查询词典 study group

Results: HE stain showed the obvious thicken of the artery, RV/ and mPAP elevated obviously (P.05) in model group in comparison with the control group, they were also elevated obviously (P.05) in the model group compared with the treatment group. The tryptase + mast cell and the chymase + mast cell obviously increased in the model group compared with the control group, also increased in the model group compared with the treatment group.

平均肺动脉压力模型组动物较对照组明显升高(P.05)，模型组较治疗组也明显升高(P.05)；右心室／模型组较对照组明显增加(P.05)，模型组较治疗组也明显增加(P.05)，肥大细胞总数（类胰蛋白酶阳性的肥大细胞）及类糜蛋白酶阳性的肥大细胞模型组较对照组明显增多(P.05)，肥大细胞在治疗组较模型组明显减少(P.05)。

The healing rate of wound of control group is obviously higher than the model group.(3) Observe under the microscope: When 3 days after we practice the surgery, HE and observe control group wound have a large number of inflammation nature cell soak, model group wound surface inflammatory cell relatively sparse in Balb/c mouse;The control group can seeⅠtype collagenic and green type collagenic day, model group type be obviously less than the control group; When 7 days after we practice the surgery,HE and bitter acid-staining observe control group peripheral inflammation cell soak, have appeared granulation organize.

3镜下观察：创伤后第3d，HE染色观察对照组伤口有大量炎性细胞浸润，模型组小鼠创面炎症细胞较稀疏，天狼猩红染色对照组可见少量呈黄色的Ⅰ型胶原和呈绿色的Ⅲ型胶原，模型组Ⅲ型胶原要明显少于对照组；创伤后第7d， HE染色观察对照组周边炎性细胞浸润，有肉芽组织出现。

To investigate the change of these indexes after treatment between traditional Chinese drug treatment group and control group, and the difference between control group and TCDT group.Methods:1. 40 cases with CHD were divided into TCDT group and control group randomly. The control group was treated by regular western medicine of Isosorbide Dinitrate and ASA while the TCDT group was treated by Guanxinshengmaiyin.

1、选择40例冠心病患者，随机分为两组，即中药治疗组与西药对照组，中药组用&冠心生脉饮&治疗，西药组常规给予消心痛和阿司匹林口服；2、检测两组病人治疗前后血清VEGF和NO的含量；3、选择16例健康体检者作为健康对照组。

In our experiment 40 Wistar male rats were divided into 5 groups at random, asfollows: Contrast group (8, abbreviated as normal group), Adjuvant Arthritis modelgroup (8, abbreviate as model group), Dazhui electropuncture group (8, abbreviated asDazhui group), Mingmen electropuncture group(8, abbreviated as Mingmengroup),Feixue electropuncture group (8, abbreviated as Feixue group).

本实验共用 Wistar 雄性大鼠 40 只，随机分为 5 组，分组如下：正常对照组(8 只，简称正常组)、佐剂性关节炎模型组(8 只，简称模型组)、电针大椎组(8 只，简称大椎组)、电针命门组(8 只，简称命门组)、电针非穴组(8 只，简称非穴组)。

Objective To examine the value of Edaravone in the treatment of vascular dementia through ethologic and pathologic changes induced by chronic cerebral ischemiaMethods Wistar rats were randomly divided into sham operated group,model group,edaravone group and duxil groupThe chronic cerebral hypoperfusion models were made by permanent bilateral occlusion of both common carotid arteriesThe memory ability and ultramicrostructure of brain tissue were observedResults The escape latencies in model group were more significantly prolonged than that in the other groups (P＜005)But there was no significant difference between edaravone and duxil groupAnd pathologic changes observed by electron microscope in nerve cell of edaravone group and duxil group and model group were fairly light than that in sham operated groupEdaravone group had increased mitochondrium and frontal membranous synaptic vesicle and more productive metabolic activityConclusions Edaravone can lessen injures of hippocampal neurons loss to improve the spatial learning and memory abilitiesAnd it is proved further that edaravone can treat VD

目的 观察依达拉奉对慢性缺血致血管性痴呆大鼠行为学及形态学的影响，探讨依达拉奉在治疗VD中的应用价值。方法实验分假手术组、模型组及都可喜对照组及依达拉奉治疗组，应用双侧颈总动脉结扎方法制备慢性脑缺血大鼠痴呆模型，观察各组大鼠的记忆功能及脑组织超微结构改变。结果缺血组水迷宫表现同对照组、都可喜组及依达拉奉组相比有显著差异(P＜005)，依达拉奉组与都可喜组相比未见显著差异。同时电镜观察结果也发现依达拉奉组及都可喜组与模型组及假手术组相比神经细胞病理改变较轻，且依达拉奉组线粒体及前膜突触小泡增多，代谢活动较都可喜组旺盛。结论依达拉奉可能通过减轻海马神经元的损伤来改善慢性脑缺血大鼠空间学习记忆障碍，证明依达拉奉对慢性缺血致VD大鼠具有治疗作用。

Methods］ninety femal sd rats,weight 230～280 gram,were rangdonmized into three groups: qiangguyin group,miacalcic group and control group.all rats were variectomized.after 10 weeks,the osteoporosis model succeed,the treatment group,contrast group were given qiangguyin,miacalcic respectively.the treatment group used qiangguyin everyday,1ml/g.the contrast group used miacalcic 0.72u/kg,everyday hyodermic.and 3,4.5,6 months after the treatment,kill 10 rats to explore.the neck of femur were cut and stined with masson golder trichrome for bone histomorphomemetric contrast group analysis.

