查询词典 spider cancer

Similarly yellow ribbons for bladder cancer, purple ribbon for pancreatic cancer, testicular cancer and thyroid cancer, brown or royal blue ribbons for colon cancer, golden, pink or light blue for childhood cancer, green ribbons for kidney cancer, pearl white ribbons for lung cancer, plain white ribbons for bone cancer, orange ribbons for blood cancer, burgundy ribbons for multiple cancer, gray ribbons for brain cancer, blue ribbons for prostate cancer and black ribbons for skin cancer.

同样的黄色丝带为膀胱癌，紫色的丝带胰腺癌，睾丸癌和甲状腺癌，棕色或皇家蓝丝带结肠癌，金，粉红色或淡蓝色的儿童癌症，绿丝带为肾癌，珍珠白丝带肺癌，纯白色的丝带为骨癌，橙色丝带为血癌，勃艮第带多个癌症，灰色丝带为脑癌，蓝丝带的前列腺癌和黑丝带的皮肤癌。

The stable clones are further identified by RT-PCR and Western blot; 6 MTT assay is used to investigate the effect of ZNRD1 on the cell growth of cells (AGS, SGC7901, MKN28, NIH3T3, GES-1); 7 Soft agar assay is used to investigate the effect of ZNRD1 on the clonality of cells (AGS, MKN28); 8 Nude mice assay is used to investigate the effect of ZNRD1 on the cell growth of gastric cancer cells (AGS, MKN28); 9 Flow cytometry is used to investigate the effect of ZNRD1 on the cell cycle distribution of cells (AGS, MKN28, NIH3T3, GES-1); 10 Flow cytometry is used to investigate the effect of ZNRD1 on the cell apoptosis of cells (AGS, MKN28, NIH3T3); 11 MTT assay is used to investigate the effect of ZNRD1 on the drug sensitivity of cancer cells (SGC7901, SGC7901/VCR, HL-60, HL-60/VCR) in vitro; 12 SRCA is used to investigate the effect of ZNRD1 on the drug sensitivity of gastric cancer cells (SGC7901, SGC7901/VCR) in vivo; 13 Flow cytometry is used to investigate the effect of ZNRD1 on adriamycin accumulation of cancer cells (SGC7901, SGC7901/VCR, HL-60, HL-60/VCR); 14 Transmission electron microscope is used to investigate the effect of ZNRD1 on the sensitivity of SGC7901 cells towards drug-induced apoptosis; 15 Flow cytometry and DNA ladder assay are used to investigate the effect of ZNRD1 on the sensitivity of cells (SGC7901, SGC7901/VCR, HL-60/VCR) towards drug-induced apoptosis; 16 Microarray is used to investigate the profiling of ZNRD1-responsive genes in gastric cancer cells (AGS, MKN28, SGC7901, SGC7901/VCR); 17 RT-PCR and Western blot are used to identify the results of microarray; 18 Reporter gene assay is used to investigate the effect of ZNRD1 on the transcriptional activity of cyclin D1; 19 Reporter gene assay is used to investigate the effect of ZNRD1 on the transcriptional activity of MDR1; 20 Kinase assay is used to investigate the effect of ZNRD1 on the activity of cyclin E-CDK2 kinase; 21 The antisensenucleic acids of p21 is used to inhibit the expression of p21, and flow cytometry is used to investigate the effect of p21 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 22 The antisensenucleic acids of p27 is used to inhibit the expression of p27, and flow cytometry is used to investigate the effect of p27 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 23 Liposome is used to up-regulate the expression of Skp2, and flow cytometry is used to investigate the effect of Skp2 on ZNRD1-induced cell cycle arrest in gastric cancer cells; 24 Western blot is used to investigate the effect of ZNRD1 on the stability of Skp2 and p27 in gastric cancer cells; 25 MVD assay is used to investigate the effect of ZNRD1 on the angiopoietic activity of gastric cancer cells; 26 ELISA is used to investigate the effect of ZNRD1 on the expression of VEGF165 in gastric cancer cells; 27 The roles of DARPP-32 in MDR of gastric cancer cells are investigated using gene transfection, MTT assay, SRCA, flow cytometry and DNA ladder assay.

