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Rosiglitazone can decrease the expression of RBP4mRNA in 3T3-L1adipocyte that used dexamethasone induced into insulin resistance model, and can improve the expression of GLUT4mRNA. 3. Rosiglitazone can increase the glucose intake in 3T3-L1 adipocyte. Chapter 2 Superficial Observed the Expression of Rebinding Protein 4 in OLETF rats OBJECTIVE Compare the level of the expression RBP4 between OLETF rats and LETO rats in adipose tissue; observe the function that rosiglitazone interfere in the expression of RBP4; observe the relationship between RBP4 and GLUT4, then approach the possible mechanism that RBP4 induce to insulin resistance.

第二章罗格列酮对OLETF鼠RBP4的影响，以及RBP4在OLETF鼠内脏脂肪表达的初步观察比较自发性2型糖尿病OLETF鼠与同家系LETO鼠脂肪组织视黄醇结合蛋白(RBP4)水平；观察罗格列酮对RBP4的干预作用；观察脂肪组织RBP4与GLUT4水平的相关性，从而探讨RBP4导致胰岛素抵抗的可能机制。

Methods: Prostate primary artery of twenty rats been severed to make relatival prostate hypoxia before rats are in adolescence, another group of rats are nature.

选取青春前期SD雄性大鼠40只，将实验组大鼠前列腺的主要动脉阻断，复制出大鼠前列腺相对性缺血缺氧状态，对照组为假手术组。

Methods: 70 adult and health rats were randomly divided into control group (10 rats ) and scald group (60 rats).

健康、成年大白鼠70只，随机分为正常对照组10只，烫伤组60只。

To detect effects on apoptosis of cortex neuron in DHCA rats, the rat model of occluding bilateral common carotid artery in the deep hypothermic (21C) rats had been established, and flow cytometry, TUNEL, electron microscope, immunohistochemistry and RT-PCR had been studied.: part I: To establish a new rat model of cerebral ischemia/protect during DHCA: 20 SD rats were used in the experiment, little animal respirator was to assist anesthetized rat's respiration after tracheal cannula, artery press through artery spile in femoral artery, electrocardiograph, temperature, blood saturation of O2 (SaO2) and arterial blood gases were monitored during the whole experiment, blood flow of bilateral common carotid artery of rat were occluded about 60 minute when its temperature was reduced to 21 C by ice, then normal temperature was resumed.

本研究采用大鼠深低温颈总动脉阻断模型，应用流式细胞仪、电镜、原位缺口末端标记法、免疫组化法和RT-PCR技术分别检测深低温脑缺血对皮层神经细胞调亡的影响方法：第一部分深低温停循环脑保护大鼠模型的建立：SD大鼠20只，麻醉后气管插管辅助呼吸，用冰块将大鼠的体温降至(21±1)℃，阻断双侧颈总动脉，记录体温、心率、血压、血氧饱和度和血气分析，60分钟后恢复颈总动脉血流，用Longa等的5级评分标准评定4小时和24小时后大鼠神经系统损伤情况。

Result: 1 Comparing with control rats, after giving sucrose stimulability ,small intestine peristalsis rate debased in the CTA rats; and in the CTA rats, when sucrose stimulability was given, small intestine peristasis rate was lower than when water stimulabitiy was given.

2给予正常大鼠蔗糖刺激时，切断迷走神经的大鼠的小肠推进率明显低于假手术组的。

INTERVENTIONS: 4 vessel occlusion(4VO) brain ischemic models in rats stained with thionine staining and GFAP immunohistochemistry staining. were used. Sixty-four rats, of which bilateral vertebral arteries were occluded permanently by electrocautery, were divided into the following 8groups: sham operation group, cerebral ischemic preconditioninggroup, ischemic insult group; BIT group; MTPG + sham operation group;MTPG+BIT group; MTPG+ischemia group and -4C3HPG+BIT coup. All the rats were killed 7 days after the operation or the final ischemic treatment. Cerebral sections were selected and stained with thionine staining and GFAP immunohistochemistry staining.

