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scar tissue相关的网络例句

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Results: Among 80 cases of ACST, 76 cases were successfully implemented LC, gallbladder puncture and decompression choledocholithotomy T-tube drainage were performed in 11 cases, choledocholithotomy T-tube drainage in 15 cases, common bile duct gall bladder fistula Toishi T-tube drainage in 14 cases, gallbladder removal choledocholithotomy T-tube drainage in 36 cases, operative time 15-120 min, bleeding 10-80 ml; 4 cases transitted laparotomy, 1 case of gallbladder and surrounding tissue adhesion serious, ampulla signs disappear, and the hepatoduodenal ligament ill-defined, 2 cases of gallbladder contraction deep within the liver, gallbladder triangle scar adhesions, and 1 case of duct stone impaction and severe adhesions; postoperative hospital stay was 8-11 d, all were well recovered; pathological diagnosis, acute cholecystitis in 41 cases (51.2%), acute suppurative cholecystitis in 24 cases (30.0%), acute gangrenous cholecystitis in 15 cases (18.8%).

结果:重症急性胆囊炎80例,76例成功实施LC,其中,胆囊穿刺减压胆总管切开取石T管引流11例,胆总管切开取石T管引流15例,胆囊造瘘胆总管切开取石T管引流14例,胆囊切除胆总管切开取石T管引流36例,手术时间15~120 min,术中出血10~85 ml;4例中转开腹,其中,1例胆囊与周围组织粘连严重,壶腹部标志消失,与肝十二指肠韧带界限不清,2例胆囊萎缩深陷肝脏内,胆囊三角区瘢痕粘连,1例胆囊管结石嵌顿且严重粘连,术后住院时间8~11 d,术后恢复均良好,治愈出院;80例术后病理诊断,急性单纯性胆囊炎41例(51.2%),急性化脓性胆囊炎24例(30.0%),急性坏疽性胆囊炎15例(18.8%)。

Intranasal endoscopic holmium laser-assisted dacryocystorhinostomy causes no scar in the face, less nasal tissue damage, shorter operation time and less hemorrhage, and does not affect the lacrimal irrigation system, which allows correction of intranasal causes of failure in traditional dacryocystorhinostomy.

鼻内镜下钬激光泪囊鼻腔造孔术后,患者面部无瘢痕,鼻部组织损伤小,手术时间短,出血少,泪液的导流系统不受影响,是传统的泪囊鼻腔吻合手术失败后的良好补救方法。

Wounds such as road accidents, gunshot injuries, fire burns and some internal medicine diseases like diabetes, tumors and palsies always accompany with muscle injuries. Muscle regeneration is relatively difficult on the conditions of serious and large area of muscle injury or loss. Finally the raw surface will be replaced by fibrous connective tissue and form scar, which may disenable the movement of body and badly influence nerves and blood-vascular system, and eventually develop into obduracy wound.

各种创伤如车祸伤、枪弹伤、烧伤及部份内科疾病如糖尿病、肿瘤和瘫痪所致的肢体坏疽都常伴肌组织损伤,对于严重或面积较大的肌损伤或缺损,肌肉的再生相对困难,创面最终被纤维结缔组织替代而形成瘢痕,导致机体运动功能障碍并累及神经、血管系统,影响患者的预后和生活质量。

Methods Nuclear β-catenin expression was detected by immunohistochemistry in 77 lesions with desmoid-type fibromatosis and 171 other spindle cell lesions, including superficial fibrmoatosis (n=18), nodular fasciitis (n=36), keloid (n=16), scar (n=10), granulation tissue (n=9), synovial sarcoma (n=38), neufibroma (n=13), solitary fibrous tumor (n=12), gastrointestinal stromal tumor (n=10), low-grade myxofibrosarcoma (n=3), low-grade fibromyxoid sarcoma (n=3), and smooth muscle tumor (n=10). In addition, the immunohistochemical expressions of ER-α, ER-β and Ki-67 were examined in all of the lesions with desmoid-type fibromatosis. The nuclear immunohistochemical staining for nuclear βcatenin and ER-β was graded as high level (≥25% of cells), low level (5%-25%) or none.