应用杂交瘤技术制备ZNRD1的首个单克隆抗体；2）利用RT-PCR、Western blot和免疫组化检测ZNRD1在胃癌组织、胃炎组织、正常胃上皮组织、胃癌细胞和正常胃组织上皮细胞中的表达；3）构建ZNRD1的小干扰RNA载体，并测序鉴定；4）利用脂质体将ZNRD1的真核表达载体及其空载体转染胃癌细胞（AGS、SGC7901、MKN28）和小鼠成纤维细胞（NIH3T3），G418筛选后进行鉴定；5）利用脂质体将ZNRD1的小干扰RNA载体及其空载体转染药敏胃癌细胞（SGC7901）、正常胃组织上皮细胞（GES-1）、对长春新碱耐药的胃癌细胞（SGC7901/VCR）、药敏白血病细胞（HL-60）、对长春新碱耐药的白血病细胞（HL-60/VCR），G418筛选后进行鉴定；6）利用MTT实验检测ZNRD1高/低表达对细胞（AGS、SGC7901、MKN28、NIH3T3、GES-1）生长的影响；7）通过软琼脂克隆形成实验检测上调ZNRD1对AGS、MKN28细胞克隆形成能力的影响；8）通过裸鼠成瘤实验检测上调ZNRD1对AGS、MKN28细胞体内成瘤性的影响；9）通过流式细胞仪分析ZNRD1高/低表达对细胞（AGS、MKN28、NIH3T3、GES-1）的细胞周期的影响；10）通过流式细胞仪分析上调ZNRD1对细胞（AGS、MKN28、NIH3T3）的凋亡的影响；11）通过MTT实验检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60、HL-60/VCR）体外药物敏感性的影响；12）通过肾包膜下移植法检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR）体内药物敏感性的影响；13）通过流式细胞仪分析ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60、HL-60/VCR）内阿霉素蓄积和泵出的影响；14）通过透射电镜检测上调ZNRD1对SGC7901细胞凋亡敏感性的影响；15）通过流式细胞仪和DNA梯度试验检测ZNRD1高/低表达对细胞（SGC7901、SGC7901/VCR、HL-60）凋亡敏感性的影响；16）通过基因芯片检测ZNRD1高/低表达对胃癌细胞内基因表达谱的影响；17）利用RT-PCR、Western blot对基因芯片的结果进行鉴定；18）利用报告基因实验检测ZNRD1对cyclin D1的启动子活性的调节作用；19）利用报告基因实验检测ZNRD1高/低表达对MDR1的启动子活性的调节作用；20）利用激酶试验检测ZNRD1对cyclin E-CDK2 激酶活力的影响；21）利用反义核酸技术抑制p21的表达；通过流式细胞仪检测抑制p21对ZNRD1介导的细胞周期阻滞的影响；22）利用反义核酸技术抑制p27的表达；通过流式细胞仪检测抑制p27对ZNRD1介导的细胞周期阻滞的影响；23）利用脂质体转染法上调Skp2的表达；通过流式细胞仪检测上调Skp2对ZNRD1介导的细胞周期阻滞的影响；24）利用Western blot检测ZNRD1对p27和Skp2的蛋白稳定性的影响；25）利用微血管密度实验检测ZNRD1对AGS、MKN28细胞裸鼠移植瘤微血管形成的影响；26）利用ELISA检测ZNRD1对AGS、MKN28细胞培养上清和移植瘤匀浆中VEGF165含量的影响；27）利用脂质体转染法、MTT实验、肾包膜下移植法、流式细胞仪和DNA梯度试验检测新耐药相关分子DARPP-32对细胞（SGC7901、SGC7901/VCR、对阿霉素耐药的胃癌细胞SGC7901/ADR）多药耐药表型的影响；利用脂质体转染法和MTT实验检测下调ZNRD1对DARPP-32介导的胃癌多药耐药的调控作用。

