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Methods 60 Wistar rats were divided into 6 groups and each group had 10 rats.

将60只Wistar雄性大鼠随机分成6组,每组10只。

Methods Totally 80 healthy Wistar rats were divided into 4 groups randomly (20 rats in each group).

选健康Wistar大鼠80只,雌雄不拘,随机分为4组,每组20只。

Methods The rats were injected abdominally with econopred 24.5mg/kg for twice per week, and the early models of HANFH were successfully induced after six weeks. 32 rats were divided randomly into two groups, the treatment group (n=l6) were administrated with Taohong Siwu decoction, and the control group (n=16) with physiological saline.

模型组每周2次腹腔注射醋酸泼尼松龙24.5mg/kg,经6周诱导出早期激素性股骨头缺血坏死的模型,随机分为治疗组16只和对照组16只,分别灌服桃红四物汤和生理盐水,在灌胃后第6、8周,检测股骨头局部TGF-β1 mRNA的转录水平。

The rest SD rats were injected abdominally to induce chronic-kindling models by PTZ 35mg/kg for 28 days,then 30 having build-up model rats were divided randomly completely into three groups: model, YXL, VPA.

将SD大鼠随机取10只作为正常组,其余大鼠以PTZ 35mg/kg腹腔注射28天作慢性点燃造模,再取造模成功大鼠随机分为3组:模型组、丙戊酸钠组和愈痫灵颗粒剂组。

The first day after C6 cells were seeded in rats bearing glioma, these rats were abdominally injected PA at the dose of 550mg/kg/d, twice a day for two weeks or DMF 570mg/kg/d in the same way.

分别在荷瘤大鼠种植胶质瘤细胞C6后第1d,对大鼠进行苯乙酸550mg/kg/d,二次/日,连续两周腹腔注射,或二甲基甲酰胺570mg/kg/d,二次/日,连续两周腹腔注射治疗。

Methods 120 healthy Wistar rats were randomly divided into 6 groups after adaptative feeding for one week: 4month control (4C) group, 14month control (14C) group, 14month JL (14JL) group, 24month control (24C) group,24month JL (24JL) group and (1424)month JL (1424JL) group, 20 rats per group, male and female were half to half in every group.

选用Wistar大鼠120只,适应性喂养1 w后随机分为6组:4月龄空白对照组(4C组)、14月龄空白对照组(14C组)、14月龄加味青麟丸组(14JL组)、24月龄空白对照组(24C组)、24月龄加味青麟丸组(24JL组)和14~24月龄加味青麟丸组(14~24JL组),每组各20只,雌雄各半。各组均按实验设计要求分别喂养后剖检。

Methods: Diabetes gastroparesis model rats were built by means of intraperitoneal injection 5% alloxanate aqu and Prepared Radix Rehmanniae once. Observed the effect of JG on rats' stomach power, serum glucose, serum motilin, cholecystokinin.

采用一次性腹腔注射5%四氧嘧啶水溶液加熟地复制糖尿病胃轻瘫模型,观察加味柴平颗粒剂对大鼠胃动力、血糖、血清胃动素、胆囊收缩素的影响。

It showed similar pattern with that in lung tissues. Conclusion: There were TGF-β1 mRNA expression in both normal rats and pulmonary fibrosis rats. AMs seemed to be the main source of TGF-β1 mRNA in lung tissues, especially at stage of alveolitis.

TGF-β1 mRNA在正常大鼠及肺纤维化大鼠中均有表达,后者表达明显高于前者;肺纤维化大鼠肺内TGF-β1 mRNA 可能主要来源于AM。

Methods: 60 SD rats were divided into 4 groups randomly, the nomal group, the model group, QZHJ group, and the hydrocortisone group. Rats were sacrificed in bdtch on the 3rd, 7th and 21st day after treatment, then the concentration of interleukin-8(IL-8) in bronchoalveolar lavage fluid and the content of transforming growth factor-β1 (TGF-β1) of lung tissue were deected, while the severity of alveolitis and fibrosis were measured.

将60只健康SD大鼠随机分为正常组、模型组、芪术合剂组和强的松组,经气管滴入博莱霉素复制肺纤维化大鼠模型,并分组予以治疗,于治疗后第3、7、21日分批处死大鼠,每组每次6只;用免疫组化法观察肺组织转化生长因子-β1的表达,并测定支气管肺泡灌洗液中中性粒细胞和白介素-8的水平。

Expressed as a fraction ofthe minimum alveolar anesthetic concentration preventingmovement in 50% of rats, the amnestic 50% effective dose (ED50)for fear to tone in control rats inhaling isoflurane and injectedwith saline intraperitoneally i.p.

用能防止50%大鼠动反应的最小肺泡浓度的一个分数来表示的遗忘音调恐惧的50%有效剂量( ED50),在吸入异氟醚且腹膜内

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这两个团体间的分歧难以掩饰。

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