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rats相关的网络例句
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MethodsThe antipyretic activity of SMPS was observed in rats with high temperature induced by dried yeast and 2,4-dinitrophenol; biliary duct cannula bile at 0.5,1,1.5,2,2.5 h after intraduodenal administration in rats was collected and counted to observe the effect of SMPS on biliary secretion;the weight of cholecyst in mice was counted to observe the effect of SMPS on the cholecyst of mice;the acute toxicity test of SMPS was done with mice.

方法采用干酵母、2,4二硝基苯酚致大鼠发热的模型观察玉米须多糖的解热作用;采用胆管引流法分别收集并计量给药后0.5,1,1.5,2 ,2.5 h的胆汁的方法观察SMPS对肝脏分泌胆汁的影响;采用测定小鼠胆囊重量的方法观察SMPS对胆囊的影响;对SMPS进行了小鼠急性毒性实验。

Rats were trained to establish CTA by intraoral infusion sucrose paired with intraperitoneal injection of LiCl to change taste preference of rats.

2建立味觉厌恶后,甜味和苦味刺激对迷走神经传出放电的影响。

Adult rats are divided into control group, intrapericardial injection group and negative group, sublingual vein injection group and negative group. 6ds after transfection, issues of heart, liver, lung and kidney are stained with X-gal to observe transfection to myocardium and non-target transfection. 3. CaN Aβ gene silencing in vivo. Adult rats are divided into Control group, hypertrophy model group, intrapericardial injection group and negative group, sublingual vein injection group and negative group.

二整体动物水平上探讨心肌有效转染途径:以PLacZ为报告基因,将成年大鼠分为空白对照纽(生理盐水心包腔内注射后超声导入);转染质粒心包腔内注射组及其阴性对照组:质粒+微泡+酶的混合物心包腔内注射,超声导入;转染质粒舌下静脉注射组及其阴性对照组:质粒+微泡混合液经舌下静脉注射,超声导入。

Rats in the model Ⅰ group and the model Ⅱ group were intratracheally instilled with suspension of klebsiella pneumoniae at different doses. Rats in the control group were intratracheally injected with 1 mL saline.

其中,模型Ⅱ组和模型Ⅰ组分别接种相同浓度不同剂量的肺炎克雷伯菌菌液,对照组接种剂量相同的生理盐水。

Methods After Wistar rats were intratracheally instilled 24h,the activities of SOD,the levels of GSH and LPO in lungs ,hearts and livers of rats were measured.

目的探讨沙尘暴细颗粒物(PM2.5)对大鼠肺、心、肝组织的氧化损伤作用。

On experimental day , the rats were intratracheally instilled with bleomycin(5mg/kg body weight)or sterile saline, and then treated with the Fluvastain and saline until they were killed .5 rats of every group were executed in 7,14,28,42 day. Immunohistochemistry and Westernblotting were used to measure the expression of TGF-β, ERK_(1/2) p-ERK protein.

本实验利用博莱酶素诱导大鼠肺间质纤维化,以氟伐他汀为代表研究他汀类药物对肺纤维化大鼠模型的干预作用,并通过观察氟伐他汀对ERK信号通路中重要调节酶ERK_(1/2)的表达及活化的影响,探索肺纤维化的信号传导机制,并为临床提供新的治疗思路。

The Sprague-Dawley rats were intratracheally instilled with lipopolysaccharide(LPS,2 mg·kg-1 per day) for two days to induce acute lung injury.The rats were sacrificed at 72 hours after LPS instillation.Lung morphology was studied.

气管内注入LPS复制大鼠急性肺损伤模型,观察72 h后的病理变化、支气管肺泡灌洗液中细胞总数的变化、免疫组织化学特点以及蓝桉油的影响。

Objectives To study the expressions of STAT1 and PDGF in lung tissue of pulmonary fibrosis model induced by bleomycin in rats, and discuss the possible mechanism.Methods Thirty Wistar rats were randomly divided into two groups (15 in each group), they were bleomycin group and control group.①The BLM group was intratracheally instilled with belomycin (5mg/kg);② NS group was intratracheally instilled with saline (5mg/kg).

目的 观察肺纤维化鼠肺组织信号转导和转录活化因子1(STAT1)和血小板源性生长因子表达的变化,探讨其在肺纤维化的作用机制方法 30只Wistar大鼠随机分博莱霉素组和对照组,①BLM组:气管内灌注BLM(5mg/kg)诱导肺纤维化;②NS组:气管内灌注NS。

we exposed the hibateral sciatic nerves of the rats by means ofexperimental modes,one side was being retained for sixty seconds,the other was justbeing exposed for same time to be the nomal contrl,so we established the rats\'ischiadic nerve crushing injuried models.

我们用实验的方法将大鼠的双侧的坐骨神经进行暴露,一侧钳夹持续一分钟,另一侧作为正常对照组,从而建立了大鼠坐骨神经挤压损伤的动物模型。

The rats in the control group were administrated with parenteral nutrition. The rats in the experimental group were administrated with parenteral nutrition and probiotics via the needle jejunostomy and neck vein for 5 d.

大鼠经盲肠结扎穿孔法及颈静脉空肠置管制成腹腔感染模型后,单独给予肠外营养或加用益生菌,持续5 d。

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