查询词典 rats

Methods: 30 adul male Sprague-Dawley rats were divided randomly into model group and control group. The rats were injected into lateral ventricle with coriaria lactone (50g/kg). The sections of the brains were processed by immunochemistry with antibodies against SYN and GFAP.

成年健康雄性SD大鼠30只，随机分为模型组及对照组，采用马桑内酯(50g/kg)侧脑室注射法建立急性癫痫动物模型，应用免疫组织化学方法观察癫痫持续状态下SYN和GFAP的表达。

Objective:To detect the rat model of non-alcohol fatty liver disease the effect of medical intervention Methods: The Wistar rats were fed with high fatty chow for 12 weeks to induce NAFLD.And then,these rats were fed the low fatty chow,and separately intervened by Rhizoma polygoni cuspidate,Polyene phosphatidylcholine and metformin hydrychloride or controlled by distill water for 4 weeks.The total RNA of adipose tissue was extracted and reversely translated into cDNA.

Non-alcohol fatty liver disease；Intervention；Rhizoma polygoni cuspidatei；Polyene phosphatidylcholine；Metformin hydrychloride非酒精性脂肪肝病（Non-alcohol fatty liver disease，NAFLD）是无过量饮酒史的肝实质细胞脂肪积贮和变性的病理综合症，是由于肝脏本身和各种肝外因素引起的肝脏脂肪代谢功能障碍，导致肝细胞内脂类物质蓄积过多的一种病理变化，常见原因有营养缺乏、肥胖、糖尿病、妊娠、糖皮质激素、肝炎及药物或毒物的损伤等[1]，发病机理尚未完全清楚。

Methods The Cynomorium polysaccharide and Cynomorium water extract were extracted and prepared chewable tablets. Wistar rats were randomly divided into normal control, aging model, VitE, Cynomorium polysaccharide chewable tablets and Cynomorium water extract chewable tablets groups. Dgalactose was injected to establish the aging model. After drug intervention, the proliferation activity of rat spleen lymphocytes stimulated by ConA was detected by MTT assay and the spleen index was measured. The pathological morphological changes of spleen were observed. The activity of superoxide dismutase, the content of malondialdehyde and nitric oxide in serum of rats were detected.

提取并制备锁阳多糖、水提取物咀嚼片；Wistar大鼠随机分为空白对照组、衰老模型组、维生素E对照组、锁阳多糖组、水提物组，以D半乳糖建立衰老大鼠模型，药物干预后，MTT法检测各组大鼠脾淋巴细胞对ConA刺激的增殖能力，检测各组大鼠脾脏指数，观察脾组织的病理形态学变化；并检测各组大鼠血清中超氧化物歧化酶活性、丙二醛及一氧化氮的含量。

In decerebrate rats, HFBcaused a progressive dose-related decrease in MAC (maximum 25%),but in intact rats, an increase from 0.8 to 1.2 neuronal responsecaused an increase in neuronal response.

在去大脑大鼠中， HFB 引起剂量相关的 MAC 渐进性减少（最多减少25％），但是在整体大鼠中， MAC 从0.8增加至1.2使得神经元反应增加。

In decerebrate rats,1.2 MAC BNZ depressed nociceptive responses by 60%, with a furtherpercentage decrease continuing from 0.8 to 1.2 MAC approximatelyequal to that found in intact rats.

去大脑大鼠中，1.2 MAC 的 BNZ 对伤害性感受反应的抑制达60％，且从0.8MAC 到1.2MAC 神经元对伤害性刺激反应百分比进一步减少，近似於在整体大鼠中的发现。

Left anterior descending coronary artery of the remaining rats were deligated to establish myocardial infarction models. Following 24 hours of deligation, 36 survival rats were equally and randomly divided into a control group and a experimental group.

健康雄性SD大鼠90只，取12只作为假手术组，剩余大鼠结扎冠状动脉左前降支建立心肌梗死模型，结扎后24 h存活36只，随机分为对照组和实验组，18只/组。

METHODS: Newborn rats in the model and laser treatment groups were treated with low-concentration oxygen (containing 8% O2 and 92% N2) following deligation of the left common carotid artery to establish models of ischemia-hypoxia brain damage. Rats in the sham operation group did not receive deligation of the left common carotid artery, but exposed to the air.

模型组、激光治疗组新生大鼠通过结扎左颈总动脉后，再吸入低浓度氧(含体积分数为8%的O2、体积分数为92%的N2)建立缺血缺氧性脑损伤模型；假手术组不结扎左颈总动脉，置于正常空气中。

Methods The rat models of heart failure were established by left anterior descending coronary artery deligation. After 6 w, 24 surviving rats were divided into sham,model and Ramipril groups at random,8 rats in each,and were treated with medicines by intragastric administration respectively. At 10 w, left ventricular enddiastolic pressure, left ventricular systolic pressure,+dp/dtmax anddp/ dtmaxof left ventricular pressure were measured.

结扎大鼠左冠状动脉前降支并饲养6 w的16只存活大鼠，随机分为模型组及雷米普利组，每组8只，另取8只大鼠为假手术组，连续灌胃给药4 w后测定大鼠血流动力学参数，ELISA方法检测血清及左心室非梗死区AngⅡ的含量，RTPCR 法测定左心室非梗死区心肌组织AT1R mRNA表达水平，Masson染色观察非梗死区心肌胶原的沉积。

ABSTRACT: Objective To observe the ultrastructural changes of detrusor in experimental T2DM rats. Methods 14 Wistar rats were divided into two groups.

目的 观察2型糖尿病(T2DM)大鼠逼尿肌细胞线粒体超微结构的改变。

The induction of LL-LTP in hippocampal CA1 region may be sexually dimorphic, and the male rats can react to a broader range of tetanizing stimulation compared with female rats.

在LL-LTP形成后，特定的刺激参数可以使其反转，代谢性谷氨酸受体拮抗剂的使用可以加强和延长这种反转效应。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。