查询词典 striatum

Relative and absolute blood flow in the corpus striatum were determined using the laser Doppler flowmetry and the autoradiography diffusible tracer technique, respectively.

大脑纹状体的相对及绝对血流量是各利用雷射都卜勒仪及自动放射显影可扩散追踪剂方法测量得到。

By the computer 3D technique, a 3-D stereotactic morphology of the corpus striatum, marginal division and globus pallidus was thus reconstructed.

建立了大鼠脑纹状体、边缘区和苍白球的三维立体结构。

In addition to the pons positive neurons were also found in cerebellum, most nucleus of thalamus and hypothalamus, and corpus striatum of chicken.

鸡脑阳性神经元除广泛分布于脑干外，小脑、丘脑的多数核团、下丘脑的背侧核、腹侧核和后核以及大脑纹状体等多数脑区也有一定分布。

RESULTS Compared with normal control group, HVA, 5-HT and GSH contents, and SOD activity in corpus striatum of model mice decreased, and MDA content increased significantly. Otherwise, numbers of Bcl-2 and caspase 3 immune reaction positive cells in substantia nigra increased significantly.

结果 与正常对照组比较，模型组小鼠脑内纹状体HVA，5-HT和GSH含量显著下降，SOD活性下降，MDA含量明显升高；黑质致密部Bcl-2和caspase 3免疫阳性细胞数增加。

Objective To investigate the effects of subthalamic nucleus -deep brain stimulation on expression of dopamine and adenosine 3'5'-monophosphate-regulated phosphor-protein (DARPP-32) and its phosphorylated proteins in corpus striatum of rat models with levodopa-induced dyskinesia.

目的 探讨丘脑底核高频电刺激对帕金森病异动症大鼠纹状体区多巴胺和cAMP调节的磷蛋白(DARPP-32)及其磷酸化蛋白表达的影响。

The protection of VOA on brain may relate to the release of monoami neurotransmitters in corpus striatum increased, oxidative stress alleviated and neuronal apoptosis in substantia nigra reduced.

VOA的脑保护作用可能与其增加脑内纹状体单胺类神经递质释放、减轻氧化应激反应以及减少黑质致密部神经元凋亡有关。

Four weeks later, 69 Parkinson disease model s were randomly divided into 3 groups:①Control group(n=24):4.5 μL normal saline was injected into head of caudate nucleus of right-side corpus striatum.

取SD雄性大鼠，右侧中脑黑质致密区注入6-羟基多巴(2g/L)3μL制备帕金森病模型，4周后将69只成功帕金森病模型随机分为3组：①对照组24只：右侧纹状体尾状核头部注入生理盐水4.5μL。

The activity of NF-κB is downregulated and the mRNA expression is up-regulated. in the prefrontal cortex, hippocampus and corpus striatum in the hypoglutamatergic schizophrenia model rats which indicate NF-κB might be involved in the pathophysiology of schizophrenia.

谷氨酸能低下精神分裂症模型大鼠前额皮质、海马、纹状体NF-κB的活性受到了抑制，而mRNA表达上调，提示NF-κB可能参与了精神分裂症的病理过程。

But the core of the cerebrum, the corpus striatum, has en1arged into the principal integrative center

小脑发达且体积大，这与鸟类飞翔动作及其协调密切相关。

Objective To investigate the gene expression of Nuclear factor-κB in the prefrontal cortex, hippocampus and corpus striatum in MK-801 induced hypoglutamatergic schizophrenia model rats to explore the molecular mechanism of schizophrenia.

目的 了解地卓西平马来酸盐[Dizocilpine maleate， MK-801]所致谷氨酸能低下精神分裂症大鼠前额皮质、海马、纹状体核因子－κB(Nuclear factor-κB， NF-κB)基因的表达，探讨其参与精神分裂症的病理机制。

Plunder melds and run with this jewel!

掠夺melds和运行与此宝石！

My dream is to be a crazy growing tree and extend at the edge between the city and the forest.

此刻，也许正是在通往天国的路上，我体验着这白色的晕旋。