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tunica intima相关的网络例句

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Results:Distribution of mast cells in study group was that minority mast cells were observed in the intima and media, but the more were observed to gather in the adventitia.

结果:研究组肥大细胞的分布,内膜或中膜均可见少数肥大细胞,外膜则较多聚集。

Experimental methods: pathological slices: To observe the changes of pathomorphology and measure the circular area of internal elastic lamina , area of neonate intima and vascular lumen, the intimal proliferatory index and percentage, the index of lumen stenosis and positive index of proliferating cell nuclear antigen through the specimens stained by HE and immunohistochemistry.

实验方法:病理切片:进行HE染色和免疫组织化学染色,分别观察病理形态学改变;测定和计算受损血管内弹力膜环绕总面积、新生内膜面积、管腔面积、内膜增生指数、内膜增生百分比、管腔狭窄指数和增殖细胞核抗原阳性指数;观察MMP-2、MMP-3、TIMP-1和PDGF-BB的免疫组化表达。

Endovascular restenosis is an abnormal repairing response of vessel against injury. Following stent implantation, vessel contractibility reconstitution, local inflammation and thrombogenesis, intima hyperplasia, mainly vascular smooth muscle hyperplasia, are found.

血管内再狭窄是血管对抗损伤的一种异常修复反应,表现为支架植入后血管的收缩性重构,局部的炎症与血栓形成,以及以血管平滑肌细胞增殖为主的内膜增生。

The ultrastructure resultshowed that in model group blood intima was adema,broadening, evendiscontinuous and incomplete under blood vessel bast. In the blood themonocyte and the lymphocyte infiltrated to intension board. Manymacrophages could befound and there were massive all levels of dissolve theenzyme body and swallow slightly soak in its cytoblastema,The endosarc netand the ribose body was increased and outside the amnion the pseudopodappeared. Endothelial cell morphogenesis changed. It is obvious thatmitochondria was dropsy, cytoblastema was non-uniformity.

超微结构结果显示模型组血管内皮下内膜水肿、增宽,甚至间断不完整;血中单核细胞及淋巴细胞向内弹力板浸润,可见较多巨噬细胞,其胞浆内含大量各级溶酶体及吞噬小泡,内质网及核糖体增多,胞膜外出现伪足;内皮细胞形态发生改变,可见线粒体水肿,胞浆不均匀;中药治疗后,上述病理损害明显减轻。

Abstract] objective to identify the relationship between serum homocysteine and carotid artery atherosclerosis in patients with cerebral infarction and effects of foliate,methylcobalamin.methods the concentrations of hcy were determined by means of fluorescence polarization immunoassay and carotid arteries was examined with color doppler ultrasound in 165 patients with cerebral infarction.the subjects were divided into 4 groups according to the severity of lesion of carotid artery.the concentrations of hcy were compared in different groups.the patients with hyperhomocysteinemia received foliate and methylcobalamin.results when lesion of carotid artery became severer,serum hcy was higher.the intima medial thicknesses of carotid artery were significantly decreased in patients with hyperhomocysteinemia who received foliate and methylcobalamin for two years.conclusion hyperhomocysteinemia may play an important role in development of carotid artery atherosclerosis.the intervention of foliate and methylcobalamin may reduce carotid artery atherosclerosis.

摘要] 目的探讨脑梗死患者颈动脉病变与血清同型半胱氨酸水平的关系及叶酸、甲钴胺长期干预对其影响。方法对165例急性脑梗死患者进行血清hcy的测定及颈动脉超声检查,并按动脉硬化的程度分组,比较不同程度颈动脉粥样硬化患者hcy水平,部分高hcy血症患者给予叶酸、甲钴胺干预,随访2年。结果随着颈动脉病变程度的加重,血清hcy浓度呈上升趋势,高hcy血症干预者动脉内中膜厚度明显减小。结论高hcy血症对颈动脉粥样硬化的形成起重要作用,长期给予叶酸、甲钴胺干预可能减轻颈动脉粥样硬化。

By location of in situ hybridization, positive location was strong in intima of hyperlipemia rabbits at 8 week.

IL-8的蛋白和基因上调主要是在高脂血症兔动脉粥样硬化模型的脂纹期。

Results In the ANCA negative group, 2 cases were without malignant hypertension ,1 was acute tubular necrosis caused by herbs, 6 were scleroderma renal crisis, including 4 with renal biopsy, indicating hypertrophic arterial media, edema, thickened intima, onion- skin lesion in interlobular arteries and afferent arterioles, as well as ischemic lesion in glomeruli.

结果 11 例SSc急性肾损伤患者中,ANCA阴性9 例,6 例临床表现为硬皮病肾危象,其中4 例肾活检示小叶间动脉和入球小动脉内皮细胞增生、肿胀,内膜黏液性水肿,内弹力纤维断裂,"洋葱皮样"改变和肾小球弥漫性缺血;无恶性高血压表现2 例;急性肾小管坏死1 例。

BACKGROUND: More and more researches indicated that inflammation starts formation of new intima, resulting in restenosis.

背景:越来越多的研究提示炎症启动了新内膜的形成,最终导致再狭窄。

Therefore, basic study of mechanism about proliferation of the intima and VSMC has become a focus.

因此内膜增生的机制及VSMC增殖的基础研究已为众人瞩目。

Aortic Intima Media Thickness is an Early Marker of Atherosclerosis in Children with Type 1 Diabetes Mellitus.

主动脉内膜厚度是1型糖尿病儿童患动脉粥样硬化的早期标志之一

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