方法］健康雌性sd大鼠90只，体重230～280g。随机分为3组，分别为治疗组、对照组和空白组，每组30只。分别采取切除双侧卵巢方法进行骨质疏松造模，10周造模成功后，开始给药，治疗组用自拟强骨饮灌胃，每日一次，每次1ml/g，对照组用密盖息，皮下注射，每日1次，每次0.72u/kg。空白组，不做处理。在给药后3、4.5、6个月每组各取10只大鼠进行检测，先测量体重，空气栓塞处死，分离股骨颈，获得股骨颈样本，经切片等处理后，显微镜下作骨形态计量学检测。

Results: In acute experiment, there was no significant difference of body weight or organ indexes between the normal control and arsenic poisoning groups ; while the CH50 level, lymphocyte accruing rate, phagocytosis rate and macrophagocyte index in arsenic poisoning group were significantly lower (P.05 or P.01) than those in normal group. There was no significant difference of body weight or organ index between arsenic poisoning group and EXD (10 g) treated arsenic poisoning group, while CH50, lymphocyte accruing rate, phagocytosis rate, and mavrophocyte index were significantly higher in EXD (10 g) treated arsenic poisoning group than those in arsenic poisoning group (P.05 or P.01). In EXD (5 g) treated arsenic poisoning group, results of all the tested indexes were the same as those of arsenic poisoning group.

结果：与正常对照组相比，砷中毒组小鼠体重、脏器指数无明显改变；CH50、淋巴细胞增殖、巨噬细胞吞噬率及吞噬指数明显降低，差异有显著性（P.05或P.01；与砷中毒组相比，竹荪治疗低剂量组小鼠体重、脏器指数、CH50、淋巴细胞增殖、巨噬细胞吞噬率及吞噬指数无明显改变化；竹荪治疗高剂量组除小鼠体重、脏器指数无明显改变外，CH50、淋巴细胞增殖、巨噬细胞吞噬率及吞噬指数明显升高，差异有显著性（P.05或P.01）。

The treatment regimens of Trial patients who were randomized into control group (Trial-control group) were quite the same with the standard treatments of Nontrial group. There was no significant difference in median survival between Trial-control group and Nontrial group. The median survival of Trial group patients who were randomized into trial group was significantly longer than that of Nontrial group patients.

在Phase III的临床试验中，病患会被随机分派到试验组(Trial-trial)或是控制组(Trial-control)，控制组的治疗方式与非临床试验组类似，两组存活时间在统计上没有差异；使用新药或新疗程的Trial-trial组的存活时间比非试验组显著较长。2000-2003年间存活时间有较长的趋势，而医疗成本部分则是2004年比起2000年显著的升高。

Electrophysciological indexes: in the experimental group ,the tardy rate of induced motorius action potential ,recovery rates of the amplitude of compound muscle action potential and twitch tension and tetanic tension of triceps surae muscle ,were significantly higher than in the control group.(4) Compared with the control group ,the triceps surae muscle wet weight was significantly greater in the experimental group.(5) histomorphological indexes: the count of myelinated nerve fibers ,diameter of regenerated axon,thickness of nerve myelin sheath and area of capillary were much more in the experimental group than in the control group.(6) observation of ultrastructure with the transmission election microscope: more abundant organella and maturer myelin sheath lay in the injured peripheral nerve of the experimental group, while not lay in the control group.

分别在术后第2、4、6周时进行各项指标的检测，结果发现：（1）实验组SD大鼠在整个实验过程中，其精神、毛发、体重，运动灵活性等一般情况优于对照组；（2）实验组的坐骨神经功能指数恢复率显著优于对照组，P＜0.01；（3）神经电生理指标：实验组运动神经诱发电位潜伏期的延迟率显著优于对照组，P＜0.05；复合肌肉动作电位振幅的恢复率显著高于对照组，两组比较第2周时P＜0.01，第4、6周时，P＜0.05；小腿三头肌单收缩力和强直收缩力的恢复率实验组显著高于对照组，P＜0.01；（4）小腿三头肌湿重的恢复率，第2、4周时实验组高于对照组，差异非常显著，P＜0.01；第6周时，差异仍显著，P＜0.05；（5）组织形态学指标：有髓神经轴突计数、再生轴突直径的恢复率，毛细血管面积，实验组均显著大于对照组，P＜0.01、P＜0.05、P＜0.001；实验组髓鞘厚度的恢复率也显著高于对照组，第2周时P＜0.01，第4、6周时，P＜0.05；（6）透射电镜超微结构观察：实验组再生轴突中细胞器丰富，髓鞘结构成熟，神经再生情况优于对照组。

Methods］ 36 twomonthold rabbits with white feather and black eyes,half male and half female,were randomly divided into 6 groups:normal group,model group,xiangsha liujun pill group, lizhu changle group, lowdose leweiyintreated group,highdose leweiyintreated group.except the normal group, others were made into ibs model of spleen deficiency which was induced by damp heat stress and gavage with fanxieye as well as method of either hunger or gorge.the drug groups were administered preventively from the mediumterm of the model reproduction to the end of modeling.the rabbits of each group were observed.no,nos,mda,sod in serum and sp in colonic membrane were detected.

方法］两月龄白毛黑眼兔36只，雌雄各半，随机分为6组：正常组、模型组、香砂6君丸组、丽珠肠乐组、乐胃饮低剂量组、乐胃饮高剂量组。除正常组外，其余whbe兔采用湿热环境应激联合番泻叶灌胃加饥饱失常复合方法致脾虚泄泻型ibs模型。从模型复制的中期开始各用药组预防给药，至造模结束。观察各组1般情况，测定血清no、nos、mda、sod含量及结肠黏膜组织中sp含量。

But this is impossible, as long as it is engaging in a market economy, there are risks in any operation.

但是，这是不可能的，只要是搞市场经济，是有风险的任何行动。

We're on the same wavelength.

我们是同道中人。