查询词典 sensibilization

In addition,the steel has also better intergranular cor rosion resistance after sensibilization treatment.

该合金钢敏化处理后，仍具有较好的抗晶间腐蚀性能。

Bronchial asthma in acute episode model was established by a manner of egg albumen sensibilization and provocation.

以卵白蛋白致敏、激发的方式建立支气管哮喘急性发作期模型

Based on the mechanism of the fiber Bragg grating's cross-sensitivity to temperature and strain, a new fiber Bragg grating superficial bonded strain sensor with temperature compensation and strain sensibilization was designed and fabricated.

在分析光纤Bragg光栅的温度应变交叉敏感机理的基础上，设计并制作了一种新型的具有温度补偿和应变增敏效果的光纤Bragg光栅表面粘贴式应变传感器。

No sensibilization effect was observed as well.

提示&软坚祛僵散&的药效可靠，使用安全。

Bronchial asthma in acute episode model was established by a manner of egg albumen sensibilization and provocation. Rats in each group were treated by different drugs. Protein expressions of Fas/FaL-s in lung tissue were assayed by immune histochemistry.

以卵白蛋白致敏、激发的方式建立支气管哮喘急性发作期模型，各组分别给予相应治疗，免疫组化ABC法检测肺组织中Fas/FaL-s的蛋白表达。

The pathogenesis of asthma can be divided into three stages in immunology. The initial stage is sensibilization, which is closely associated with the activation of T cells. B7/CD28 and CD69 also play a significant role in this process. The second stage is chronic allergic inflammation. The immune dysfunction resulting from the disturbance of TH1/TH2 is a major cause of chronic allergic inflammation, and dominates asthma attack. And the third stage is airway remodeling due to chronic inflammation. Airway remodeling is one of the major causes of refractory asthma, which also contributes to the death from asthma.

哮喘的发病从免疫学角度可以分为三期：初期为致敏阶段，与T细胞激活关系密切，B7/CD28和CD69在这一过程也扮演重要角色；第二期为慢性过敏性炎症阶段，TH1/TH2失衡导致免疫功能紊乱是此阶段的主要原因，在哮喘发作过程中起主要作用；第三期为慢性炎症引起的气道结构重构阶段，是难治性支气管哮喘的重要发病和死亡原因。

Methods 24 SD rats were randomly divided into normal group,model group,aminophylline treatment group(35 mg/kg), 8 rats in each group. The bronchial asthma model in rats was built by egg protein sensibilization and longterm inhalation provocation. The rats in treatment group were lavaged each day from the first time of provocation.After 4week treatment, the rats were killed to obtain lung tissue for staining with HE.

24只SD大鼠随机分为正常组、模型组、治疗组，每组8只，除正常组外以卵蛋白致敏并吸入激发法制备大鼠哮喘模型，治疗组、模型组从第1次哮喘激发开始(造模第3周)分别给予氨茶碱、0.9%氯化钠溶液灌胃给药，1次/d，用药4周处死大鼠，取肺组织HE染色，用彩色图像分析仪测量支气管壁面积、支气管平滑肌面积，用ELISA双抗体夹心法测定肺组织MMP9和TIMP1含量。

The bronchial asthmatic model was established by egg protein sensibilization and longterm inhalation provocation.

40只SD大鼠随机分为正常组、模型组、咳喘宁高、低剂量组(27 和13.5 g生药/kg体重)、桂龙咳喘宁胶囊对照组(0.41 g/kg体重)，每组8只。

The bronchial asthmatic model was established by egg protein sensibilization and longterm inhalation provocation.The rats of each treatment groups were lavaged each day from the first time of provocation to execution.After 4 weeks of treatment,the rats were killed to obtain lung tissue for dyeing of HE.A computer assisted image analysis system was used to determine the thickness change of bronchus wall and smooth muscle.The contents of MMP9 and TIMP1 in lung tissue were determined by ELISA double antibody sandwich method.

除正常组外以卵蛋白致敏并吸入激发法制备大鼠支气管哮喘模型，各治疗组均从第1次哮喘激发开始(造模第3周)至处死前每天灌胃给药，激发并给药4 w后处死大鼠，取肺组织HE染色，彩色图像分析仪测量支气管管壁厚度、平滑肌厚度，采用ELISA双抗体夹心法测定肺组织MMP9、TIMP1含量。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。