干预：采用大鼠四血管闭塞全脑缺血模型，应用硫堇染色和胶质纤维酸性蛋白免疫组化法。64只大鼠椎动脉凝闭后分为假手术组、单纯预处理组、单纯缺血组、脑缺血耐受组，MTPG+假手术组、MTPG+脑缺血耐受组、MTPG+缺血组和-4C3HPG+脑缺血耐受组，所有动物均在手术后或末次缺血后7 d处死取材，行脑组织切片，硫堇染色和胶质纤维酸性蛋白免疫组化染色。

After administerded for 30 days, all rats were killed and anatomized. The nephric exterior of model control group rats was offwhite, instead, that of Yi Shen Granule groups rats showed red grain in offwhite exterior. There were tumefaction of tissue and sediment of adenine in all kidney.

病理组织学观察发现，与空白组比较，造模动物肾小管均有扩张，给药后其扩张程度有显著性差异，采用半定量的方法对肾小管扩张程度进行分级比较，结果显示大剂量组动物肾小管扩张程度较轻。

SC acan turn pneuma fine, add appetite, increase activity and improve color pattern and anischuria of RHR rats.SC didn't have evidence effect on SHR rats'body weight, but can increase RHR rats'body weight. 3.3.3 Find The effect on endothelin、nitrogen monoxidum、atrial natriuretic peptide and renin-angiotensin system of SC .

3.3.3三草降压汤对血浆内皮素、一氧化氮、心钠素及肾素-血管紧张素系统的影响 SC能明显降低SHR、RHR两种高血压大鼠模型血浆ET浓度、升高血浆NO浓度、调整二者的平衡，这可能是其发挥降压作用的重要机制。

METHODS: 40 rats were randomly divided into four groups:normal group, asthenic spleen group, high-dosage group and low-dosage group with 10 rats in each group, the asthenic spleen rat models were established in group B, C group as well as group D. Rats in group B, group C, group D were fed with normal distilled water, high-dosage of Guizhi decoction and low-dosage of Guizhi decoction respectively, NFAT mRNA, IL-4 mRNA, IFN-γmRNA were determined by PCR.

40只大鼠随机分为空白对照组、脾虚组、桂枝汤大剂量组和桂枝汤小剂量组等4组，每组10只；用规范的脾气虚证大鼠模型造模方法将B组、C组、D组大鼠造模，B组、C组、D组分别用蒸馏水、桂枝汤大剂量、桂枝汤小剂量灌胃，标本采集后用RT-PCR检测NFAT mRNA，IL-4 mRNA和IFN-γ mRNA表达水平。

Methods The experimental arrhythmia models were induced with barium chloride (BaCl2),uabaina and aconitine in rats and cobayaes.The incidence rate and mortality rate of arrhythmia,the emergence time and the duration of arrhythmia induced with BaCl2 were recorded in rats pretreated with CASI (in dosages of 25,50, and 100 mg·kg-1 sublingual vein injection).At the same time,the dosages of uabaina and aconitine were observed when the ventricular premature contraction,ventricular tachycardia,ventricular fibrillation and cardiac asystole were appeard in rats and cobayaes pretreated with CASI (in dosages of 25,50, and 100 mg·kg-1 sublingual vein injection).

制备氯化钡(BaCl2)、哇巴因及乌头碱诱导的大鼠及豚鼠实验性心律失常模型，CASI按25、50、100 mg·kg-1分为3个剂量组，舌下静脉给药1次，不给CASI作为对照组，记录大鼠给BaCl2后心律失常的发生率、出现时间、持续时间及动物的死亡率；观察豚鼠及大鼠出现室性早搏、室性心动过速、心室颤动及心脏停搏时哇巴因及乌头碱的用量。

According to the clear water experiment, aeration performance of the new equipment is good with high total oxygen transfer coefficient and oxygen utilization ratio.

曝气设备的动力效率在叶轮转速为120rpm～150rpm时取得最大值，此时氧利用率和充氧能力也具有较高值。

The environmental stability of that world - including its crushing pressures and icy darkness - means that some of its most famous inhabitants have survived for eons as evolutionary throwbacks, their bodies undergoing little change.

稳定的海底环境─包括能把人压扁的压力和冰冷的黑暗─意谓海底某些最知名的栖居生物已以演化返祖的样态活了万世，形体几无变化。