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renal colic相关的网络例句

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Objective:to explore the evaluation of color doppler ultrasonography in differential diagnosis of renal cystic disease.methods:theultrasonographic finds of cystic renal cancer in 4 cases,renal cyst in 30 cases, renal tuberculosis in 3 cases,nephropyosis in 8 cases,renal hydatid in 2 cases were analyzed.compared with each other in size,shape,edge, echo of cyst wall and cyst water,intracapsular membrane,blood flood.results:two-dimensional and color ultrasonography are helpful in showing the characteristic of structure and blood flow.the signal of blood flow can be found in cystic wall.node of cystic wall and intracapsular membrane of cystic renal cancer.the rate ofintracapsular membrane and node of cystic wall is higher than nephropyosis,renal tuberculosis and cystic renal disease.

目的:探讨彩色多普勒超声在肾脏囊性病变鉴别诊断中的价值。方法:对临床和病理证实的4例囊性肾癌,8例肾脓肿,2例肾包虫,3例肾结核,30例肾囊肿的二维超声及彩色多普勒超声表现进行了分析,对每例病灶的大小、形态、边界、囊壁回声、囊液回声、囊内分隔及彩色血流的有无、性质进行比较。结果:二维超声和彩色多普勒超声可以反映肾脏囊性病变的结构特征及血流特征。

Results The main imaging of adult polycystic kidney disease signs: abdominal plain film (45 cases) showed renal shadow was lobulated increase; cystic wall calcification (14 cases); polycystic kidney disease and renal milk of calcium (8 cases); pelvis angiography (61 cases), the performance of the renal pelvis, renal calices separated, distorted, elongated; retroperitoneal inflatable angiography (6 cases): Performance increases of renal kidney shadow Shadow and the relationship between the neighboring organs, its boundaries clear and identified; CT scan (30 cases): the performance of the majority of the size of the renal low-density areas, ranging from thin-walled circular; B-mode ultrasonography (47 cases) showed a circular echo-free renal multiple classes of liquid dark area .

结果 成人多囊肾的主要影像学征象:腹部平片(45 例)显示肾影呈分叶状增大;囊壁钙化(14例);多囊肾合并肾钙乳(8例);肾盂造影(61例),表现为肾盂、肾盏相互分离、变形、拉长;腹膜后充气造影(6例):表现为肾影增大肾影与邻近器官之间的关系,其界限清晰可辨;CT扫描(30例):表现为肾内多数大小不等的薄壁圆形低密度区;B型超声(47例)表现为肾内多个类圆形无回声的液性暗区。结论影像学检查有助于提高多囊肾的诊断的准确性。

Were used to detect the experimental group and control group, supine position when the left renal vein compression section diameter of the expansion of paragraph a, b, left renal vein peak velocity pressure above Va, the pressure gradient, pressure, as well as the expansion of left renal vein peak velocity Vb paragraph and to observe the two forms of the spectrum, and then testing all the main subjects with renal renal artery, interlobar artery and renal blood flow velocity of the main renal artery resistance index; record of the experimental group urine test subjects in the number of red blood cells.

分别检测实验组及对照组平卧位时左肾静脉受压段、扩张段内径a、b,左肾静脉受压段峰值血流速度Va、压力阶差、压力,以及左肾静脉扩张段峰值血流速度Vb并观察两处的频谱形态,再检测所有受检者双肾主肾动脉、叶间动脉的血流速度及双肾主肾动脉阻力指数;记录实验组受检者尿常规化验中红细胞数量。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后,血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少,Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少,血小板聚集程度及红细胞聚集指数显著上升,病理切片显示胸主动脉内皮细胞破坏明显,脑心肾组织内的血管进一步增厚甚至几乎阻塞,导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR,脑体重比显著升高;心肌内出现明显的缺血梗塞疤痕灶,心体重比显著升高;肾小球明显萎缩、玻璃样变性,肾小管也变性甚至坏死,使肾硬化程度显著增加呈颗粒性固缩肾,肾体重比也显著减少。

Results: in contrast with model group, SSM can (1) increase the survival rate of ARF in rats, cut down Scr and BUN at 24hr, improve renal function, slightly surpass Vp in its therapeutic effect;(2) mitigate the extent of renal pathologicchanges, decrease the numbers of renal tubule necroses and casts;(3) protect renal ultrastructure such as microvilli, mitochondria, endoplasmic reticulum of TEC, podocytic process of glomerulus epithelial cell,endothelial cell etc, better than Vp;(4) enhance SOD activity, lower MDA contents in renal cortex;(5) enhance NO contents in serum, reduce ET levels in plasma;(6) evidently cut down TNF- a contents in serum, superior to Vp;(7) antagonize calcium overload in TEC;(8) alleviate renal pathological changes in ARF metaphase (at 3d), advance regeneration and recovery of TEC, restore renal structure in ARF convalescence (at 5d), and better than Vp;(9) increase the expression of TEC PCNA and prepro EGFmRNA in ARF metaphase.

