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shock-head相关的网络例句

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AIM: To investigate alteration and cross link of the aortic and platelet endogenous L-arginine/NOS/NO pathway induced by septic shock. METHODS: The septic shock model was made in rats by caecal ligation and puncture.

目的:观察感染性休克对大鼠血小板及血管L-精氨酸/一氧化氮合酶/一氧化氮通路的影响及其相互联系,探讨感染性休克损伤的机制。

Even when treated, mortality from cardiogenic shock after MI and from septic shock is high (60 to 65%).

即使加以治疗,心肌梗塞后的心源性休克和败血症性休克的死亡率也很高(60-65%)。

Causes are listed in Table 2: Shock and Fluid Resuscitation: Mechanisms of Cardiogenic and Obstructive Shock .

原因见表2:心源性和阻塞性休克机制。

This suggests that antibody or inhibition of cGMP-dependent protein kinase may prevent vascular hyperpermeability of severity burn shock and septic shock,and fundamentally take on a new look in treatment of shocks instead of traditional and passive clysis.

提示运用PKG抗体或抑制剂有可能防治大面积烧伤和中毒性休克时的血管通透性增加导致的血容量减少的发生,从根本上改观传统的、被动的烧伤补液抗休克方法。

We used maize seedlings (Zea mays L.) and tobacco (Nicotiana plumbaginjfolia L.) as material. In the first section, we have investigated cold- shock-induced chilling, drought, heat and salt stress resistance in maize seedlings. In the second section, by modifying the Ca~ and CaM levels, we have tried to establish the involvement of calcium messenger system in the cold-shock-induced cross adaptation. In the third section, we have tried to establish a transgenic tobacco system expression CaMICaMIBP genes, so that the others can use it to investigate the process of physiology and biochemistry which endonuclear and cytosolic calmodulin have taken part in.

以玉米幼苗和烟草为实验材料,本文第一部分通过将玉米幼苗进行短期冷驯化后对冷胁迫、干旱胁迫、热胁迫及盐胁迫的影响,试图证明玉米幼苗中交叉适应现象的存在及其机理;第二部分通过外源修改Ca~(2+)和CaM水平,研究了钙信使系统在交叉适应现象中的可能作用;第三部分通过建立诱导性表达CaM/CaMBP转基因烟草体系,便于研究受细胞质CaM和细胞核CaM调控的各种生理生化过程。

Therefore, how to prevent and cure endotoxic shock is a big issue that needs to be concerned. LPS is the primary cause for endotoxic shock.

因此,如何防治内毒素休克的发生是重症医学所关注并亟待解决的重大课题。

Effects of sustained posttraumatic shock and initial fluid resuscitation on extravascular lung water content and pulmonary vascular pressures in a porcine model of shock[J].

急性超容量血液稀释对氧供氧耗、循环血容量和血管外肺水的影响[J]。

Objective: Through studying change of serum level of vital inflammatory cytokines and vital organs function in patients of severe craniocerebral injury with haemorrhagic shock, to investigate effects of severe trauma with haemorrhagic shock bypass on inflammatory reaction and organs function, and to discuss relations of these three aspects.

目的:通过对重度颅脑损伤伴失血性休克患者的主要炎性反应介质和重要器官功能指标的研究,旨在探讨重度创伤伴失血性休克对机体炎性反应和重要器官功能的影响及其三者之间相互影响的关系。

Results The peak of antibacterial activity appeared at 36-48 hafter induced by four inducements (Because many M.domestica larvae pupated,we did not observe the peak of antibacterial activity by heat-shock). The antibacterial activity of antibacterial proteins by S.aureus was best,and the antibacterial activity of antibacterial proteins by heat-shock was better than control group.

结果 各诱导组诱导家蝇幼虫产生抗菌蛋白达最大活性时间为诱导后36~48h(热诱导后家蝇幼虫很快化蛹,未观察抗菌蛋白产生高峰时间);金黄色葡萄球菌诱导后的抗菌蛋白抗菌活性最佳,热诱导后的抗菌蛋白抗菌活性也较未诱导组好。

In the middle of 90's, it has been found that hydrogen sulfide (H2S) is one of the metabolite of aminothiopropionic acid, which might regulate the function of nervous system, especially hippocamp, and might regulation the tension of digestive tube and vascular, which has the similar biological function with NO and CO. Recently, major concern has emerged that H2S might be a novel endogenous agent during the patho-physiological reactions during endotoxic shock. Until now, the role of H2S playing in endotoxic shock is still quite confusing.

九十年代中叶,人们发现在体内半胱氨酸代谢过程中生成小分子气体硫化氢(hydrogen sulfide,H2S),对神经系统特别是海马功能具有调节作用,且可以调节消化道和血管平滑肌张力,其生物学作用类似NO和CO.H2S作为内毒素休克时一系列病理生理反应中的一种新的内源性介质,已开始受到人们的重视。

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推荐网络例句

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。