Characters in order of appearance: Deadpool, Arcade, Basilisk, Ares + Hercules, Nova, Annihilus, Ant-Man, Ms. Marvel, Baron Zemo, Black Box, Ghost Rider, Black Goliath, Black Bolt, Beta Ray Bill + skrull, Norman Osborn/ Green Goblin, Thunderbolts (Venom + Bullseye + Songbird), Nightcrawler, Juggernaut, Black Tom Cassidy, Agency X (a guy I don't know, Outlaw, Agent X, Taskmaster, a girl I don't know), Air Walker, Gambit + Bishop, High Evolutionary, Baron Von Strucker, Mighty Avengers (Jocasta + Scarlet Witch + Wasp/ H. Pym), New Avengers (Spider-Man + Ronin + Ms. Marvel + Luke Cage + Captain America/ Bucky + Spider-Woman + Wolverine), Eel, Dr. Strange, Ajak, Generation X (Jubilee + Husk + Synch + Skin + M + Chamber), Angel, Hulk, Fing Fan Foom + Hulk, Belasco + Illyana Rasputin, Shadowcat, Omega Red, Vulture, Young Avengers (Vision + Wiccan + Patriot + Hulkling + Hawkeye + Speed), Ms. Marvel + Jean Grey + Mr. Fantastic + Wolverine + Captain America + Iron-Man + Invisible Woman + Thing + Spider-Man + Charles Xavier, Abomination, Thanos + Galactus, Blastaar, Aegis, Thor + Captain America/ Bucky + Wolverine, Atlas, Avalanche, Avengers (Thor + Iron-Man + Captain America + Scarlet Witch + Ant-Man + Wasp), Baron Zemo + Avengers (Captain America + Thor + Giant-Man), Prodigy + Batwing + Sunstreak + Gorilla Girl + Boulder + Annex, Banshee, Arana, Cyclops + Professor Xavier, Green Goblin + Spider-Man, Kang The Conqueror, Nick Fury, Northstar + Cyclops, Northstar + Aurora, Punisher, Iron Fist, Thor, Black Goliath, X-Men (Banshee + Angel + Iceman + Cannonball + Warpath + Cyclops + Colossus + Beast + Bishop + Longshot + Cable + Wolverine + Nightcrawler + Havok + Gambit), Storm, Fantastic 4 (Invisible Woman + Human Torch + Thing + Mr. Fantastic)+ Black Panther + Storm, Gambit + Wolverine, Captain America + Logan, a kree + Ms. Marvel + Captain Marvel + Captain Marvel + Avengers (Giant-Man + Hulk + Captain America + Thor + Wasp), X-Statix (Dead Girl + Mr. Sensitive + U-Go Girl + Saint Anna + Venus Dee Milo), Black Panther, Swordsman, Iceman + Iron-Man + Human Torch + Spider-Man + Colossus + Nightcrawler + Quicksilver + Wolverine + Captain America + Hulk + Mr. Fantastic + Thor + Storm + Thing, Shadowcat + Rogue, Whiplash, X-Corps (Avalanche + Blob + Banshee + Husk + Jubilee), Adam X, Crimson Dynamo, Mr. Sinister, Agents Of Atlas (the Uranian + M-11 + Jimmy Woo + Namora + Venus + Gorilla Man), Apocalypse, Taskmaster, Agent 0 + Wolverine, Battlestar, Baron Mordo, Batwing + Spider-Man, Union Jack, Ancient One, Arclight, Anole, Iron-Man.

享受！ 在外观订购特点：Deadpool，商场，虺蛇，战神+大力神新星，Annihilus，蚁民，女士惊奇，男爵泽莫，黑盒，幽灵骑士，黑巨人，黑螺栓Skrull +β射线条例草案，诺曼奥斯本/绿精灵，桑德博茨，Nightcrawler，剑圣，黑色汤姆卡西迪，代理第十章（一家伙，我不知道，奥特洛，X制剂，工头，一个女孩，我不知道），空气沃克，开局+主教，高级进化，男爵冯施特吕克尔，全能的复仇者（乔卡斯塔+大红女巫+黄蜂/ H的皮姆），新复仇者（蜘蛛侠浪人+++女士惊奇笼+美国队长卢克/布基+蜘蛛女+狼獾），鳗鱼，博士奇怪，阿贾克， X一代（银禧+壳+同步+皮肤+ M +的商会），天使，绿巨人，Fing头范宽+绿巨人，贝拉斯科+ Illyana拉斯普京，Shadowcat，欧米茄红色，雕，杨复仇者（视觉+ Wiccan +爱国者+ Hulkling +霍克艾尔+速度），让灰色女士惊奇+++金刚狼+奇先生美国队长+铁人+隐形女+物+蜘蛛侠+查尔斯泽维尔，憎恶，萨诺斯+ Galactus， Blastaar，宙斯盾，索尔+美国上尉/布基+金刚狼，阿特拉斯，雪崩，复仇者（索尔+铁人+美国队长+女巫+大红蚂蚁满+黄蜂），男爵泽莫+复仇者（美国上尉雷神++巨男子），神童+蝙蝠翼+ Sunstreak +大猩猩女+博尔德+附件，女妖，阿拉纳，独眼巨人+泽维尔教授，绿精灵+蜘蛛侠，康的征服者，尼克骚动，诺斯斯塔+独眼巨人，诺斯斯塔+极光，惩罚者，铁拳，雷神，黑巨人，X战警（女妖+天使+冰人+独眼巨人++沃帕斯巨像+野兽+主教+朗肖特+电缆++金刚狼+ Nightcrawler Havok的开局坎南伯尔++），风暴，神奇四（看不见的女人+人力火炬+物+神奇先生）+黑豹+风暴，开局+金刚狼，美国队长+洛根，1 kree女士惊奇++船长船长惊奇Marvel公司++复仇者（巨人+绿巨人+美国队长+雷神+黄蜂）和X - Statix（敏先生死亡女孩++ U型转到女孩安娜++圣迪米罗维纳斯），黑豹，武侠小说，冰人+铁人+人力火炬+蜘蛛侠+巨像+ Nightcrawler +水银+金刚狼+美国队长+绿巨人+神奇先生风暴+++雷神物，Shadowcat +盗贼，头颈部，阿特拉斯的X团（雪崩+斑点+女妖+壳+银禧），亚当十，深红迪纳摩，邪恶先生，代理商（即天王星+的M - 11 ++纳莫拉+麦宇金星+大猩猩人），启示，监工，代理0 +狼獾，太空堡垒，男爵莫尔多，蝙蝠翼+蜘蛛侠，米字旗，古一，ArcLight的，变色龙，铁人。

