查询词典 trophocyte
- 与 trophocyte 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Methods: The expression of survivin in the trophocyte of 14 cases of normal early gestation villi, 14 cases of hydatidiform mole, 12 cases of invasive hydatidiform mole and 12 cases of choriocarcinoma were detected by immunohistochemistry test.
采用免疫组化SP法检测14例正常早孕绒毛、14例葡萄胎、12例侵蚀性葡萄胎及12例绒癌滋养细胞中survivin的表达。
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This study firstly discover and denounced that hCG is not only an important endocrine hormone in pregnancy, but also an important autocrine hormone for trophocyte and JAR cell. It directly influence cellular proliferation and endocrine function of JAR or trophoblastic cells.
六、本研究首次揭示并提出了hCG不仅是重要的妊娠期内分泌激素,而且对于JAR细胞而言,可能也适合正常妊娠滋养细胞,hCG是重要的自分泌激素,直接影响JAR细胞或滋养细胞的生长增殖与内分泌功能,可能也是必须的在局部产生作用的自分泌激素之一。
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Methods: Paraffin section HE stain, light microscopy calculate the area of trophocyte area rate.
石腊切片,伊红、苏木素染色,光镜分析,计算滋养细胞面积比率。
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In normal pregnancy, trophocyte secrete varies hormones, besides hCG, it participate in synthesis of steroid hormones, hPL and so on. JAR cell posses the function of secretion such hormones.
三、正常妊娠时滋养细胞分泌多种激素,除了hCG以外尚参与了类固醇激素,hPL等多种激素的合成与分泌,JAR细胞也具有分泌这些激素的功能。
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There was no adverse effect. It showed that Bushen Yangtai Pill could increase the cure rate of development and functional defect of trophocyte effectively. The mechanism of action may relate with the improvement of womb and ovarian, the promotion of growth and development of the blood current of womb and ovarian, and precipitate of secretion of yellow body of ovary and endometrial gland.
说明补肾养胎丸可明显提高滋养细胞发育和功能不全的治愈率,作用机理可能与改善子宫与卵巢的血流,促进卵巢及子宫的生长发育,促使卵巢黄体及子宫内膜腺体的分泌有关。
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From this study, hCG is a autocrine hormone for trophocyte, as a kind of autocrine hormone, hCG regulate trophoblastic tumor strain JAR cellular 〓 and P secretion at least in vitro.
从本研究结果看,hCG对滋养细胞而言也是一种自分泌激素,作为一种自分泌激素,至少调节着滋养细胞肿瘤细胞株JAR细胞的〓、P的分泌。
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From this study, both low hCG and high hCG are adversity to pregnancy, hCG level reflect the growth and function situation of trophocyte which is the main component of placenta, whereas, the level of steroid hormone which close to hCG directly influence whether pregnancy will maintain, but in some cases being short of oxygen in uterine placenta local, usually display hCG level higher than normal pregnancy.
在正常妊娠时hCG过低往往预示着流产的可能,从本研究结果看,可能不仅仅是hCG水平过低对妊娠不利,hCG水平很高对妊娠也不利。hCG水平的高低反映了作为胎盘主要组份的滋养细胞的生长情况与功能,而与hCG水平高低密切相关的类固醇激素的高低直接影响了妊娠能否持续下去。
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That because dyoxygen induced trophocyte proliferation and hCG secretion, at that moment, hPL level is not high, which is consistent with hPL level in placenta dysfunction, What this study show that hCG increase being not promote hPL secretion significantly may explain the phenomenon.
而在一些妊娠晚期子宫胎盘局部缺氧的病例,往往表现为hCG水平高于正常相同孕周妊娠者,这是因为缺氧诱发了滋养细胞的增生,hCG大量分泌,但此时hPL水平不高,是与胎盘功能不良时hPL水平往往不高相一致,本研究结果显示的hCG增高并不明显促进hPL的分泌可能可以解释这一现象。
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The activation of endothelial cell caused by TNF-α has detrimental effects on the vasculature. TNF-α can induce the increased apoptosis of gravid trophocyte. TNF-α has been showing up-regulate the vasoconstriction factors such as endothelin and down-regulate the vascular relaxing factors such as NO on the contrary.
肿瘤坏死因子α作为炎性分子,激活血管内皮细胞、间接影响血管舒缩功能,通过各种机制诱导胎盘滋养细胞凋亡增加,增加血管收缩因子并减少血管舒张因子的生成和释放,影响与脂类代谢相关的细胞因子。
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Measure normal early pregnancy choria tissue and JAR cell by RT-PCR, LH/hCG receptor mRNA expression on both pregnancy choria tissue and JAR cell which is consistent with that be measured in ovarian granulosa cells, indicate that JAR cell and normal choria trophocyte presence LH/hCG receptor, hCG is one of the autocrine hormone.
二、用RT-PCR法可测到正常妊娠早孕绒毛组织、JAR细胞的LH/hCG受体mRNA表达,与正常卵巢颗粒细胞所表达的LH/hCG受体mRNA一致,提示JAR细胞与正常绒毛滋养细胞存在LH/hCG受体,hCG为其自分泌激素之一。
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。