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synovia相关的网络例句

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与 synovia 相关的网络例句 [注:此内容来源于网络,仅供参考]

BACKGROUND: Early rehabilitation following total knee replacement has arisen more attention. The aim of continuous passive joint motion is to recover knee function, prevent anchylosis, accelerate blood and synovia circulation, therefore, enhance tissue repair.

摘要背景:膝关节置换后的早期康复治疗逐渐引起关注,进行持续被动关节运动的主要目的是恢复膝关节功能,防止关节僵硬,同时促进手术部位血液和关节滑液循环,从而加快组织修复。

In order to find the potential factors resulting in significant improvements in the lubricating be- havior of a compound artificial synovia , which was prepared by ourselves with hyaluronic acid mainly and some proteins and phospholipids, the technology of Atomic Force Microscope was used to observe the appearance of adsorbed film on material surface formed by the compound artificial synovia .

用原子力显微技术观测复合人工滑液在材料表面形成的吸附膜的表面形貌图像,吸附膜上清晰地显示有大量的椭球形分子。

Optic and electron microscopic examinations were conducted to observe the pathological changes of articular cartilage and synovia and Mankin's scale were performed.

观察3组滑膜组织的光镜、电镜改变及软骨的光镜、基质金属蛋白酶-1(MMP-1)免疫组化改变,比较软骨Mankin′s评分及关节液中IL-1的浓度。

Methods TGFβ1 protein in 53 cases of RA synovia [including 26 cases of RA,20 cases of control non-RA (14 cases of OA and 6 cases of traumatic arthritis) and 7 cases of normal synovia] was detected by immunohistochemical LSAB method.

目的 了解转化生长因子β1(TGFβ1)在类风湿关节炎滑膜组织中的表达及与病理改变的关系,以探讨TGFβ1在RA发病中的作用。

Methods TGFβ1 protein in 53 cases of RA synovia [including 26 cases of RA,20 cases of control non-RA (14 cases of OA and 6 cases of traumatic arthritis) and 7 cases of normal synovia] was detected by immunohistochemical LSAB method.

采用免疫组织化学LSAB法检测了53例(其中26例RA,20例其他关节炎和7例正常人)滑膜组织中TGFβ1蛋白的表达情况,并用目镜网格测微尺和图像分析系统分别进行TGFβ1阳性细胞的半定量计数和TGFβ1染色强度及面积的定量分析。

The results showed enhanced expression of TGFβ1 protein in RA synovia and it may be related with active pathological changes in RA synovia including synoviocyte hyperplasia, inflammatory cell infiltration,sublining angiogenesis and granuloma formation.

RA滑膜TGFβ1蛋白合成、分泌增多,并且增多的TGFβ1可能与RA滑膜病变活动有关,包括与衬里层滑膜细胞增生、炎症细胞浸润、衬里下层血管增生及肉芽组织形成有关。

The results showed enhanced expression of TGFβ1 protein in RA synovia and it may be related with active pathological changes in RA synovia including synoviocyte hyperplasia, inflammatory cell infiltration,sublining angiogenesis and granuloma formation.

RA滑膜TGFβ1蛋白合成、分泌增多,并且增多的TGFβ1可能与RA滑膜病变儿童活动有关,包括与衬里层滑膜情人细胞增生、炎症运动情人细胞浸润、衬里下层血管增生及肉芽组织形成有关。

Methods: Set up the CIA model. Do HE dyeing and the immunohistochemistry attaining on sections of the synovia. Test the expression of necrosis factor alpha and cyclooxygenase-2 in the synovia.

制备CIA大鼠模型,取双膝关节滑膜组织病理切片行HE染色和免疫组化染色,观察滑膜血管新生等病理改变,检测大鼠膝关节滑膜组织TNF-α和COX-2的表达。

Estrogen deficiency can increase the mRNA expression of MMP-1、MMP-3 and IL-1β in OA synovia tissue. Estrogen could inhibit the mRNA expression of MMP-1、 MMP-3 and IL-1β in OA synovia tissue. Progestin could even decrease this expression. Estrogen could slightly increase the mRNA expression of TIMP-1. It is suggested that low dose of estrogen supplement play a protective effect on OA cartilage, and progestin could increase this protective effect.

雌激素缺乏可使OA关节滑膜中MMP-1、MMP-3和IL-1β mRNA表达增加,雌激素对关节滑膜组织中MMP-1、MMP-3和IL-1β mRNA表达有抑制作用,孕激素可增强雌激素的这一抑制作用,雌激素对TIMP-1有轻度升高作用,提示低剂量雌激素对OA关节软骨有一定保护作用,而加用孕激素这一保护作用增强。

The results showed enhanced expression of TGFβ1 protein in RA synovia and it may be related with active pathological changes in RA synovia including synoviocyte hyperplasia, inflammatory cell infiltration,sublining angiogenesis and granuloma formation. Arthritis,rheumatoid; Transforming growth factor beta; Synovitis

RA滑膜TGFβ1蛋白合成、分泌增多,并且增多的TGFβ1可能与RA滑膜病变活动有关,包括与衬里层滑膜细胞增生、炎症细胞浸润、衬里下层血管增生及肉芽组织形成有关。

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