The "Spider-Man 3" three big villain: venom, sand and green demons II, sand is the largest concentration of top villain, that he lights up the body, can become the same material as sand, everywhere flow And the venom people in the Spider-Man is injected into the body of a meteorite material in outer space, black spider clothes, although on the face of Spider-Man enhance the ability of many, but the fact Otherwise, people actually like venom version of the villain Spider-Man, his clothes have chest spider logo; Green Magic II is the first one is green demons after his death, his son Harry Green Magic play the next-.....

《蜘蛛侠3》有三个大反派：毒液人、沙人和绿魔二代，沙人是本集中的头号大坏蛋，他那发亮的身体，能够变成像沙一样的物质，到处流窜；而毒液人则是在蜘蛛侠身体中注射进一种外层空间陨石物质，让蜘蛛服变黑，虽然表面上蜘蛛侠的功力增强不少，但事实不然，毒液人其实就像是坏蛋版的蜘蛛侠，他的衣服胸口也有蜘蛛图案；绿魔二代则是第一集绿魔死后，绿魔儿子哈利接下来扮演的复。。。。。

Results Among 3 cases of repetitive cancer,2 cases were homocheonou double cancer of ascending colon cancer and rectum cancer or of right colic flexure cancer and descending colon cancer,1 case was heterochronic triple cancer of stomach cancer、lung cancer and transverse colon cancer.

结果 3例重复癌患者，有2例同时性双重癌，分别患升结肠癌及直肠癌和结肠肝曲癌及降结肠癌，1例异时性三重癌，患有胃癌、肺癌、横结肠癌。

Five models were used to investigate the "Changdong"" s anti-tumor effect: mice S180 sarcoma solid model, mice liver cancer H22 solid model, mice Lewis lung cancer model, as well as human liver cancer QGY and colon cancer LOVO implanted in naked mice; mice liver cancer H22 was also used to evaluate the enhancive effect of "Changdong" when it was co-applied with other anti-tumor agent or with radiotherapy; The "Changdong"" s cytotoxic effect in vitro on such tumors as QGY, LOVO, lung cancer A549, breast cancer MCF-7, as well as mice leukemia P388 was observed through MTT method. And investigate the effect of "Changdong" on proliferation of spleen lymphocyte, on activity of NK cell and on synthesis of IL-2 in mice bearing Lewis lung cancer, respectively. With propidium iodide straining, cell aigptosis and cell circle were analyzed by flow cytometry. Result:"Changdong" has an evident tumor inhibitive effect on mice tumor model as well as human tumor implanted in naked mice with a quantity-effect relationship.

