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sarcomere相关的网络例句

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与 sarcomere 相关的网络例句 [注:此内容来源于网络,仅供参考]

Clinical evaluations demonstrated familial disease in 6 of 11 probands with sarcomere gene mutations.

经过临床调查证实,11个先证者有6个有肌节基因突变。

These genes encode sarcomere proteins that, when mutated, cause adult-onset cardiomyopathies.

这些基因编码的肌节蛋白质,当其突变时会导致成人心肌疾病。

In chapter one, the molecular architecture of the sarcomere, the structures of myosin and actin, and the mechanochemical actin-activated myosin ATPase cycle are introduced.

第一章主要介绍肌节的结构、肌球蛋白和肌动蛋白的分子结构、肌球蛋白工作循环中的机械化学偶联。

Fig. 14 B group PCAM, The structure of sarcomere of skeletal muscle is clear, The structure of muscle fiber restore to normal.

图14 B组PCAM,骨骼肌肌节结构清楚,肌纤维结构已恢复正常×20 000

Some recent studies suggest that DOMS may result from multifactor such as the formation of sarcomere, disorder of microcirculation, and changing of peripheral nervous system.

新近的一些研究成果表明延迟性肌肉酸痛可能与肌节重塑过程、微循环紊乱和外周神经系统的变化有关。

We speculated that mutations in sarcomere protein genes known to cause hypertrophic cardiomyopathy and dilated cardiomyopathy may be associated with left ventricular noncompaction.

我们推测:已知的可导致肥厚性心肌病和扩张性心肌病的肌节蛋白基因突变,可能和左室心肌致密化不全相关。

During contraction, these filaments slide past one another, thereby shortening the length of the sarcomere.

由于肌球蛋白和肌动蛋白的作用,粗丝和细丝相对滑动引起肌肉的收缩。

LVEDP is as high as 30mmHg,sarcomere length is still 2.2 m.

左室舒张末压高达30mmHg时,肌小节长度仍是2.2 m。

In the present study, we investigated whether protein composition and function of the sarcomere are altered in a homogeneous familial hypertrophic cardiomyopathy patient group with frameshift mutations in MYBPC3 (MYBPC3mut).

在本次实验中,我们以MYBPC3基因移码突变的同一种族的家族性肥厚性心肌病患者为研究对象,对这些患者肌小节的蛋白构成及功能是否发生改变进行了研究。

Only a limited number of sarcomere gene defects (eg, cardiac actin Glu101Lys) consistently produce apical HCM.

只有少数的肌节基因缺陷(例如Glu101Lys心肌肌动蛋白)一贯性的造成心尖肥厚型心肌病。

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