结果:与模型组相比,SSM能(1)提高ARF大鼠的存活率,降低24hr时Scr及BUN,改善肾功能,其疗效略优于维拉帕米;(2)减轻ARF肾脏病变程度,减少24hr肾小管坏死数及管腔内管型数;(3)对肾脏的超微结构如肾小管上皮细胞微绒毛、线粒体、内质网、肾小球上皮细胞足突、内皮细胞等形态结构均有一定的保护作用,其疗效略优于维拉帕米;(4)提高肾皮质SOD活性,降低MDA含量;(5)提高血清NO水平,同时降低血浆ET含量;(6)显著降低血清TNF-α的水平,疗效明显优于维拉帕米;(7)拮抗小管上皮细胞[Ca~(2+)]_i的升高;(8)使ARF中期(第3d时)肾组织病变较同期其它两组明显减轻,并使肾小管细胞再生修复提前,至ARF恢复期(第5d时)肾组织结构恢复重建良好;作用明显优于维拉帕米;(9)促进ARF中期肾小管上皮细胞PCNA的表达及EGF前体mRNA的合成,从而增加EGF的合成释放,而维拉帕米无此作用。

Methods: Selective renal artery embolization was performed in 68 patients via femoral artery with Seldinger technique, 30 cases were embolized with gelatin sponge chips before operation for renal carcinoma, and 8 cases were embolized with MMC and iodized oil, and gelatin sponge for palliative treatment of advanced renal carcinoma, 6 cases were embolized with thread segments for symptomatic renal angiomyolipoma, 24 cases were embolized with self blood coagulum, gelatin sponge and/or steel coils for traumatic renal hemorrhage.

采用改良Seldinger技术经皮股动脉穿刺插管,对68例进行选择性肾动脉栓塞治疗,其中30例为肾癌术前辅助治疗,应用明胶海绵颗粒栓塞;8例为晚期肾癌姑息性治疗,采用丝裂霉素+碘油的乳剂行末梢栓塞,然后用明胶海绵行肾动脉主干栓塞;6例症状性肾血管平滑肌脂肪瘤,采用真丝线段栓塞;24例肾创伤出血,采用自身血块、明胶海绵或弹簧钢圈栓塞。

Objective: Chronic renal failure is damage to kidney which is caused by various kinds of idiopathic or successive renal disease. Half-backed renal function leads to a series of clinical symptoms which are caused by decompensate. The renal pathological changes develops slowly but it is one-way and irrecoverable. It evolves as terminal renal disease at last. The pathogenesis of CRF can be unpuzzled with the theory of Chinese medical that it is correlated with asthenia, turbid, and stagnancy.

目的: 慢性肾功能衰竭是由各种原发或继发性肾脏疾患导致肾单位破坏,肾脏功能发生障碍引起的一系列症状和代谢紊乱,虽进展缓慢,但其病程单向不可逆,而且在病程某一阶段呈进行性发展或加重,最终发展为终末期肾病。

Contrast-enhanced Ultrasound(1) Renal occupied lesions: the contrast-enhanced imaging feature of renal cell carcinomas are multiformity. It was different in time of start-enhancing and start- expurgation and intensio.3 cases revealed wash-in contemporarily and 6 cases revealed wash- in quickly in time of start-enh ancing.In time of start-expurgating, 3 cases showed wash-out quickly, 3cases revealed wash-out contemporarily,3 cases showed wash-out slowly,6 cases revealed a higher contrast enhancement and 3 cases revealed a even contrast enhancement in enhancing intension; 4 cases with renal cyst s,outline had more clear and inside had no contrast media washing in and enhancing;3cases with renal angiomyolipomas washed in slowly,washed out slowly and equal or low enhanced ; 2cases with renal columnar hypertrophy were no outline appearance and the same enhancement and ech o as the nomal rena tissue.

超声造影诊断结果:(1)肾占位性病变:9例肾透明细胞癌,其超声造影表现多种多样,造影后肾脏恶性病灶造影开始增强的时间、开始廓清的时间及增强强度不等,在开始增强的时间方面,与正常肾皮质同时灌注增强的有3例,表现为快进的有6例,在开始廓清的时间方面,快退3例,同退3例,慢退3例,在增强的强度方面,6例病灶表现为高增强,3例为等增强表现;4例肾囊性病灶,超声造影轮廓较二维超声清晰,但内部始终未见增强;3例肾血管平滑肌脂肪瘤表现为慢进慢退,回声呈等、低增强表现;2例肾柱肥大超声造影无明显包块轮廓出现,造影增强的动态变化时间、强度与正常肾组织一致。

Methods: 1. Male Sprague-Dawley rats were established renal calcium oxalate calculi model. 2. The different dose of LBP, medlar soak and apozem was given. The calcium oxalate crystallization, the renal cell apoptosis and the renal free radical level were detected. 3. The relationship of the renal cell apoptosis index, the renal free radical level and the calcium oxalate crystallization grade in the kidney tubule was determined.

1、给大鼠建立肾草酸钙结石模型;2、予以不同剂量的枸杞多糖,不同浓度的枸杞浸泡剂和煎剂灌喂,比较肾小管内草酸钙结晶的形成,测定肾小管上皮细胞的凋亡指数,自由基水平;3、分析结晶等级评分、细胞凋亡指数和自由基的相互关系,判断枸杞多糖预防肾草酸钙结石的作用机制和效果。

Pathology findings:there were 294 cases of clear renal cell carcinoma (83.76%),including 2 cases of cystis renal cell carcinoma ,27 cases of Grannular renal cell carcinoma ( 7.69 %), 14 cases of papilliform renal cell carcinoma , 6 cases of the mixed type renal cell carcinoma,10 cases of other cell types .

病理结果:透明细胞癌294 例(83.76%,其中囊性肾细胞癌2例),颗粒细胞癌27 例(7.69%),乳头状肾细胞癌14例(3.98%),肾混合细胞癌6例(1.7%),其它癌10例。

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