使用小鼠S180肉瘤、小鼠肝癌H22实体瘤和小鼠Lewis肺癌模型，以及人体肝癌QGY和人体肠癌LOVO裸鼠异种移植模型，进行"长动"的抗肿瘤药效学研究：用小鼠肝癌H22进行"长动"合并放疗和化疗的增效作用研究；用MTT法进行"长动"对肝癌QGY、肠癌LOVO、肺癌A549、乳腺癌MCF-7、小鼠白血病P388等10株人体和动物肿瘤细胞的体外细胞毒性作用研究；用淋巴细胞转化试验、小鼠NK细胞活性试验、小鼠胸腺细胞增殖法分别测定"长动"对荷Lewis肺癌小鼠的脾淋巴细胞增殖的影响、自然杀伤细胞活性的影响和对小鼠产生白介素-2(IL-2)的影响；用流式细胞仪PI单染色法进行"长动"对人体肝癌QGY细胞的体外诱导凋亡作用和对肿瘤细胞周期的影响的研究。

Objective To explore livin, MTA1 and the caspase3 protein expression, their correlation and clinical pathology in colon cancer Methods The expressions of livin, MTA1 and caspase3 protein in 88 cases of colon cancer were detected by immunohistochemistry stainingResults Livin, caspase3 protein and MTA1 positive expression rates in colon cancer tissues were more than those in tissues beside the cancer Furthermore there were obvious relevance between livin protein, MTA1 positive expression rate and degree of histodifferentiation and lymph node metabasis and between caspase3 protein positive expression rate and degree of histodifferentiation of colon cancer Caspase3 protein expression had prominent inverse correlation with the livin expressionConclusions ①Over expression of livin having inhibtion on colon cancer is one of important factors of colon carcinogenesis ②Caspase3 protein expression in colon cancer tissues is inhibited to less and has prominent inverse correlation with livin expression Accordingly, suppressing caspase3 protein activity is one of channels, by which livin promotes colon carcinogenesis ③MTA1 plays the important role in histodifferentiation degree and lymph node metastasis of colon cancer

采用免疫组织化学染色方法检测88例结肠癌组织中Livin，Caspase3 及MTA1的表达情况。结果（1）结肠癌组织中Livin的阳性表达率显著高于癌旁组织，且Livin的表达与结肠癌的组织分化程度及淋巴结转移程度显著相关。（2）结肠癌组织中Caspase3蛋白的阳性表达率显著高于癌旁组织，且Caspase3蛋白的表达与结肠癌的组织分化程度显著相关。（3）结肠癌组织中MTA1的阳性表达率显著高于癌旁组织，且MTA1的表达与结肠癌的组织分化程度及淋巴结转移程度显著相关。（4）Caspase3蛋白的表达又与Livin的表达呈显著负相关。结论（1）Livin在结肠癌组织中过表达，它可能是促进结肠癌发生的重要因素之一。（2）Caspase3蛋白在结肠癌组织中表达被抑制而降低，且与Livin的表达呈负相关。因此，抑制Caspase3蛋白的活性是Livin促进结肠癌发生的途径之一。（3）MTA1对结肠癌的组织分化及淋巴转移发挥重要作用。

All 5 patients of benign polyp were found to have excellent effect after Nd-YAG laser therapy, and so for, there was no recurrence. Two (esophageal cancer 1 and rectal cancer 1) of 19 patients of G-I cancer had excellent results with tumor size reduced over 90% and endoscope could pass through the stenotic area smoothly after Nd-YAG laser treatment. Six (esophageal cancer 2, gastric cancer 4) of them had good results with tumor size reduced 50-90% and patient could take semiliquid food after laser management. Eight patients (esophageal cancer 2, gastric cancer 4, rectal cancer 2) had fair effects with tumor size reduced less than 50%, but the oral intake or stool passage was improved. Three patients (15.8%) refused further endoscopy and laser management because of poor tolerance.

这5例良性息肉经雷射治疗后，肿瘤完全消失且目前无后发现象。19位恶性癌中，经Nd-YAG雷射治疗后，效果极佳（肿瘤大小减少90%以上，内视镜可通过肿瘤狭窄处）者2例（食道癌及直肠癌各1；效果佳（肿瘤大小减少50~90%，且病人可进食半流体食物）者6例（食道癌2，胃癌4）；效果尚可（肿瘤大小减少50%以下，但饮食进步或排便较过去进步）者8例（食道癌2，胃癌4，直肠癌2；3例患者(15.8%)因不能忍受内视镜及雷射治疗之痛苦，拒绝进一步的治疗而失败。

The percentage of TGF-β〓 positive cell in the pancreatic cancer tissue (43.8±5.2)％ was significantly higher than that in adjacent pancreatic tissue (28.7±3.6)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of TGF-β〓. 2. The percentage of Tr positive cell in the pancreatic cancer tissue (41±4)％ was significantly higher than that in adjacent pancreatic tissue (23±3)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of Tr, but the expression of Tr protein was not correlated with lymphy node metastasis (P＞0.05). 3. The percentage of β-GCD positive cell in the pancreatic cancer tissue (62.5± 4.1)％ was significantly higher than that in adjacent pancreatic tissue (33.5±2.8)％, P＜0.05. The worse the cancer cells differentiated and lymphy node metastasis, the more over-expression of β-GCD in pancreatic cancer tissue, but the expression of β-GCD protein was not correlated with lymphy node metastasis. P＞0.05. 4. the expression of β-GCD was significantly correlated with TGF-β〓 and Tr in the pancreatic cancer tissue.

