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Results: Compared with IR group , XSOL could significantly decrease the cerebral water content, the cerebral index, the brain vascular permeability in rats of acute incomplete brain ischemia-reperfusion (P.01,P.05). Cerebral tissue morphology showed that neuronic damage of rats subjected to acute incomplete brain ischemia-reperfusion was ameliorated following administration of XSOL (2.4, 9.6g/kg). Compared with IR group , XSOL could significantly decrease the content of MDA and NO in brain tissue(P.01,P.05)and increase SOD,GSH-px activity. The levels of cerebral Glu,Asp were cut down(P.01,P.05). Conclusion: XSOL shows protective effects on cerebral ischemia-reperfusion.

结果:与缺血再灌注组比较,XSOL大、小剂量组可以显著降低急性不完全性脑缺血再灌注模型大鼠的脑含水量(P.01,P.05)、脑指数(P.01,P.05)、脑毛细血管通透性(P.01,P.05);光镜下脑组织形态学显示各剂量组可不同程度的减轻急性不完全性脑缺血再灌注大鼠脑组织的神经元受损形态;与缺血再灌注组比较,XSOL大、小剂量组可以显著降低缺血再灌注大鼠脑组织中的MDA的含量(P.01,P.05),升高SOD、GSH-Px在脑组织中的活性(P.01,P.05);XSOL大、小剂量组可显著降低脑组织中NO的含量(P.01,P.05),降低脑组织中Glu、Asp含量(P.01,P.05)。

Results: Comparing with movement fatigue group, blood glucose contents were significantly increased but oxypropionic acid and urea nitrogen contents were significantly increased; testosterone and T/C were significantly advanced, myocardium SOD activity was significantly increased, while MDA was significantly decreased in cluytyl alcohol preparation group.

结果:和运动疲劳组相比,补充二十八烷醇制剂组大鼠血糖显著升高,血乳酸、血尿素氮显著降低;T、T/C比值显著增高,心肌SOD活性显著增强,MDA含量显著降低,且效果优于丙酮酸-肌酸组。

Increase in SOD and POD activities in Paeonia suffruficosa leaves enhanced the resistance to drought stress.

干旱胁迫下,牡丹叶片SOD和POD活性增强,在一定程度上提高了植株的抗旱能力。

Fenvalerate could cause certain oxidative damage on pakchoi, and cause SOD, CAT, POD activities change in different degrees.

氰戊菊酯可对小白菜产生一定的氧化损伤,导致SOD、CAT、POD活性发生不同程度的变化。

The drug of Xin xiebai powder can raise the content of SOD , at the same time reduce the content of the toxic oxygen free radicals and the injury by lipid peroxidate and lighten the lung damage by the oxidation effects of stress reaction.It confirms the security of Xin xiebai powder by acute toxic experiment.

能够提高机体 SOD 的含量,从而减轻毒性氧自由基的含量,降低机体内脂质过氧化,减轻氧化应激反应引起的肺损伤。

The MDA content and SOD activity play an important role in the process of hepatocyte apoptosis and lipid peroxidation.

MDA含量和SOD活力在酒精性肝病的肝细胞凋亡过程及脂质过氧化反应中发挥重要作用。

Eight QTLs for SOD activity were detected on chromosomes 1, 2, 3, 4, 7, 8 and 9, which could explain 42. 44% of phenotypic variance.

检测到8个与SOD酶活性有关的QTL,分别位于第1、2、3、4、7、8和第9染色体上,可以解释表型变异的42.44%。

Methods: Experimental mice were perfused with different dosages of tetracycline. Eighteen hours later, the serum levels of ALT, AST were measured and the liver tissues were observed by photics microscope to find the relative suitable dosage that can obviously induce acute liver injury. Then the suitable dosage was given in the same way, and the levels of ALT, SOD, MDA, GSH-PS, IL-18 in serum were measured 18, 36, 54, 72 h later respectively. The condition of cell apoptosis was analyzed, and the tissues were observed by photics microscope and electron microscope to find the relative suitable time.

先以不同剂量四环素灌胃,18h后检测小鼠血清ALT、AST水平并进行光镜观察,探索导致明显急性肝损伤的相对合适剂量;再以相对合适剂量四环素灌胃,18、36、54、72h后分别检测小鼠血清ALT、SOD、MDA、GSH-PX、IL-18水平及肝细胞凋亡情况,并进行光镜、电镜观察,探索导致明显急性肝损伤的相对合适时间及可能机制。

A rapid and effective' test of SOD activity in fresh tomato by the polarographic oxygen electrode is reported in this paper.

本文用极谱氧电极法检测新鲜西红柿中SOD的活力,使用了不同的抽提方法,结果表明用pH7。

A rapid and effective' test of SOD activity in fresh tomato by the polarographic oxygen electrode is reported in this paper.

本文用极谱氧电极法检测新鲜西红柿中SOD的活力,使用了不同的抽提方法,结果表明用pH7.8磷酸缓冲液加定量KH2PO4的抽提方法,可达到快速、微量、准确的检测效果

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