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ruthenium red相关的网络例句

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与 ruthenium red 相关的网络例句 [注:此内容来源于网络,仅供参考]

But the levels of 〓 elevation were lower than that in normal Tyrode's solution (P<0.01 respectively), indicating that Ca〓 influx did contribute to the elevation of 〓. Ryanodine remarkably blocked the 〓 elevation by Str 1, 100nmol/L and 10μmol/L in normal Tyrode's solution, while ryanodine 10μmol/L, ruthenium red 10μmol/L and caffeine 10 mmol/L completely blocked the effects of Str on 〓 elevation in Ca〓- free Tyrode's solution (P<0.01 respectively).

去除细胞外液中的钙离子,Str 1,100nmol/L和10μmol/L仍能分别升高〓到13±5%,71±8%和145±14%,但幅度低于其在正常台氏液中的作用。10μmol/L的ryanodine,10μmol/L钌红或10mmol/L咖啡因均可完全阻断Str 1,100nmol/L和10μmol/L在无钙台氏液中的作用(P<0.01)。

These stains may fade in time, e.g. ruthenium red. Or the stain may damage the specimen if left, for example SCHULTZE ' S SOLUTION, which dissolves cell walls and causes swelling.

这种染料经过一段时间后会褪色,如钌红;也可能会因为长时间存在而损伤标本,如 Schultze 溶液可以溶解细胞壁使细胞膨胀至死。

RESULTS: Both HPC and treatment with ruthenium red (5μmol/L) during the first 10 min reoxygenation improved recovery of LVDP±dp/dt and decreased LVEDP, which was associated with reduced infarct size and lactate dyhydrogenase release.

用TTC染色法测心肌梗死面积。结果:低氧预处理和低氧后复氧期的前10min用钌红(5μmol/L)处理均可促进LVDP、±dp/dt,和LVEDP的恢复,减小心肌梗死面积,减少LDH释放。

RESULTS: Both HPC and treatment with ruthenium red (5 μmol/L) during the first 10 min reoxygenation improved recovery of LVDP,±dp/dtmax and decreased LVEDP, which was associated with reduced infarct size and lactate dyhydrogenase release.

用TTC染色法测心肌梗死面积。结果:低氧预处理和低氧后复氧期的前10 min用钌红(5 μmol/L)处理均可促进LVDP、±dp/dtmax和LVEDP的恢复,减小心肌梗死面积,减少LDH释放。复氧期的前10 min用精胺(20 μmol/L)处理可减弱低氧预处理的心脏作用。

However, no additive effect could be found when caffeine and ETX were concomitantly incubated. Ruthenium red, which is a blocker of cation channels and perhaps a blocker of CICR, could inhibit the effects of caffeine and ETX by 94% and 80%, respectively.〓, replacing 〓 in Kreb's solution, inhibited ETX-evoked CGRP release by 82%. Ryanodine (1-100μM) or Tris-ATP had no effect on both basal and ETX-induced CGRP release.

钌红能分别抑制咖啡因或内毒素引起的CGRP释放,如以氯化镁代替Kreb's液中的氯化钙时,内毒素引起的CGRP释放可降低82%,Ryanodine 1-100μmol/L或Tris-ATP对CGRP基础释放和内毒素引起的CGRP释放均未见明显影响。

Current properties reveal that the mCa1 channel underlies the human MCU and that the mCa2 channel is responsible for the ruthenium red–insensitive/low-sensitivity non-MCU–type mitochondrial Ca2+ uptake.

现有的功能研究表明mCa1是人单向转运体的基础,mCa2通道则在钉红不敏感/轻度敏感的非单向转运体钙离子摄取途径中扮演重要角色。

The result using inhibitors of Ca〓 channels supported above observation. When Ca〓 channels in plasma membrane were blocked by LaCl〓, verapamil or nifedipine, and the Ca〓 channels in endomembranes opened naturally, extracellular CaM couldn't induce 〓 increase. When Ca〓 channels in endomembranes were blocked by ruthenium red and the Ca〓 channels in plasma membrane opened naturally, extracellular CaM induced Ca〓 increase in guard cells, but the extent of Ca〓 increase was lower than that of control.

使用质膜Ca〓通道抑制剂LaCl〓、异博定、尼群地平及细胞内Ca〓通道抑制剂钌红的结果支持这种推测:在质膜Ca〓通道被阻断、细胞内Ca〓通道仍正常开放的情况下,保卫细胞〓不能被CaM诱导增加;细胞内Ca〓通道被抑制、质膜Ca〓通道正常开放的条件下,细胞外CaM诱导保卫细胞内〓升高的幅度不能达到不使用Ca〓通道抑制剂的水平。

Transmission electron microscope samples with ruthenium red electron microscope cytochemistry technique was prepared for detecting the cell-wall polysaccharide and extracelluar polysaccharide of Cladosiphon okamuranus Tokida.

为进行经济海藻枝管藻细胞壁多糖和胞外多糖的超微定位,本文以钌红电镜细胞化学方法制备枝管藻透射电镜样品。

The concentration-response curve of GABA was shifted downward by hypotonic without any change of the threshold value.The values of two curves were very close(33.02μM and 29.41μM). Hypotonic decreased the maximal amplitude of IGABA by(31.38±2.13)%.③This inhibitory effect was reversed by ruthenium red, a nonselective TRPV4 receptor antagonist, and was increased by 4ɑ-PDD, a selectively TRPV4 receptor agonist.

低渗透压+GABA的量效曲线较单独的IGABA量效曲线明显下移,而两者的阈值和最大反应浓度基本不变,分别为10μM和1000μM;两条曲线的EC50值非常接近,分别为29.41μM和33.02μM;预加低渗透压后最大反应浓度时,IGABA的幅值减少了(31.38±2.13)%。

Unilateral perineural application of 3% capsaicin selectively affects afferent C-fibers, and results in reduction of SP-and CGRP-IR and increase of NADPH diaphorase activity in ipsilateral superficial laminae of dorsal horn; Also, the application of Cap results in significant decrease in GABA-IR neuroprofile and increase in expression of cfos in many neurons the same area; Ruthenium red, as a functional antagonist of capsaicin, can completely prevent capsaicin-induced depletion of SP-and GABA-IR; Capsaicin-induced GABA-IR reduction can be partially blocked by pretreatment with a NK-1 receptor antagonist spantide and a NMDA receptor antagonist APV to the spinal dorsal horn.

辣椒素急性施加于外周神经可选择性影响C类传入纤维,引起末梢终止区域内SP-和CGRP-IR的减少,NO合成酶活力增加;同时也引起此区域内GABA-IR的减弱,GABA-IR神经元数目的减少以及背角浅层C-fos的大量表达;Cap的功能拮抗剂能完全翻转Cap所致的SP和GABA-IR的减少;应用NK-1受体和NMDA受体拮抗剂均可部分阻断Cap引起的GABA-IR的减少。

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