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Cloning and identification of minor subunit fedF gene of fimbriae F18ab from Escherichia coli

放射性标记单克隆抗体有直接法和用双功能联结剂的间接法。

The high molecular weight glutenin subunit composition of 49 wheat varieties were fractionated by SDS-PAGE. The results showed that the frequency of subunits 5+10 was 64% in 39 Chinese wheat varieties.

利用SDS-PAGE电泳分析了49个国内外小麦品种的高分子量谷蛋白亚基组成，结果表明：39个国内小麦品种中，5+10亚基的频率达64%。

Subunit network composed by manifold s and laterals which are all multi-diameter pipes is the important part of micro-irrigation system.

微灌是一种先进的节水灌溉技术，其田间管网是微灌系统的重要组成部分，组成田间管网的支管和毛管均为多孔出流管。

Objective: To study the relationship between luteinizing hormone β-subunit gene Gly102Ser polymorphism and polycystic ovary syndrome and premature ovarian failure.

目的：探讨黄体生成素β亚单位基因Gly102Ser多态性与中国妇女多囊卵巢综合征及卵巢早衰的关系。

Dysregulation of the protein kinase A system, caused by mutation of the protein kinase A regulatory subunit gene PRKAR1A, causes the inherited tumor syndrome Carney complex, which includes cardiac myxomas as one of its cardinal features.

蛋白激酶A调节亚基因PRKAR1A的变化引起蛋白激酶A系统失调，导致了遗传性卡尼复合肿瘤综合症，其中包括了心脏粘液瘤作为它的一个主要特征。

BACKGROUND: Protein kinase A signaling has long been known to play an important role in cardiac function. Dysregulation of the protein kinase A system, caused by mutation of the protein kinase A regulatory subunit gene PRKAR1A, causes the inherited tumor syndrome Carney complex, which includes cardiac myxomas as one of its cardinal features.

背景：长期以来，蛋白激酶A信号途径被认为在心功能中起着重要作用，蛋白激酶A调节亚单位基因PRKAR1A的突变导致蛋白激酶A系统失调，引起遗传性肿瘤综合征－Carney合成，其中心脏粘液瘤作为其主要特征。

Background— Protein kinase A signaling has long been known to play an important role in cardiac function. Dysregulation of the protein kinase A system, caused by mutation of the protein kinase A regulatory subunit gene PRKAR1A, causes the inherited tumor syndrome Carney complex, which includes cardiac myxomas as one of its cardinal features. Mouse models of this genetic defect have been unsatisfactory because homozygote null animals die early in development and heterozygotes do not exhibit a cardiac phenotype.

背景——蛋白激酶A信号传导在心功能中发挥了重要作用早已为人们所知蛋白激酶A调节亚基因PRKAR1A的变化引起蛋白激酶A系统失调，导致了遗传性卡尼复合肿瘤综合症，其中包括的心脏粘液瘤作为它的一个主要特征此种遗传缺陷的小鼠模式仍然不能令人满意，因其纯合体空白动物在发展中早期死亡，而杂合体不能显示心脏表型

2Fc2+, which can be rationalized by the equivalent environment of the ferrocene subunit in each case.

2Fe~(2+)的过程，说明两个目标化合物中的两个二茂铁基所处的电化学环境基本相同。

The subunit translocates to the nucleus, where it activates transcription.

胞外结构的二聚体化引起跨膜结构域在膜内进行横向扩散，并启动与胞质结构域的接触。

Nuclear trans location of the subunit p65 and p50, was observed by indirect immunofluorescence assay when fibroblast-like synoviocytes were respectively stimulated by 10^(-4) Mol/L E for 1, 2, 4, 8 and 24 boors. Results: E induced the nuclear translocation of p65 at 4, 8 and 24 hours.

结果：荧光显微镜观察染色的滑膜细胞，发现E2处理4、8、24h的滑膜细胞p65发生了核转位，其转位率分别为68%、95%和52%，在8h时达到峰值。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。