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treatment group相关的网络例句

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ABSTRACT:The therapeutic group was compared with 5 control groups, which were normal group, ventolin treatment group, ion-guide group, external application group and model group.

用复方麻辛酊敷贴离子导入疗法对哮喘豚鼠进行治疗,并与正常组、喘乐宁对照组、离子导入组、敷贴组、模型对照组相比较,观察各组动物白蛋白激发后气道阻力和肺顺应性的变化。

The treatment of acquired cold urticaria with Huangqi Injection in the acupoint of Zusanli (ST36) combined with antihistaminic in the treatment group was compared with that in the control group with antihistaminic only.

方法用黄芪注射液于足三里处进行穴位注射配合抗组胺药物治疗获得性寒冷性荨麻疹并与单纯使用抗组胺药物治疗进行对比。

Objective To observe the recent effect of treatment by wags of combination of traditional Chinese medicine and western medicine in patients with atonic constipation.Methods Under the same diet,206 volunteer patients were divided at random into two groups-treatment group(104patients)and contrast group (102 patients).

目的 探讨老年性便秘患者采用中西医结合治疗的近期疗效方法在基本相同膳食的提下,对自愿参与206例老年性便秘患者随机分治疗组与对照组,共2组。

12H treatment group compared with 12h model group: the density of light of HSP70 of the Gardenoside group is not different and which of it of TGFβ〓 increased with the model group; the density of light of the HSP70 of the Baicalin group in not different and which of the TGF-β〓 is no different with the model group, the density of HSP70 of the Concha Margatitifera Usta is not different and which of TGFβ〓 increased than the model group; the density of the HSP70 of the Cholic Acid group is no different and the density of TGF-β〓 increased than the model; the density of the light of HSP70 of the Hefang Group increased and which of TGF-β〓 increased than the model group.

合方治疗脑缺血12小时组缺血脑组织TGF-β1光密度值较缺血12小时模型组明显升高(p<0.01)。大鼠局灶性脑缺血24小时模型组缺血脑组织TGF-β1光密度值含量较正常组明显增高(p<0.01)。珍珠母治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。胆酸治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。栀子苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组无显著性差异(p>0.05)。黄芩苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高(p<0.01)。

12H treatment group compared with 12h model group: the level of MCP-1 of the Gardenoside group decreased and MIP-1 is no different with the model group; the level of the MCP-1, MIP-1 of the Baicalin group are no different with the model group; the level of MCP-1, MIP-1 of the Concha Margatitifera Usta decreased than the model; the level of MCP-1 of the Cholic Acid group is no different and the level of MIP-1 decreased than the model; the level of MCP-1 of the Hefang Group decreased and the level of MIP-1 of it decreased than the model group.

合方治疗脑缺血24小时组缺血脑组织nNos光密度值较缺血24小时模型组明显降低(p<0.01)。细胞的死亡分为坏死和凋亡两种方式,凋亡是细胞为避免对机体造成更大损伤而采取的一种程序性的死亡方式。珍珠母、胆酸、栀子苷、黄芩苷、合方治疗组较脑缺血24小时模型组可明显降低P53阳性表达,考虑药物抑制细胞凋亡可以起到对缺血脑组织的保护作用。

12H treatment group compared with 12h model group: the level of SOD, MDA of the Gardenoside group is no different with the model group; the level of the SOD of the Baical in group increase and the level of MDA is no different; the level of SOD of the Concha Margatitifera Usta is no different and the MDA of it decreased than the model; the level of SOD of the Cholic Acid group increased and the level of MDA decreased; the level of SOD of the Hefang Group increased and the level of MDA of it decreased than the model group.

考虑nNos可能只是参与缺血早期的神经毒性作用。到缺血24小时这一时程已经减低或耗竭。大鼠局灶性脑缺血12小时模型组缺血脑组织P53光密度值含量较正常组明显增高(p<0.01)。珍珠母治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低。胆酸治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低(p<0.05)。栀子苷治疗脑缺血12小时组缺血脑组织P53光密度值较缺血12小时模型组明显降低(p<0.05)。

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中,脑缺血能够诱导P53蛋白表达,P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程,药物通过抑制细胞凋亡,从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高(p<0.05)。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异(p>0.05)。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异(p>0.05)。

12H treatment group compared with 12h model group: the density of light of nNos of the Gardenoside group decreased and P53 decreased with the model group; the density of light of the nNos of the Baicalin group decreased and which of the P53 is no different with the model group, the density of nNos of the Concha Margatitifera Usta decreased and which of P53 decreased than the model; the density of of nNos of the Cholic Acid group is no different and the density of P53 decreased than the model; the density of the light of nNos of the Hefang Group is no different and which of P53 decreased than the model group.

珍珠母治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异(p>0.05)。胆酸治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异(p>0.05)。栀子苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高(p<0.01)。黄芩苷治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组明显增高(p<0.01)。合方治疗脑缺血24小时组缺血脑组织HSP70光密度值较缺血24小时模型组无显著性差异(p>0.05)。

Methods40 adult male SD rats were used in this study and divided into 4 groups at random: normal group, aging model group, TNY treatment group, and negative control group, each group had 10 rats. The subacutely aging rats were made by injecting Dgal into the rats' abdominal cavity continuosly. The learning and memory ability of rats in each group were detected with Morris water maze. The monoamine transmitter contents in hypothalamus and hippocampus were detected with HPLCECD.

方法选用成年雄性SD大鼠40只,随机分为4组,每组10只:正常组、衰老模型组、TNY用药组、阴性对照组。D半乳糖连续腹腔注射制作亚急性衰老的大鼠模型,采用Morris水迷宫检测各组大鼠的空间学习记忆能力及采用高效液相色谱电化学法检测各组大鼠下丘脑、海马NE,DA,5HT的含量。

Methods:The 62 patients with HSP from 1999 to 2002 were randomly divided into the heparin calcium treatment group (32 cases) and the non-heparin calcium treatment group (30 cases). At the same time, 25 cases of non-HSP belonged to the normal control group. Alb, 13 2-MG and IgG of urine were monitored constantly.

将62例过敏性紫癜患儿随机分成肝素钙治疗组(32例)和对照治疗组(30例),另设非HSP对照组25例,追踪监测尿微量白蛋白,尿β2-微球蛋白(β2-MG),尿免疫球蛋白G,从而判断肾损害的情况。

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