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12H treatment group compared with 12h model group: the density of light of HSP70 of the Gardenoside group is not different and which of it of TGFβ〓 increased with the model group; the density of light of the HSP70 of the Baicalin group in not different and which of the TGF-β〓 is no different with the model group, the density of HSP70 of the Concha Margatitifera Usta is not different and which of TGFβ〓 increased than the model group; the density of the HSP70 of the Cholic Acid group is no different and the density of TGF-β〓 increased than the model; the density of the light of HSP70 of the Hefang Group increased and which of TGF-β〓 increased than the model group.

合方治疗脑缺血12小时组缺血脑组织TGF-β1光密度值较缺血12小时模型组明显升高（p＜0.01）。大鼠局灶性脑缺血24小时模型组缺血脑组织TGF-β1光密度值含量较正常组明显增高（p＜0.01）。珍珠母治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。胆酸治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。栀子苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组无显著性差异（p＞0.05）。黄芩苷治疗脑缺血24小时组缺血脑组织TGF-β1光密度值较缺血24小时模型组明显升高（p＜0.01）。

The time of cornea diabrosis, metastases into cervical lymph nodes and lung, survival time and ratio of CD4+ and CD8+ T cells in peripheral blood were observed.RESULTS: The time of cornea diabrosis in GTVC group or GTV group was later than that in PBS group, but no statistical difference between GTVC group and GTV group. Metastatic incidence in cervical lymph nodes of GTVC group mice or GTV group mice was lower than that in PBS group 18 days after the model had been established. There was no statistical difference in metastatic incidence between GTV group and GTVC group.

将荷瘤模型鼠分为磷酸盐缓冲液（phosphatebuffered saline，PBS）治疗组，IL12基因修饰肿瘤细胞疫苗（gene modified tumor cell vaccine， GTV）治疗组及GTV 联合CpGODN治疗组；鼠荷瘤模型建立后的第3d及第10d分别在各组鼠结膜下注射30μL PBS，30μL（3.0×106个细胞）灭活的GTV及含20μg CpGODN灭活的GTV，进行角膜溃破时间、淋巴结和肺部转移瘤发生率、生存时间及外周血CD4＋和CD8＋T细胞比例的观测。

Results On the basis of the percentage of GPI-anchored PLAP conversion,the sera GPI-PLD activities of total 172 patients,included 26 severe acute viral hepatitis as group A,29 liver cirrhosis as group B,32 chronic viral hepatitis as group C,55 mild acute viral hepatitis as group D,30 primary hepatocellular carcinoma as group E,were measured.As compared with 182 healthy presons as control group,the sera GPI-PLD activities of group A and B were significantly reduced;By contraries,the activities of group D and E were significantly raised.The sera GPI-PLD activities of group C compared with healthy control group were not significantly altered.However,when paired Q-test,the changes of serum GPI-PLD activity between all paired groups among this five groups were remarkable.

结果检测172例肝脏疾病患者(包括A组：26例急性重症病毒性肝炎；B组：29例肝硬化；C组：32例慢性病毒性肝炎；D组：55例急性非重症病毒性肝炎；E组：30例原发性肝癌)血清中GPI-PLD的活性，发现A、B两组患者的该酶活性较正常人(182名)显著性降低，而D、E两组则较正常人显著性增高，C组患者的酶活性与正常人无显著性差异，但A、B、C、D、E两组之间的酶活性水平改变却均有显著性。

Genetic diversity of the demes was expressed as follow: yellow peach group＞honey peach group＞flat peach group＞red leaf peach group＞crisp peach group＞bitao group and juicy peach group＞nectarine group＞shouxingtao group＞weeping peach group.

各类群遗传多样度为黄肉桃＞蜜桃＞蟠桃＞红叶桃＞硬肉桃＞碧桃、水蜜桃＞油桃＞寿星桃＞垂枝桃。

In school children, tibial bone strength of obese group and overweight group, normal weight group was in the increasing order; the descending risk of bone strength in obese and overweight group was 9.73 and 2.54 times of normal group respectively. Radialis bone strength of obese group was worse than overweight group and normal weight group, while the bone strength of overweight group and normal weight group was not different.

学龄儿童正常、超重、肥胖组的胫骨骨强度依次下降，肥胖、超重组胫骨骨强度下降风险是正常组的9.73和 2.54倍；学龄肥胖组桡骨骨强度低于正常组，超重组与正常组无差别。

Result: The nm-HAP-sol present the dispersed and uniformity needle. A431cells inhibition ratio was maximizing at the 5 day in the 400mg/L nm-HAP-sol group, the 200mg/L nm-HAP-sol group and the 100mg/L nm-HAP-sol group, inhibition ratio of the 400mg/L nm-HAP-sol group, the positive control group and the 50mg/L nm-HAP-sol group was respectively (77.89±6.29)%,(77.46±8.26)% and (1.23±0.15)%. It was detected that the number of A431 cells were persistently increasing in the 50mg/L nm-HAP-sol groups while decreasing in the 400mg/L nm-HAP-sol group, the positive control group and abidingly increasing for 4 and 3 days then decreasing in the 100mg/L and 200mg/L nm-HAP-sol group by inverted microscope.

