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thymocytes相关的网络例句

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The supplementation of nucleotides can accelerate the repair of damaged DNA in mouse thymocytes in vitro.

外源核苷酸能明显促进受损小鼠胸腺细胞DNA的修复,且核苷酸的作用与其添加水平有关。

The changes of caspase- 3 protein and the expression of p38MAPK proteins were detected by western blotting at 0,30,60 and 180 min after dexamethasone- induced thymocytes apoptosis.

运用 Western blotting技术检测地塞米松处理的 Balb/c小鼠胸腺细胞 0, 30, 60, 180 min时相点 caspase- 3蛋白与 p38MAPK的表达变化。

Results The results showed that Cd might cause time-and dose-dependent apoptosis in primary cultured thymocytes.

结果镉能引起原代培养的胸腺细胞发生凋亡,并呈时间-反应和剂量-效应关系。

The immunoreactive nerve fibers for every peptide were seen, and all of them contacted closely with the thymocytes.

结果:各神经肽的免疫反应性主要位于髓质,均可见到阳性神经纤维,并与胸腺细胞密切接触。

Aim To investigate the gene regulation effect during the thymocytes' apoptosis induced by candida albicans.

目的 探讨白色念珠菌诱导小鼠胸腺细胞凋亡过程中的基因调控作用。

The effect of ionizing irradiation on IL-12 expression in thymocytes, splenocytes and macrophages.

鉴于IL-12在免疫反应中起着至关重要的作用,已被越来越多的学者所关注。

Objective:To study the relationship between the expression of Fas/Fas-L or HSP70 and the heat stress-induced apoptosis of thymocytes in vitro.

目的:探讨体外热应激诱导胸腺细胞凋亡与细胞表达Fas、Fas-L及HSP70的关系。

Three days later, circulating leukocytes, splenocytes, and thymocytes were collected for immune analysis.

实验进行三天后收集血液、脾脏及胸腺进行免疫分析。

Fas and Fas-L are both important molecules which mediate heat stress-induced apoptosis of thymocytes in vitro.

HPS70是热应激胸腺细胞损伤的标志,Fas与Fas-L是介导热应激诱导胸腺细胞凋亡的重要分子。

Purpose To compare the dynamic changes of CD4(superscript +)CD25(superscript +) thymocytes development in vivo and in vitro.

目的 比较研究小鼠体内外胸腺细胞CD4CD25双阳性T细胞发育的动态变化,为天然调节性T细胞的研究提供基础数据。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。