采用免疫组织化学染色法检测77例韧带样型纤维瘤病和171例其它良、恶性梭形细胞病变(包括结节性筋膜炎36例、浅部纤维瘤病11例、瘢痕疙瘩16例、增生性瘢痕10例、肉芽组织9例、滑膜肉瘤38例、神经纤维瘤13例、孤立性纤维性肿瘤12例、胃肠间质肿瘤10例、低度恶性黏液纤维肉瘤3例、低度恶性纤维黏液样肉瘤3例及平滑肌肿瘤10例)组织中β-catenin核阳性的表达,同时检测韧带样型纤维瘤病中ER-α、ER-β和Ki-67的表达。

Our studies indicate that the increased contents of glucose and accumulation of active metabolic intermediates due to metaboic disorder contribute to the cutaneous environmental alterations associated with diabetes mellitus.Thus,the cutaneous environmental alteration is one of the initial events for impaired healing.The succession of progressive injury in deep partial thickness burn wounds is carried out depending on the physiological outcome of parabiotic tissues.The loss of dernal template due to the damage to the integrity and the continuity of dermal tissue may be one of the leading reasons of hyperplastic scar formation.

通过对糖尿病合并创面难愈、烧伤创面的进行性损害以及增生性瘢痕形成的机制研究,我们发现:糖尿病代谢紊乱所致皮肤组织中糖含量增高和代谢产物蓄积引起的皮肤微环境改变,是导致糖尿病创面难愈的始动因素之一;烧伤后的组织进行性损害的发生发展取决于&间生态&组织的病理转归;真皮组织缺损导致的模板作用缺失可能是引起增生性瘢痕形成的始动环节。

In which, microglia phagocytize waste products of tissue cells, partake the damage treatment and formation of scar through proliferation or migration during neural development, degeneration or excitotoxicity.

其中的小神经胶细胞会在神经发育与退化时,藉由proliferation、migration或分泌细胞激素的方式,进行吞噬受损细胞的残骸,及参与伤害的治疗。

At 2 weeks following injury, hematoxylin-eosin staining showed that brain tissue breakage at damaged site as scar connection, remarkable porosis in the blank group; typical morphological changes as neural cells at the transplanted site in the control group; typical morphological changes as neural cells without cavity in the experimental group.

伤后2周苏木精-伊红染色空白组可见损伤处脑组织断裂,为瘢痕连接,有明显空洞形成;对照组在移植部位出现典型的神经细胞样形态学改变;实验组出现典型的神经细胞样形态学改变且空洞消失。

Attachment scar concave, with umbilical tissue, margin with ringlike emergence.

附件痕凹,控制用的具组织,边缘具出现。

Objective: To offer a method for the large soft tissue defect in forearm after scar excised.

目的:前臂瘢痕切除后,为创面修复提供简单、想的治疗方法。

This study was conducted to examiune the fibrotic effect of Ni-Ti and 317L al loys in esophagus.The extract fluid from Ni-Ti,317L alloys was made according t o the ASTM standards of U.S.A. The Fb of esophageal scar was cultured primarily ,then incubated with alloy abstract fluid. The proliferating activity of Fb was measured by MTT at 4, 24, 48, 72 hours in the course of culturing. The esophagu s embedding test of Ni-Ti,317L alloys was made according to ASTM standards of U .S.A.The tissue around the alloys was taken at weeks 2 and 12,and the pathologi c changes were analysed.

为探讨新型支架材料Ni-Ti、317L合金在食管局部的致纤维化作用,按美国ASTM标准制备NiTi、317L合金的金属浸提液;&组织块培养法&原代培养食管壁疤痕的成纤维细胞,传代后以金属浸提液进行培养,分组后分别培养4、24、48、72 h,MTT法检测不同培养时间后Fb增殖功能的变化;按美国ASTM标准进行NiTi、317L合金试件的食管壁内包埋实验,即将金属试件经表面处理后直接置入食管壁粘膜层与肌层之间,术后2、12周取出包埋组织,分析试件周围组织的病理变化,并进行胶原纤维染色,观察纤维形成状况。

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