetic defects.

该疾病通常是非渐进性的,能由病毒感染,药物,维生素A缺乏或遗传缺陷所引起。

While AcMNPV and BmNPV had no stimulatory effect in nonsensitive cell lines BmNPV hr3 has been demonstrated to increase the transcriptional activity of cislinked hel510 promoter. In this study, Sf-21, Bm-N and Hi-5 cell lines were transfected with pBmhel510luc-hr3down, and then were infected with each of AcMNPV, BmNPV and HyNPV. The results indicated that viral factors could interact with hr3 to significantly copromote the transcriptional level of hel510 promoter.

BmNPV hr3已被证明具有增强子功能,将hr3与hel510启动子顺式连接的质粒pBmhel510luc-hr3down转染Sf-21、Bm-N和Hi-5细胞后,再用上述三种病毒分别感染每一种细胞,hel510启动子的转录活性得到显著提高,表明病毒因子能够识别hr3,二者通过相互作用,共同增强解旋酶基因启动子的转录水平。

These results suggested that RBSDV-S6 encoded a viral nonstructural protein.

此结果表明,RBSDV-S6编码的蛋白是一种非结构蛋白。

A century ago, children received one vaccine, smallpox, which contained about 200 structural and nonstructural viral proteins.

一个世纪以前,孩子们只有一种疫苗,这就是天花疫苗,含有200多种病毒的结构或非结构蛋白。

He SPRY domain of TRIM5α, which may be responsible for recognition of incoming viral capsids showed higher nonsynonymous/synonymous substitution ratios than the non-SPRY domain, indicating that the adaptive evolution of TRIM5α in primates might be an innate strategy developed in defending retrovirus infection during primate evolution.

RIM5α的 SPRY 结构域可能负责识别入侵病毒的衣壳,这个结构域比基因的其它部分在进化过程中具有更高的错义突变同义突变比率,表明 TRIM5α的适应性进化可能是灵长类发展抗反转录病毒感染策略的结果。

First 46 viral sequence's PA fragments in year 2009 are analyzed. Then the siRNA target genes with strong ability of interference are selected on the basis of the secondary structure of siRNA target sequence. Our research reveals that the outbreaking H1N1 virus in 2009 is characterized by steady heredity, high sequence conservativeness, uniform siRNA target gene sequence, and high conservative structure.

对2009年的46株病毒序列的PA片段进行分析,从经过序列分析所获得的众多靶系列中,采用结构分析手段对靶序列进行筛选,获得较易干扰的靶序列及设计出相应的siRNA。

The section covers metabolic, membrane, receptor and immunological disorders as well as viral, bacterial and parasitological diseases, and includes the biochemistry of differentiation disorders, tissue damage and aging.

其内容涵盖代谢,生物膜,受体和免疫失调,病毒、细菌和寄生虫疾病、各种失调、组织损伤以及与老化有关的生物化学等。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。