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tachycardiac相关的网络例句

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与 tachycardiac 相关的网络例句 [注:此内容来源于网络,仅供参考]

Methods 72 critically illness with tachycardiac arrhythmia received intravenous amiodarone and oral amiodarone were included in this study.

目的 评价合并快速型心律失常的危重患者静脉应用胺碘酮的疗效及安全性。

It is efficacy and safety of intravenous amiodarone of the therapy for critically illness with tachycardiac arrhythmia,especially for whom with heart failure.

合并快速型心律失常的危重患者,特别是合并心力衰竭的患者,静脉应用胺碘酮是安全的,疗效是确切的。

Atrial fibrillation and sinus tachycardiac may worsen hemodynamic change and heart function.

房颤和窦速加重血流动力学改变和使心功能恶化。

On the basis of the above studies, the effects and mechanisms of microinjection of glucocorticoid or mineralocorticoid into the rVLM on the arterial blood pressure, HR, pressor and tachycardiac responses produced by stimulation of the dorsal periaquaductal gray in the midbrain were further observed and analysed in the acute experiments on normotensive rats.

在此基础上,进一步在急性动物实验中观察分析肾上腺糖、盐皮质激素在rVLM区对血压、心率、电刺激防御反应区诱发的防御性升压和加快心率反应的作用及其机制。

Microinjection of glucocorticoid or mineralocorticoid (40 or 10 ng/0.1 ul/site) into the rVLM bilaterally caused an increase in BP, HR and the pressor response induced by the dPAG stimulation in the normotensive rat, respectively. Dose-dependence of both drugs was observed on the above effects in three directions. Nevertheless, there was no effect of both drugs on the tachycardiac response induced by the dPAG stimulation.

二、分别微量注射糖、盐皮质激素(40或10ng/0.1μl/site)至双侧rVLM,可引起正常血压大鼠的BP升高,HR加快,以及电刺激中脑中央周围灰质背侧区诱发的防御性升压反应增强,并均呈现出剂量-效应依赖性关系,而对电刺激dPAG诱发的加快心率反应无明显改变作用。

After microinjection of the antagonists of glucocorticoid RU 38486 or mineralocorticoid spironolactone (40 ng/0.1 ul/site, respectively) into the rVLM bilaterally, a decrease in BP, HR and the pressor response induced by the dPAG stimulation was observed in the normotensive rat, and there was no change on the tachycardiac response induced by the dPAG stimulation.

分别微量注射糖、盐皮质激素的拮抗剂RU 38486(40ng/0.1μl/site)或螺旋内酯(40ng/0.1μl/site)至双侧rVLM,则引起正常血压大鼠的BP下降、HR减慢,以及电刺激dPAG诱发的防御性升压反应减弱,对电刺激dPAG诱发的加快心率反应无明显改变作用。

On the normotensive rat, bilateral microinjection of L-arginine (16 n mol/0.1 ul/site), a precursor of nitric oxide into the rVLM, caused a decrease in BP, HR and the pressor response induced by the dPAG stimulation, while microinjection of NG-methyl-Larginine (L-NMMA, 16 n mol/0.1 ul/site), a blockade for NO to form, into the rVLM bilaterally, caused an increase in BP, HR and the pressor response induced by the dPAG stimulation. There was no effect of both drugs mentioned above on the tachycardiac response induced by the dPAG stimulation.

五、在正常血压大鼠,于双侧rVLM微量注射一氧化氮前体(16nmol/0.1μl/site)可引起BP降低、HR减慢、以及电刺激dPAG诱发的防御性升压反应减弱;而于同区微量注射生成一氧化氮的拮抗剂N〓-甲基-L-精氨酸(16nmol/0.1μl/site)则可引起BP升高、HR加快、以及电刺激dPAG诱发的防御性升压反应增强;而上述两药对电刺激dPAG诱发的防御性加快心率反应均无明显作用。

Results During isoprenaline administration the heart rate of arrhythmia in6cases went faster gradually and They were diagnosed as chronic sustained junctional tachycardia(3cases)and chronic atrial tachycardia(2cases)and sinoatrial reentrant tachycardia(1case)respectively;while the arrhythmias in other9cases disappered and turned to sinus tachycardiac and several minites after isoprenaline administration the arrhythmia in5cases recurred again,As a reˉsult,they were diagnosed non-paroxysmal ventricular(3cases)and non-paroxysmal junctional tachycardia(6cases)respectively.

结果 6例患者心动过速的频率逐渐增快,分别诊断为慢性持续性交界性心动过速(3例)、慢性房性心动过速(2例)和窦房结折返性心动过速(1例);另9例异位心律失常消失,转变为窦性心动过速,停止滴注后数分钟5例再次出现原心律失常,诊断为非阵发性室性心动过速(3例)和非阵发性交界性心动过速(6例)。

Results During isoprenaline administration the heart rate of arrhythmia in6cases went faster gradually and They were diagnosed as chronic sustained junctional tachycardia(3cases)and chronic atrial tachycardia(2cases)and sinoatrial reentrant tachycardia(1case)respectively;while the arrhythmias in other9cases disappered and turned to sinus tachycardiac and several minites after isoprenaline administration the arrhythmia in5cases recurred again,As a reˉsult,they were diagnosed non-paroxysmal ventricular(3cases)and non-paroxysmal junctional tachycardia(6cases)respectively.

结果 6例患者心动过速的频率逐渐增快,分别诊断慢性持续性交界性心动过速(3例)、慢性房性心动过速(2例)和窦房结折返性心动过速(1例);另9例异位心律失常消失,转变为窦性心动过速,停止滴注后分钟5例再次出现原心律失常,诊断为非阵发性室性心动过速(3例)和非阵发性交界性心动过速(6例)。

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