结果如下：1、胰腺癌组织TGF-β〓阳性细胞百分率(43.8±5.2)％明显高于癌旁胰腺组织(28.7±3.6)％，P＜0.05；且癌细胞分化愈差或有淋巴结转移TGF-β〓过度表达愈多。2、胰腺癌组织Tr阳性细胞百分率(41±4)％，明显高于癌旁胰腺组织(23±3)％，P＜0.05；且不同分化程度胰腺癌组织Tr表达强度不同，分化程度愈低，表达强度愈高，P＜0.05；但胰腺癌Tr表达强度与淋巴结是否转移无关，P＞0.05.3、对于胰腺癌组织TGF-β〓和Tr表达，检测胰腺癌组织(32例)β-GCD阳性细胞百分率分别为(62.5±4.1)％或(62±4)％，分别明显高于癌旁胰腺组织β-GCD阳性细胞百分率(33.5±2.8)％或(43±3)％，P＜0.05；不同分化程度胰腺癌组织β-GCD表达强度不同，分化程度越低，表达强度越高，P＜0.05；但胰腺癌组织β-GCD表达强度与淋巴结是否转移无关，P＞0.05.4、TGF-β〓、Tr和β-GCD在胰腺癌组织中的表达随着分化程度的改变，呈现一致性的关系，而且TGF-β〓与淋巴结转移有关，Tr和β-GCD与淋巴结是否转移无关。

Result: With a high cancer-inhibiting rate on the animal model having been transplanted the cancer, this medicine can remarkably restrain the growth of cancer cells cultivated in vitro and improve the life quality of mouse model, and also shows obvious promotion and enhancement function on the DTH ear-swelling of mouse with cancer, the proliferation of mouse spleen lymphocyte induced by ConA and the cytokine (TNF-α、IFN-γ) of mouse with cancer respectively. It can obviously decrease the adverse effects like weight reduction, damage of viscera and degradation of life quality caused by the chemotherapeutic medicines, and has a remarkable stimulation on the lymphocyte proliferation induced by ConA. It is also able to improve the ratio of SMMC-7721 cells in G0-G1 period and reduce the cell amount in G2/M period to cut down the possibility of stepping into the division period; additionally, it can obviously inhibit some cancer genes of the cancer cells and meanwhile up regulate the cancer-inhibiting gene.

结果：本品能明显抑制体外培养肿瘤细胞的生长，对在体肿瘤移植模型动物具有较高的抑瘤率，能明显提高模型小鼠的生存质量；对荷瘤小鼠DTH耳肿胀、ConA诱导的小鼠脾淋巴细胞增殖、荷瘤小鼠细胞因子（TNF-α、IFN-γ）呈现明显的促进或提高作用；能显著减少化疗药所引起的小鼠体重降低、脏器损害以及生存质量下降等不良作用；本品含药血清对ConA诱导的淋巴细胞增殖具有明显的促进作用；能提高SMMC-7721细胞G0-G1期比例，降低G2/M期细胞数目，而减少其进入分裂期；对肿瘤细胞的多个癌基因具有明显的抑制作用，同时上调抑癌基因等。

Finally it offers the analysis of the fault-tolerance system as well as its test verification.

这样，运行于空间环境中的系统的设计目标就是在保证系统实时性

However, it is still difficult to find out the real reason of the increase of the keratinized gingiva during the orthodontic extrusion whether it is due to the proliferation of the gingival tissue or to its elastic nature, that is because the proliferation of the fibroblasts as well as the connective tissue changes in volume and the intra-fibers spaces were all immeasurable in most of the studies.

然而，仍然难以找到真正的原因增加的角化牙龈在正畸挤压它是否是由于扩散的牙龈组织或其弹性性质，这是因为增殖的成纤维细胞以及作为结缔组织的变化量和内部纤维空格都是不可估量的，大多数的研究。