结果：超声乳化法配制的HAP纳米溶胶呈均匀分散的针状；400mg/L、200mg/L和100mg/L溶胶组对A431细胞抑制率在第5天达到各组最大值，此时400mg/L溶胶组与阳性对照组的抑制率分别为（77.89±6.29）%和（77.46±8.26）%， 50mg/L溶胶组抑制率为（1.23±0.15）%；倒置显微镜下见50mg/L溶胶组细胞数目持续增加，聚集成簇，细胞呈现多角形或梭形，胞浆透亮，而100mg/L 和200mg/L溶胶组的细胞数目分别增加至第4天和第3天开始下降，400mg/L溶胶组和阳性对照组细胞数目持续减少，细胞体积缩小，漂浮细胞增多。

There are the normal group,the AS group,the pure swimming group,the swimming plus AS group,the simvastatin group(simvastatin plus AS of overstrain),the large dose Tongxinluo group,the small dose Tongxinluo group,the bulbus allii macrostemonis four drugs group and the ginseng group.

目的对络气虚滞型动脉粥样硬化模型中9个编号组即正常组、动脉粥样硬化组、单纯游泳组、游泳加动脉粥样硬化组、辛伐他汀组(即辛伐他汀+过劳加动脉粥样硬化，下同)、通心络大剂量组、通心络小剂量组、薤白四味组、人参组进行综合评价。

Result: The ROM of group A had no significant difference with that of the control group. The ROM of group B had no significant difference with that of the control group except rotation, each ROM of group C and group D was significant difference with the control group, and the ROM of group D was the biggest. The ROM of group E was the smallest and had statistically significant difference in each motion compared with the control.

结果：A组ROM值与对照组无显著性差异；B组除旋转外与对照组无显著性差异，旋转稳定性也达到了对照组的61.86%；C组与对照组均有显著性差异；D组ROM值最大，与对照组有显著性差异；E组除后伸外ROM值均明显小于对照组，差异有显著性(P.05)。

12H treatment group compared with 12h model group: the level of MCP-1 of the Gardenoside group decreased and MIP-1 is no different with the model group; the level of the MCP-1, MIP-1 of the Baicalin group are no different with the model group; the level of MCP-1, MIP-1 of the Concha Margatitifera Usta decreased than the model; the level of MCP-1 of the Cholic Acid group is no different and the level of MIP-1 decreased than the model; the level of MCP-1 of the Hefang Group decreased and the level of MIP-1 of it decreased than the model group.

合方治疗脑缺血24小时组缺血脑组织nNos光密度值较缺血24小时模型组明显降低（p＜0.01）。细胞的死亡分为坏死和凋亡两种方式，凋亡是细胞为避免对机体造成更大损伤而采取的一种程序性的死亡方式。珍珠母、胆酸、栀子苷、黄芩苷、合方治疗组较脑缺血24小时模型组可明显降低P53阳性表达，考虑药物抑制细胞凋亡可以起到对缺血脑组织的保护作用。

24H treatment group compared with 24h model group: the level of MCP-1 of the Gardenoside group decreased, but the level of MIP-1 of it is no different with the model group; the level of the MCP-1 of the Baicalin group increase and the level of MIP-1 of it all decreased; the level of MCP-1 and MIP-1 of the Concha Margatitifera Usta all decreased; the level of MCP-1 of the Cholic Acid group decreased and the level of MIP-1 is no different with the model group; the level of MCP-1 and MIP-lof the Hefang Group decreased than the model group.

在本实验中，脑缺血能够诱导P53蛋白表达，P53蛋白可能参与调控缺血后神经细胞延迟性死亡的病理生理过程，药物通过抑制细胞凋亡，从而发挥治疗作用。五、大鼠局灶性脑缺血12小时模型组缺血脑组织HSP70光密度值含量较正常组明显增高（p＜0.05）。珍珠母治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。胆酸治疗脑缺血12小时组缺血脑组织HSP70光密度值较缺血12小时模型组无显著性差异（p＞0.05）。

But this is impossible, as long as it is engaging in a market economy, there are risks in any operation.

但是，这是不可能的，只要是搞市场经济，是有风险的任何行动。

We're on the same wavelength.

我们是同道中人。